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Role of TRPM2 Channel in Mediating H2O2-Induced Ca2+ Entry and Endothelial Hyperpermeability

Circulation research, 2008-02, Vol.102 (3), p.347-355

2008

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Ca2+ Signaling, TRP Channels, and Endothelial Permeability

Microcirculation (New York, N.Y. 1994), 2006-12, Vol.13 (8), p.693-708

2006

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Sphingosine 1-Phosphate-induced Mobilization of Intracellular Ca2+ Mediates Rac Activation and Adherens Junction Assembly in Endothelial Cells

The Journal of biological chemistry, 2005-04, Vol.280 (17), p.17320-17328

2005

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Functional role of TRPC channels in the regulation of endothelial permeability

Pflügers Archiv, 2005-10, Vol.451 (1), p.131-142

2005

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CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia

Journal of neurophysiology, 2016-01, Vol.115 (1), p.345-354

2016

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Nitric Oxide Modulates Cardiac Na+ Channel via Protein Kinase A and Protein Kinase G

Circulation research, 2001-11, Vol.89 (11), p.1005-1013

2001

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TRPM2 channel regulates endothelial barrier function

Advances in experimental medicine and biology, 2010, Vol.661, p.155

2010

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Gαq-TRPC6-mediated Ca2+ Entry Induces RhoA Activation and Resultant Endothelial Cell Shape Change in Response to Thrombin

The Journal of biological chemistry, 2007-03, Vol.282 (11), p.7833-7843

2007

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Palytoxin disrupts cardiac excitation-contraction coupling through interactions with P-type ion pumps

American Journal of Physiology: Cell Physiology, 2004-08, Vol.287 (2), p.C527-C538

2004

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Protein Kinase Cα Phosphorylates the TRPC1 Channel and Regulates Store-operated Ca2+ Entry in Endothelial Cells

The Journal of biological chemistry, 2004-05, Vol.279 (20), p.20941-20949

2004

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Tumor necrosis factor-alpha-induced TRPC1 expression amplifies store-operated Ca2+ influx and endothelial permeability

American journal of physiology. Lung cellular and molecular physiology, 2004-12, Vol.287 (6), p.L1303-L1313

2004

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CaV3.2 T-type Ca 2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia

Journal of neurophysiology, 2016-01, Vol.115 (1), p.345-354

2016

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RhoA interaction with inositol 1,4,5-trisphosphate receptor and transient receptor potential channel-1 regulates Ca2+ entry. Role in signaling increased endothelial permeability

The Journal of biological chemistry, 2003-08, Vol.278 (35), p.33492

2003

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RhoA Interaction with Inositol 1,4,5-Trisphosphate Receptor and Transient Receptor Potential Channel-1 Regulates Ca2+ Entry

The Journal of biological chemistry, 2003-08, Vol.278 (35), p.33492-33500

2003

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Analysis of moricizine block of sodium current in isolated guinea-pig atrial myocytes: Atrioventricular difference of moricizine block

Vascular pharmacology, 2002-03, Vol.38 (3), p.131-141

2002

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Protein kinase Cα phosphorylates the TRPC1 channel and regulates store-operated Ca2+ entry in endothelial cells

The Journal of biological chemistry, 2011-10, Vol.286 (41), p.36162-36162

2011

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A novel mutation causing complete deficiency of thyroxine binding globulin

Clinical endocrinology (Oxford), 1997-07, Vol.47 (1), p.1-5

1997

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Analysis of moricizine block of sodium current in isolated guinea-pig atrial myocytes

Vascular pharmacology, 2002-03, Vol.38 (3), p.131-141

2002

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Role of TRPM2 Channel in Mediating H 2 O 2 -Induced Ca 2+ Entry and Endothelial Hyperpermeability

Circulation research, 2008-02, Vol.102 (3), p.347-355

2008

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Role of TRPM2 Channel in Mediating H sub(2)O sub(2)-Induced Ca super(2+) Entry and Endothelial Hyperpermeability

Circulation research, 2008-02, Vol.102 (3), p.347-355

2008

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Changes in Ca(2+) cycling proteins underlie cardiac action potential prolongation in a pressure-overloaded guinea pig model with cardiac hypertrophy and failure

Circulation research, 2000-03, Vol.86 (5), p.558-570

2000

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Neutrophil‐induced regulation of [Ca2+]i in human endothelial cell:role of redox sensitive ion channels

The FASEB journal, 2006-03, Vol.20 (5), p.A1179-A1179

2006

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Galphaq-TRPC6-mediated Ca2+ entry induces RhoA activation and resultant endothelial cell shape change in response to thrombin

The Journal of biological chemistry, 2007-03, Vol.282 (11), p.7833-7843

2007

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GÎ±q-TRPC6-mediated Ca2+ Entry Induces RhoA Activation and Resultant Endothelial Cell Shape Change in Response to Thrombin

The Journal of biological chemistry, 2007-03, Vol.282 (11), p.7833

2007

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G alpha sub(q)-TRPC6-mediated Ca super(2+) Entry Induces RhoA Activation and Resultant Endothelial Cell Shape Change in Response to Thrombin

The Journal of biological chemistry, 2007-03, Vol.282 (11), p.7833-7843

2007

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Role of TRPM2 Channel in Mediating H2O2-Induced Ca2+ Entry and Endothelial Hyperpermeability

Ca2+ Signaling, TRP Channels, and Endothelial Permeability

Sphingosine 1-Phosphate-induced Mobilization of Intracellular Ca2+ Mediates Rac Activation and Adherens Junction Assembly in Endothelial Cells

Functional role of TRPC channels in the regulation of endothelial permeability

CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia

Nitric Oxide Modulates Cardiac Na+ Channel via Protein Kinase A and Protein Kinase G

TRPM2 channel regulates endothelial barrier function

Gαq-TRPC6-mediated Ca2+ Entry Induces RhoA Activation and Resultant Endothelial Cell Shape Change in Response to Thrombin

Palytoxin disrupts cardiac excitation-contraction coupling through interactions with P-type ion pumps

Protein Kinase Cα Phosphorylates the TRPC1 Channel and Regulates Store-operated Ca2+ Entry in Endothelial Cells

Tumor necrosis factor-alpha-induced TRPC1 expression amplifies store-operated Ca2+ influx and endothelial permeability

CaV3.2 T-type Ca 2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia

RhoA interaction with inositol 1,4,5-trisphosphate receptor and transient receptor potential channel-1 regulates Ca2+ entry. Role in signaling increased endothelial permeability

RhoA Interaction with Inositol 1,4,5-Trisphosphate Receptor and Transient Receptor Potential Channel-1 Regulates Ca2+ Entry

Analysis of moricizine block of sodium current in isolated guinea-pig atrial myocytes: Atrioventricular difference of moricizine block

Protein kinase Cα phosphorylates the TRPC1 channel and regulates store-operated Ca2+ entry in endothelial cells

A novel mutation causing complete deficiency of thyroxine binding globulin

Analysis of moricizine block of sodium current in isolated guinea-pig atrial myocytes

Role of TRPM2 Channel in Mediating H 2 O 2 -Induced Ca 2+ Entry and Endothelial Hyperpermeability

Role of TRPM2 Channel in Mediating H sub(2)O sub(2)-Induced Ca super(2+) Entry and Endothelial Hyperpermeability

Changes in Ca(2+) cycling proteins underlie cardiac action potential prolongation in a pressure-overloaded guinea pig model with cardiac hypertrophy and failure

Neutrophil‐induced regulation of [Ca2+]i in human endothelial cell:role of redox sensitive ion channels

Galphaq-TRPC6-mediated Ca2+ entry induces RhoA activation and resultant endothelial cell shape change in response to thrombin

GÎ±q-TRPC6-mediated Ca2+ Entry Induces RhoA Activation and Resultant Endothelial Cell Shape Change in Response to Thrombin

G alpha sub(q)-TRPC6-mediated Ca super(2+) Entry Induces RhoA Activation and Resultant Endothelial Cell Shape Change in Response to Thrombin

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