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The YAP1 Signaling Inhibitors, Verteporfin and CA3, Suppress the Mesothelioma Cancer Stem Cell Phenotype

Molecular cancer research, 2020-03, Vol.18 (3), p.343-351

2020

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Inhibition of YAP function overcomes BRAF inhibitor resistance in melanoma cancer stem cells

Oncotarget, 2017-12, Vol.8 (66), p.110257-110272

2017

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ACTL6A suppresses p21Cip1 expression to enhance the epidermal squamous cell carcinoma phenotype

Oncogene, 2020-09, Vol.39 (36), p.5855-5866

2020

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Transglutaminase 2 Is a Direct Target Gene of YAP-TAZ-Response

Cancer research (Chicago, Ill.), 2017-09, Vol.77 (17), p.4736-4736

2017

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ACTL6A suppresses p21Cip1 tumor suppressor expression to maintain an aggressive mesothelioma cancer cell phenotype

Oncogenesis (New York, NY), 2021-10, Vol.10 (10), p.70-70, Article 70

2021

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Cell-Impermeable Inhibitors Confirm That Intracellular Human Transglutaminase 2 Is Responsible for the Transglutaminase-Associated Cancer Phenotype

International journal of molecular sciences, 2023-08, Vol.24 (16), p.12546

2023

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Identification of a population of epidermal squamous cell carcinoma cells with enhanced potential for tumor formation

PloS one, 2013-12, Vol.8 (12), p.e84324

2013

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C-Jun NH2 terminal kinase (JNK) is an essential mediator of Toll-like receptor 2-induced corneal inflammation

Journal of leukocyte biology, 2008-04, Vol.83 (4), p.991-997

2008

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Polycomb Group Proteins Are Key Regulators of Keratinocyte Function

Journal of investigative dermatology, 2011-02, Vol.131 (2), p.295-301

2011

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Loss of epidermal AP1 transcription factor function reduces filaggrin level, alters chemokine expression and produces an ichthyosis-related phenotype

Cell death & disease, 2017-06, Vol.8 (6), p.e2840-e2840

2017

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Inhibition of corneal inflammation by liposomal delivery of short-chain, C-6 ceramide

Journal of leukocyte biology, 2008-06, Vol.83 (6), p.1512-1521

2008

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Expression of Bmi-1 in Epidermis Enhances Cell Survival by Altering Cell Cycle Regulatory Protein Expression and Inhibiting Apoptosis

Journal of investigative dermatology, 2008-01, Vol.128 (1), p.9-17

2008

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Role of oxidative stress in intermittent hypoxia-induced immediate early gene activation in rat PC12 cells

The Journal of physiology, 2004-06, Vol.557 (3), p.773-783

2004

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AP1 Transcription Factors in Epidermal Differentiation and Skin Cancer

Journal of Skin Cancer, 2013-01, Vol.2013 (2013), p.107-115

2013

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Suppression of AP1 transcription factor function in keratinocyte suppresses differentiation

PloS one, 2012-05, Vol.7 (5), p.e36941

2012

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Toll-like Receptors at the Ocular Surface

The ocular surface, 2008-07, Vol.6 (3), p.108-116

2008

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Pericentrosomal Localization of the TIG3 Tumor Suppressor Requires an N-Terminal Hydrophilic Region Motif

Journal of investigative dermatology, 2014-05, Vol.134 (5), p.1220-1229

2014

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Sulforaphane Induction of p21Cip1 Cyclin-dependent Kinase Inhibitor Expression Requires p53 and Sp1 Transcription Factors and Is p53-dependent

The Journal of biological chemistry, 2012-05, Vol.287 (20), p.16168-16178

2012

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NRP-1 interacts with GIPC1 and α6/β4-integrins to increase YAP1/∆Np63α-dependent epidermal cancer stem cell survival

Oncogene, 2018-08, Vol.37 (34), p.4711-4722

2018

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Abstract LB215: Transglutaminase 2 maintains hepatocyte growth factor signaling to enhance the epidermal cancer stem cell phenotype

Cancer research (Chicago, Ill.), 2021-07, Vol.81 (13_Supplement), p.LB215-LB215

2021

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NRP-1 interacts with GIPC1 and [alpha]6/[beta]4-integrins to increase YAP1/[increment]Np63[alpha]-dependent epidermal cancer stem cell survival

Oncogene, 2018-08, Vol.37 (34), p.4711

2018

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NRP‐1 interacts with GIPC1 and SYX to activate p38 MAPK signaling and cancer stem cell survival

Molecular carcinogenesis, 2019-04, Vol.58 (4), p.488-499

2019

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NRP-1 interacts with GIPC1 and α6/β4-integrins to increase YAP1/ΔNp63α-dependent epidermal cancer stem cell survival

Oncogene, 2018-05, Vol.37 (34), p.4711-4722

2018

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VGLL4 inhibits YAP1/TEAD signaling to suppress the epidermal squamous cell carcinoma cancer phenotype

Molecular carcinogenesis, 2021-07, Vol.60 (7), p.497-507

2021

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The YAP1 Signaling Inhibitors, Verteporfin and CA3, Suppress the Mesothelioma Cancer Stem Cell Phenotype

Inhibition of YAP function overcomes BRAF inhibitor resistance in melanoma cancer stem cells

ACTL6A suppresses p21Cip1 expression to enhance the epidermal squamous cell carcinoma phenotype

Transglutaminase 2 Is a Direct Target Gene of YAP-TAZ-Response

ACTL6A suppresses p21Cip1 tumor suppressor expression to maintain an aggressive mesothelioma cancer cell phenotype

Cell-Impermeable Inhibitors Confirm That Intracellular Human Transglutaminase 2 Is Responsible for the Transglutaminase-Associated Cancer Phenotype

Comprehensive molecular profiling of UV-induced metastatic melanoma in Nme1/Nme2-deficient mice reveals novel markers of survival in human patients

Identification of a population of epidermal squamous cell carcinoma cells with enhanced potential for tumor formation

C-Jun NH2 terminal kinase (JNK) is an essential mediator of Toll-like receptor 2-induced corneal inflammation

Polycomb Group Proteins Are Key Regulators of Keratinocyte Function

Loss of epidermal AP1 transcription factor function reduces filaggrin level, alters chemokine expression and produces an ichthyosis-related phenotype

Inhibition of corneal inflammation by liposomal delivery of short-chain, C-6 ceramide

Expression of Bmi-1 in Epidermis Enhances Cell Survival by Altering Cell Cycle Regulatory Protein Expression and Inhibiting Apoptosis

Role of oxidative stress in intermittent hypoxia-induced immediate early gene activation in rat PC12 cells

AP1 Transcription Factors in Epidermal Differentiation and Skin Cancer

Suppression of AP1 transcription factor function in keratinocyte suppresses differentiation

Toll-like Receptors at the Ocular Surface

Pericentrosomal Localization of the TIG3 Tumor Suppressor Requires an N-Terminal Hydrophilic Region Motif

Sulforaphane Induction of p21Cip1 Cyclin-dependent Kinase Inhibitor Expression Requires p53 and Sp1 Transcription Factors and Is p53-dependent

NRP-1 interacts with GIPC1 and α6/β4-integrins to increase YAP1/∆Np63α-dependent epidermal cancer stem cell survival

Abstract LB215: Transglutaminase 2 maintains hepatocyte growth factor signaling to enhance the epidermal cancer stem cell phenotype

NRP-1 interacts with GIPC1 and [alpha]6/[beta]4-integrins to increase YAP1/[increment]Np63[alpha]-dependent epidermal cancer stem cell survival

NRP‐1 interacts with GIPC1 and SYX to activate p38 MAPK signaling and cancer stem cell survival

NRP-1 interacts with GIPC1 and α6/β4-integrins to increase YAP1/ΔNp63α-dependent epidermal cancer stem cell survival

VGLL4 inhibits YAP1/TEAD signaling to suppress the epidermal squamous cell carcinoma cancer phenotype

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