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Exposure of Hematopoietic Stem Cells to Benzene or 1,4‐Benzoquinone Induces Gender‐Specific Gene Expression

Stem cells (Dayton, Ohio), 2004-09, Vol.22 (5), p.750-758

2004

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Human CD34+ Hematopoietic Progenitor Cells Are Sensitive Targets for Toxicity Induced by 1,4-Benzoquinone

Toxicological sciences, 2004-05, Vol.79 (1), p.82-89

2004

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Genetic susceptibility to benzene-induced toxicity: role of NADPH: quinone oxidoreductase-1

Cancer research (Chicago, Ill.), 2003-03, Vol.63 (5), p.929

2003

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Male Mice Deficient in Microsomal Epoxide Hydrolase Are Not Susceptible to Benzene-Induced Toxicity

Toxicological sciences, 2003-04, Vol.72 (2), p.201-209

2003

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Variations in Prkdc and Susceptibility to Benzene-Induced Toxicity in Mice

Toxicological sciences, 2003-10, Vol.75 (2), p.321-332

2003

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Potential role of treatment artifact in the effect of cell density upon frequencies of C3H/10T1/2 cell transformation

Cancer research (Chicago, Ill.), 1985-12, Vol.45 (12 Pt 1), p.6314

1985

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Genetic susceptibility to benzene-induced toxicity: Role of NADHP: Quinone oxidoreductase-1

Cancer research (Chicago, Ill.), 2003, Vol.63 (5), p.929-935

2003

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Effect of growth state, tumor promoters, and transformation upon intercellular communication between C3H/10T1/2 murine fibroblasts

Carcinogenesis (New York), 1989-01, Vol.10 (1), p.113-121

1989

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Promotion of C3H/10T1/2 morphological transformation by polychlorinated dibenzo-p-dioxin isomers

Carcinogenesis (New York), 1987-10, Vol.8 (10), p.1485-1490

1987

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2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) promotes the transformation of C3H/10T1/2 cells

Carcinogenesis (New York), 1985-04, Vol.6 (4), p.651-653

1985

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Reversible expression of morphological transformation in C3H/10T1/2 mouse embryo cultures exposed to 12-O-tetradecanoylphorbol-13-acetate

Carcinogenesis (New York), 1986-11, Vol.7 (11), p.1793-1796

1986

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Alterations of intercellular communication associated with the transformation of C3H/10T1/2 cells

Carcinogenesis (New York), 1987-02, Vol.8 (2), p.321-325

1987

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Effect of mouse skin tumor promoters upon [3H]uridine exchange and focus formation in cultures of C3H/10T1/2 mouse fibroblasts

Carcinogenesis (New York), 1986-07, Vol.7 (7), p.1095-1099

1986

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Factors influencing the promotion of transformation in chemically-initiated C3H/10T1/2 Cl 8 mouse embryo fibroblasts

Carcinogenesis (New York), 1983, Vol.4 (6), p.709-715

1983

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Cytotoxicity and mutagenicity of dinitrotoluenes in Chinese hamster ovary cells

Mutation research. Mutation research letters, 1982, Vol.103 (1), p.53-59

1982

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The effect of biotransformation of 2,4-dinitrotoluene on its mutagenic potential

Mutagenesis, 1987-11, Vol.2 (6), p.415-418

1987

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Weak promotion of C3H/10T1/2 cell transformation by repeated treatments with formaldehyde

Cancer research (Chicago, Ill.), 1983-07, Vol.43 (7), p.3236

1983

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Initiation of C3H/10T1/2 cell transformation by N-methyl-N′ -nitro-N-nitrosoguanidine and aflatoxin B1

Carcinogenesis (New York), 1982, Vol.3 (4), p.391-395

1982

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Initiation of C3H/10T1/2 cell transformation by N-methyl-N′ -nitro-N-nitrosoguanidine and aflatoxin B 1

Carcinogenesis (New York), 1982, Vol.3 (4), p.391-395

1982

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Toxicity of Platinum (IV) Salts for Cells of Pulmonary Origin

Environmental health perspectives, 1975-12, Vol.12, p.45-56

1975

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Enhanced sensitivity of the C3H/10T1/2 cell transformation system to alkylating and chemotherapeutic agents by treatment with 12-O-tetradecanoylphorbol-13-acetate

Environmental mutagenesis, 1984, Vol.6 (1), p.81-89

1984

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The mutagenicity of dinitrotoluenes in Salmonella typhimurium

Mutation Research/Genetic Toxicology, 1981-12, Vol.90 (4), p.373-383

1981

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Development of the National Cancer Institute's patient-reported outcomes version of the common terminology criteria for adverse events (PRO-CTCAE)

JNCI : Journal of the National Cancer Institute, 2014-09, Vol.106 (9), p.dju244-dju244

2014

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American Society of Clinical Oncology Provisional Clinical Opinion: The Integration of Palliative Care Into Standard Oncology Care

Journal of clinical oncology, 2012-03, Vol.30 (8), p.880-887

2012

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Exposure of Hematopoietic Stem Cells to Benzene or 1,4‐Benzoquinone Induces Gender‐Specific Gene Expression

Human CD34+ Hematopoietic Progenitor Cells Are Sensitive Targets for Toxicity Induced by 1,4-Benzoquinone

Genetic susceptibility to benzene-induced toxicity: role of NADPH: quinone oxidoreductase-1

Male Mice Deficient in Microsomal Epoxide Hydrolase Are Not Susceptible to Benzene-Induced Toxicity

Variations in Prkdc and Susceptibility to Benzene-Induced Toxicity in Mice

Potential role of treatment artifact in the effect of cell density upon frequencies of C3H/10T1/2 cell transformation

Genetic susceptibility to benzene-induced toxicity: Role of NADHP: Quinone oxidoreductase-1

Effect of growth state, tumor promoters, and transformation upon intercellular communication between C3H/10T1/2 murine fibroblasts

Promotion of C3H/10T1/2 morphological transformation by polychlorinated dibenzo-p-dioxin isomers

2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) promotes the transformation of C3H/10T1/2 cells

Reversible expression of morphological transformation in C3H/10T1/2 mouse embryo cultures exposed to 12-O-tetradecanoylphorbol-13-acetate

Alterations of intercellular communication associated with the transformation of C3H/10T1/2 cells

Effect of mouse skin tumor promoters upon [3H]uridine exchange and focus formation in cultures of C3H/10T1/2 mouse fibroblasts

Factors influencing the promotion of transformation in chemically-initiated C3H/10T1/2 Cl 8 mouse embryo fibroblasts

Cytotoxicity and mutagenicity of dinitrotoluenes in Chinese hamster ovary cells

The effect of biotransformation of 2,4-dinitrotoluene on its mutagenic potential

Weak promotion of C3H/10T1/2 cell transformation by repeated treatments with formaldehyde

Initiation of C3H/10T1/2 cell transformation by N-methyl-N′ -nitro-N-nitrosoguanidine and aflatoxin B1

Initiation of C3H/10T1/2 cell transformation by N-methyl-N′ -nitro-N-nitrosoguanidine and aflatoxin B 1

Toxicity of Platinum (IV) Salts for Cells of Pulmonary Origin

Enhanced sensitivity of the C3H/10T1/2 cell transformation system to alkylating and chemotherapeutic agents by treatment with 12-O-tetradecanoylphorbol-13-acetate

The mutagenicity of dinitrotoluenes in Salmonella typhimurium

Development of the National Cancer Institute's patient-reported outcomes version of the common terminology criteria for adverse events (PRO-CTCAE)

American Society of Clinical Oncology Provisional Clinical Opinion: The Integration of Palliative Care Into Standard Oncology Care

Validity and Reliability of the US National Cancer Institute's Patient-Reported Outcomes Version of the Common Terminology Criteria for Adverse Events (PRO-CTCAE)

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