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Local bradykinin generation in hereditary angioedema

Journal of allergy and clinical immunology, 1999-12, Vol.104 (6), p.1321-1322

1999

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Activation of the coagulation cascade in C1-inhibitor deficiencies

Blood, 1997-05, Vol.89 (9), p.3213-3218

1997

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Hereditary and acquired angioedema: Problems and progress: Proceedings of the third C1 esterase inhibitor deficiency workshop and beyond

Journal of Allergy and Clinical Immunology, 2004-09, Vol.114 (3), p.S51-S131

2004

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Bradykinin and the pathophysiology of angioedema

International immunopharmacology, 2003-03, Vol.3 (3), p.311-317

2003

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Pathogenetic and clinical aspects of C1 inhibitor deficiency

Immunobiology (1979), 1998-08, Vol.199 (2), p.366-376

1998

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Plasma bradykinin in angio-oedema

The Lancet (British edition), 1998-06, Vol.351 (9117), p.1693-1697

1998

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Aminopeptidase P in individuals with a history of angio-oedema on ACE inhibitors

The Lancet (British edition), 2002-06, Vol.359 (9323), p.2088-2089

2002

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Drug-induced angioedema without urticaria. Incidence, prevention and management

Drug safety, 2001, Vol.24 (8), p.599-606

2001

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Frequent de novo mutations and exon deletions in the C1inhibitor gene of patients with angioedema

Journal of allergy and clinical immunology, 2000-12, Vol.106 (6), p.1147-1154

2000

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Angiotensin-converting enzyme inhibitor-associated angioedema is characterized by a slower degradation of des-arginine(9)-bradykinin

The Journal of pharmacology and experimental therapeutics, 2002-10, Vol.303 (1), p.232

2002

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Hereditary and acquired C1-inhibitor deficiency: biological and clinical characteristics in 235 patients

Medicine (Baltimore), 1992-07, Vol.71 (4), p.206-215

1992

Volltextzugriff (PDF)

Increase of Bradykinin in Plasma of Patients Undergoing Cardiopulmonary Bypass

Chest, 2001-12, Vol.120 (6), p.1776-1782

2001

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Hereditary Angioedema

The New England journal of medicine, 1996-06, Vol.334 (25), p.1666-1667

1996

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A Variant in XPNPEP2 Is Associated with Angioedema Induced by Angiotensin I–Converting Enzyme Inhibitors

American journal of human genetics, 2005-10, Vol.77 (4), p.617-626

2005

Volltextzugriff (PDF)

Parallel reduction of plasma levels of high and low molecular weight kininogen in patients with cirrhosis

Thrombosis and haemostasis, 1999-11, Vol.82 (5), p.1428

1999

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Side effects of long-term prophylaxis with attenuated androgens in hereditary angioedema: Comparison of treated and untreated patients

Journal of allergy and clinical immunology, 1997-02, Vol.99 (2), p.194-196

1997

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Increase of bradykinin in plasma of patients undergoing cardiopulmonary bypass: The importance of lung exclusion

Chest, 2001-12, Vol.120 (6), p.1776-1782

2001

Volltextzugriff (PDF)

Vasopeptidases and their inhibitors

Recenti progressi in medicina, 2002-10, Vol.93 (10), p.519

Agostoni, Angelo

2002

Volltextzugriff (PDF)

Idiopathic nonhistaminergic angioedema

The American journal of medicine, 1999-06, Vol.106 (6), p.650-654

1999

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Angioedema associated with angiotensin-converting enzyme inhibitor use: outcome after switching to a different treatment

Archives of internal medicine (1960), 2004-04, Vol.164 (8), p.910

2004

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Contraindications to the use of ace inhibitors in patients with c1 esterase inhibitor deficiency

The American journal of medicine, 1991, Vol.90 (1), p.278-278

1991

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Activation of the contact system and fibrinolysis in autoimmune acquired angioedema: A rationale for prophylactic use of tranexamic acid

Journal of allergy and clinical immunology, 1994-05, Vol.93 (5), p.870-876

1994

Volltextzugriff (PDF)

The region 1–11 of Alzheimer amyloid-β is critical for activation of contact-kinin system

Neurobiology of aging, 2001, Vol.22 (1), p.63-69

2001

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Placebo and EU guidelines

The Lancet (British edition), 1995-12, Vol.346 (8991), p.1710-1710

Agostoni, Angelo

1995

Volltextzugriff (PDF)

Antibodies to tissue‐type plasminogen activator in plasma from patients with primary antiphospholipid syndrome

British journal of haematology, 2000-03, Vol.108 (4), p.871-875

2000

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Local bradykinin generation in hereditary angioedema

Activation of the coagulation cascade in C1-inhibitor deficiencies

Hereditary and acquired angioedema: Problems and progress: Proceedings of the third C1 esterase inhibitor deficiency workshop and beyond

Bradykinin and the pathophysiology of angioedema

Pathogenetic and clinical aspects of C1 inhibitor deficiency

Plasma bradykinin in angio-oedema

Aminopeptidase P in individuals with a history of angio-oedema on ACE inhibitors

Drug-induced angioedema without urticaria. Incidence, prevention and management

Frequent de novo mutations and exon deletions in the C1inhibitor gene of patients with angioedema

Angiotensin-converting enzyme inhibitor-associated angioedema is characterized by a slower degradation of des-arginine(9)-bradykinin

Hereditary and acquired C1-inhibitor deficiency: biological and clinical characteristics in 235 patients

Increase of Bradykinin in Plasma of Patients Undergoing Cardiopulmonary Bypass

Hereditary Angioedema

A Variant in XPNPEP2 Is Associated with Angioedema Induced by Angiotensin I–Converting Enzyme Inhibitors

Parallel reduction of plasma levels of high and low molecular weight kininogen in patients with cirrhosis

Side effects of long-term prophylaxis with attenuated androgens in hereditary angioedema: Comparison of treated and untreated patients

Increase of bradykinin in plasma of patients undergoing cardiopulmonary bypass: The importance of lung exclusion

Vasopeptidases and their inhibitors

Idiopathic nonhistaminergic angioedema

Angioedema associated with angiotensin-converting enzyme inhibitor use: outcome after switching to a different treatment

Contraindications to the use of ace inhibitors in patients with c1 esterase inhibitor deficiency

Activation of the contact system and fibrinolysis in autoimmune acquired angioedema: A rationale for prophylactic use of tranexamic acid

The region 1–11 of Alzheimer amyloid-β is critical for activation of contact-kinin system

Placebo and EU guidelines

Antibodies to tissue‐type plasminogen activator in plasma from patients with primary antiphospholipid syndrome

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