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Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo

Biochimica et biophysica acta, 2010-06, Vol.1797 (6-7), p.1281-1291

2010

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The Expression Level of the Voltage-Dependent Anion Channel Controls Life and Death of the Cell

Proceedings of the National Academy of Sciences - PNAS, 2006-04, Vol.103 (15), p.5787-5792

2006

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Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase

Biochimica et biophysica acta, 2009-05, Vol.1787 (5), p.421-430

2009

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Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALS

Proceedings of the National Academy of Sciences - PNAS, 2016-09, Vol.113 (36), p.10198-10203

2016

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Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALS

International journal of molecular sciences, 2022-09, Vol.23 (17), p.9946

2022

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Macrophage Migration Inhibitory Factor as a Chaperone Inhibiting Accumulation of Misfolded SOD1

Neuron (Cambridge, Mass.), 2015-04, Vol.86 (1), p.218-232

2015

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VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins

Journal of cell science, 2009-06, Vol.122 (11), p.1906-1916

2009

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MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALS

Cell death & disease, 2018-01, Vol.9 (2), p.107-14, Article 107

2018

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A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALS

Frontiers in cellular neuroscience, 2019-08, Vol.13, p.346

2019

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Localization of the voltage-dependent anion channel-1 Ca2+ -binding sites

Cell calcium (Edinburgh), 2007-03, Vol.41 (3), p.235-244

2007

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An N-terminal nucleotide-binding site in VDAC1: Involvement in regulating mitochondrial function

Journal of cellular physiology, 2007-08, Vol.212 (2), p.551-561

2007

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Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1

Cell calcium (Edinburgh), 2008-02, Vol.43 (2), p.196-204

2008

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Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1

The Journal of biological chemistry, 2008-05, Vol.283 (19), p.13482-13490

2008

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In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death

Biochemical journal, 2004-01, Vol.377 (Pt 2), p.347-355

2004

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Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1: MAPPING THE SITE OF BINDING

The Journal of biological chemistry, 2008-05, Vol.283 (19), p.13482-13490

2008

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Fluoxetine (Prozac) interaction with the mitochondrial voltage-dependent anion channel and protection against apoptotic cell death

FEBS letters, 2005-09, Vol.579 (22), p.5105-5110

2005

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Misfolded SOD1 Accumulation and Mitochondrial Association Contribute to the Selective Vulnerability of Motor Neurons in Familial ALS: Correlation to Human Disease

ACS chemical neuroscience, 2017-10, Vol.8 (10), p.2225-2234

2017

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Macrophage migration inhibitory factor as a component of selective vulnerability of motor neurons in ALS

Rare diseases (Austin, Tex.), 2015-01, Vol.3 (1), p.e1061164-e1061164

2015

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Dominant-negative VDAC1 mutants reveal oligomeric VDAC1 to be the active unit in mitochondria-mediated apoptosis

Biochemical journal, 2010-07, Vol.429 (1), p.147-155

2010

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Localization of the voltage-dependent anion channel-1 Ca super(2+)-binding sites

Cell calcium (Edinburgh), 2007-03, Vol.41 (3), p.235-244

2007

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Localization of the voltage-dependent anion channel-1 Ca 2+-binding sites

Cell calcium (Edinburgh), 2007, Vol.41 (3), p.235-244

2007

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A Photoactivable Probe for Calcium Binding Proteins

Chemistry & biology, 2005-11, Vol.12 (11), p.1169-1178

2005

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Identifying barriers and facilitators of translating research evidence into clinical practice: A systematic review of reviews

Health & social care in the community, 2022-11, Vol.30 (6), p.e3265-e3276

2022

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Hospital centered surveillance of births in Gaza, Palestine, 2011–2017 and heavy metal contamination of the mothers reveals long-term impact of wars

Reproductive toxicology (Elmsford, N.Y.), 2019-06, Vol.86, p.23-32

2019

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Gaza Midwives’ Lived Experience of Providing Maternity Care During COVID-19

European journal of midwifery, 2022-08, Vol.6 (August), p.1-6

2022

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Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo

The Expression Level of the Voltage-Dependent Anion Channel Controls Life and Death of the Cell

Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase

Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALS

Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALS

Macrophage Migration Inhibitory Factor as a Chaperone Inhibiting Accumulation of Misfolded SOD1

VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins

MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALS

A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALS

Localization of the voltage-dependent anion channel-1 Ca2+ -binding sites

An N-terminal nucleotide-binding site in VDAC1: Involvement in regulating mitochondrial function

Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1

Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1

In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death

Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1: MAPPING THE SITE OF BINDING

Fluoxetine (Prozac) interaction with the mitochondrial voltage-dependent anion channel and protection against apoptotic cell death

Misfolded SOD1 Accumulation and Mitochondrial Association Contribute to the Selective Vulnerability of Motor Neurons in Familial ALS: Correlation to Human Disease

Macrophage migration inhibitory factor as a component of selective vulnerability of motor neurons in ALS

Dominant-negative VDAC1 mutants reveal oligomeric VDAC1 to be the active unit in mitochondria-mediated apoptosis

Localization of the voltage-dependent anion channel-1 Ca super(2+)-binding sites

Localization of the voltage-dependent anion channel-1 Ca 2+-binding sites

A Photoactivable Probe for Calcium Binding Proteins

Identifying barriers and facilitators of translating research evidence into clinical practice: A systematic review of reviews

Hospital centered surveillance of births in Gaza, Palestine, 2011–2017 and heavy metal contamination of the mothers reveals long-term impact of wars

Gaza Midwives’ Lived Experience of Providing Maternity Care During COVID-19

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