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HER2 stabilizes EGFR and itself by altering autophosphorylation patterns in a manner that overcomes regulatory mechanisms and promotes proliferative and transformation signaling

Oncogene, 2013-08, Vol.32 (35), p.4169-4180

2013

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First Discovery of a Fast Radio Burst at 350 MHz by the GBNCC Survey

The Astrophysical journal, 2020-12, Vol.904 (2), p.92

2020

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Inhibition of SHP2 leads to mesenchymal to epithelial transition in breast cancer cells

Cell death and differentiation, 2008-06, Vol.15 (6), p.988-996

2008

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The Green Bank North Celestial Cap Survey. VII. 12 New Pulsar Timing Solutions

The Astrophysical journal, 2023-02, Vol.944 (2), p.154

2023

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The Green Bank North Celestial Cap Survey. IX. Timing Follow-up for 128 Pulsars

The Astrophysical journal, 2024-02, Vol.962 (2), p.167

2024

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The Green Bank North Celestial Cap Survey. VIII. 21 New Pulsar Timing Solutions

The Astrophysical journal, 2023-10, Vol.956 (1), p.40

2023

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SHP2 is up-regulated in breast cancer cells and in infiltrating ductal carcinoma of the breast, implying its involvement in breast oncogenesis

Histopathology, 2008-10, Vol.53 (4), p.389-402

2008

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Modulation of α-catenin Tyr phosphorylation by SHP2 positively effects cell transformation induced by the constitutively active FGFR3

Oncogene, 2006-11, Vol.25 (54), p.7166-7179

2006

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Regulation of anti-apoptotic signaling by Kruppel-like factors 4 and 5 mediates lapatinib resistance in breast cancer

Cell death & disease, 2015-03, Vol.6 (3), p.e1699-e1699

2015

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Abstract P6-20-17: Targeting SHP2 for the treatment of HER2-positive breast cancer

Cancer research (Chicago, Ill.), 2019-02, Vol.79 (4_Supplement), p.P6-P6-20-17

2019

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The Green Bank North Celestial Cap Pulsar Survey. V. Pulsar Census and Survey Sensitivity

The Astrophysical journal, 2020-04, Vol.892 (2), p.76

2020

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The Green Bank Northern Celestial Cap Pulsar Survey. VI. Discovery and Timing of PSR J1759+5036: A Double Neutron Star Binary Pulsar

The Astrophysical journal, 2021-11, Vol.922 (1), p.35

2021

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Concomitant activation of the PI3K-Akt and the Ras-ERK signaling pathways is essential for transformation by the V-SEA tyrosine kinase oncogene

Oncogene, 2002-01, Vol.21 (5), p.697-707

2002

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Development of an Efficient “Substrate-trapping” Mutant of Src Homology Phosphotyrosine Phosphatase 2 and Identification of the Epidermal Growth Factor Receptor, Gab1, and Three Other Proteins as Target Substrates

The Journal of biological chemistry, 2003-04, Vol.278 (16), p.13952-13958

2003

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P-43 Correlation of mesothelin expression with recurrence in colorectal cancer (CRC) patients

Annals of oncology, 2022-06, Vol.33, p.S262

2022

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The phosphotyrosine phosphatase SHP2 is a critical mediator of transformation induced by the oncogenic fibroblast growth factor receptor 3

Oncogene, 2003-10, Vol.22 (44), p.6909-6918

2003

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ATP-stimulated smooth muscle cell proliferation requires independent ERK and PI3K signaling pathways

American journal of physiology. Heart and circulatory physiology, 1998-10, Vol.275 (4), p.H1209-H1215

1998

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Molecular Mechanism for a Role of SHP2 in Epidermal Growth Factor Receptor Signaling

Molecular and Cellular Biology, 2003-11, Vol.23 (21), p.7875-7886

2003

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Characterization of a new monoclonal antibody to triplex DNA and immunofluorescent staining of mammalian chromosomes

The Journal of biological chemistry, 1994-03, Vol.269 (9), p.7019-7023

1994

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Triplex DNA in the nucleus: direct binding of triplex-specific antibodies and their effect on transcription, replication and cell growth

Biochemical journal, 1996-06, Vol.316 ( Pt 2) (2), p.461-466

1996

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Modulation of -catenin Tyr phosphorylation by SHP2 positively effects cell transformation induced by the constitutively active FGFR3

Oncogene, 2006-11, Vol.25 (54), p.7166-7179

2006

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Molecular mechanisms of ATP and insulin synergistic stimulation of coronary artery smooth muscle growth

American journal of physiology. Heart and circulatory physiology, 2001-02, Vol.280 (2), p.H795-H801

2001

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P2-02-05: Molecular Mechanism for Src Homology Phosphotyrosyl Phosphatase 2 Regulation of Cell Motility and Migration

Cancer research (Chicago, Ill.), 2011-12, Vol.71 (24_Supplement), p.P2-P2-02-05

2011

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P3-01-15: The Role of Src Homology Phosphotyrosyl Phosphatase-2 in Basal-Type/Triple-Negative Breast Cancer – Implications for Targeted Therapy

Cancer research (Chicago, Ill.), 2011-12, Vol.71 (24_Supplement), p.P3-P3-01-15

2011

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A Rho-associated Protein Kinase, ROKα, Binds Insulin Receptor Substrate-1 and Modulates Insulin Signaling

The Journal of biological chemistry, 1998-02, Vol.273 (8), p.4740-4746

1998

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HER2 stabilizes EGFR and itself by altering autophosphorylation patterns in a manner that overcomes regulatory mechanisms and promotes proliferative and transformation signaling

First Discovery of a Fast Radio Burst at 350 MHz by the GBNCC Survey

Inhibition of SHP2 leads to mesenchymal to epithelial transition in breast cancer cells

The Green Bank North Celestial Cap Survey. VII. 12 New Pulsar Timing Solutions

The Green Bank North Celestial Cap Survey. IX. Timing Follow-up for 128 Pulsars

The Green Bank North Celestial Cap Survey. VIII. 21 New Pulsar Timing Solutions

SHP2 is up-regulated in breast cancer cells and in infiltrating ductal carcinoma of the breast, implying its involvement in breast oncogenesis

Modulation of α-catenin Tyr phosphorylation by SHP2 positively effects cell transformation induced by the constitutively active FGFR3

Regulation of anti-apoptotic signaling by Kruppel-like factors 4 and 5 mediates lapatinib resistance in breast cancer

Abstract P6-20-17: Targeting SHP2 for the treatment of HER2-positive breast cancer

The Green Bank North Celestial Cap Pulsar Survey. V. Pulsar Census and Survey Sensitivity

The Green Bank Northern Celestial Cap Pulsar Survey. VI. Discovery and Timing of PSR J1759+5036: A Double Neutron Star Binary Pulsar

Concomitant activation of the PI3K-Akt and the Ras-ERK signaling pathways is essential for transformation by the V-SEA tyrosine kinase oncogene

Development of an Efficient “Substrate-trapping” Mutant of Src Homology Phosphotyrosine Phosphatase 2 and Identification of the Epidermal Growth Factor Receptor, Gab1, and Three Other Proteins as Target Substrates

P-43 Correlation of mesothelin expression with recurrence in colorectal cancer (CRC) patients

The phosphotyrosine phosphatase SHP2 is a critical mediator of transformation induced by the oncogenic fibroblast growth factor receptor 3

ATP-stimulated smooth muscle cell proliferation requires independent ERK and PI3K signaling pathways

Molecular Mechanism for a Role of SHP2 in Epidermal Growth Factor Receptor Signaling

Characterization of a new monoclonal antibody to triplex DNA and immunofluorescent staining of mammalian chromosomes

Triplex DNA in the nucleus: direct binding of triplex-specific antibodies and their effect on transcription, replication and cell growth

Modulation of -catenin Tyr phosphorylation by SHP2 positively effects cell transformation induced by the constitutively active FGFR3

Molecular mechanisms of ATP and insulin synergistic stimulation of coronary artery smooth muscle growth

P2-02-05: Molecular Mechanism for Src Homology Phosphotyrosyl Phosphatase 2 Regulation of Cell Motility and Migration

P3-01-15: The Role of Src Homology Phosphotyrosyl Phosphatase-2 in Basal-Type/Triple-Negative Breast Cancer – Implications for Targeted Therapy

A Rho-associated Protein Kinase, ROKα, Binds Insulin Receptor Substrate-1 and Modulates Insulin Signaling

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