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Sigmar1’s Molecular, Cellular, and Biological Functions in Regulating Cellular Pathophysiology

Frontiers in physiology, 2021-07, Vol.12, p.705575-705575

2021

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Sigmar1 regulates endoplasmic reticulum stress-induced C/EBP-homologous protein expression in cardiomyocytes

Bioscience reports, 2017-08, Vol.37 (4)

2017

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Pleiotropic effects of mdivi-1 in altering mitochondrial dynamics, respiration, and autophagy in cardiomyocytes

Redox biology, 2020-09, Vol.36, p.101660-101660, Article 101660

2020

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Diastolic dysfunction in Alzheimer’s disease model mice is associated with Aβ-amyloid aggregate formation and mitochondrial dysfunction

Scientific reports, 2024-07, Vol.14 (1), p.16715-19, Article 16715

2024

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Cardiac Dysfunction in the Sigma 1 Receptor Knockout Mouse Associated With Impaired Mitochondrial Dynamics and Bioenergetics

Journal of the American Heart Association, 2018-10, Vol.7 (20), p.e009775

2018

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Sigmar1 ablation leads to lung pathological changes associated with pulmonary fibrosis, inflammation, and altered surfactant proteins levels

Frontiers in physiology, 2023-03, Vol.14, p.1118770-1118770

2023

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Deletion of Sigmar1 leads to increased arterial stiffness and altered mitochondrial respiration resulting in vascular dysfunction

Frontiers in physiology, 2024-04, Vol.15, p.1386296-1386296

2024

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Dysfunctional Mitochondrial Dynamic and Oxidative Phosphorylation Precedes Cardiac Dysfunction in R120G-αB-Crystallin-Induced Desmin-Related Cardiomyopathy

Journal of the American Heart Association, 2020-12, Vol.9 (23), p.e017195-e017195

2020

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Molecular Perspectives of Mitochondrial Adaptations and Their Role in Cardiac Proteostasis

Frontiers in physiology, 2020-08, Vol.11, p.1054-1054

2020

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Aberrant Mitochondrial Fission Is Maladaptive in Desmin Mutation-Induced Cardiac Proteotoxicity

Journal of the American Heart Association, 2018-07, Vol.7 (14)

2018

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Neurogranin regulates eNOS function and endothelial activation

Redox biology, 2020-07, Vol.34, p.101487, Article 101487

2020

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Mitochondrial dysfunction and autophagy activation are associated with cardiomyopathy developed by extended methamphetamine self-administration in rats

Redox biology, 2022-12, Vol.58, p.102523, Article 102523

2022

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Methamphetamine causes cardiovascular dysfunction via cystathionine gamma lyase and hydrogen sulfide depletion

Redox biology, 2022-11, Vol.57, p.102480, Article 102480

2022

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Pathological Sequelae Associated with Skeletal Muscle Atrophy and Histopathology in G93ASOD1 Mice

Muscles, 2023-03, Vol.2 (1), p.51-74

2023

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Targeted deletion of T‐cell S1P receptor 1 ameliorates cardiac fibrosis in streptozotocin‐induced diabetic mice

The FASEB journal, 2018-10, Vol.32 (10), p.5426-5435

2018

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Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration

Scientific reports, 2019-02, Vol.9 (1), p.2002, Article 2002

2019

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Depletion of T lymphocytes ameliorates cardiac fibrosis in streptozotocin-induced diabetic cardiomyopathy

International immunopharmacology, 2016-10, Vol.39, p.251-264

2016

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Inhibition of PKC-[theta] preserves cardiac function and reduces fibrosis in streptozotocin-induced diabetic cardiomyopathy

British journal of pharmacology, 2014-06, Vol.171 (11), p.2913

2014

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Monitoring Mitochondrial Morphology and Respiration in Doxorubicin-Induced Cardiomyopathy

Methods in molecular biology (Clifton, N.J.), 2022, Vol.2497, p.207

2022

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Chemical Architecture of Block Copolymers Differentially Abrogate Cardiotoxicity and Maintain the Anticancer Efficacy of Doxorubicin

Molecular pharmaceutics, 2020-12, Vol.17 (12), p.4676-4690

2020

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Molecular Characterization of Skeletal Muscle Dysfunction in Sigma 1 Receptor (Sigmar1) Knockout Mice

The American journal of pathology, 2022-01, Vol.192 (1), p.160-177

2022

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Inhibition of PKC‐θ preserves cardiac function and reduces fibrosis in streptozotocin‐induced diabetic cardiomyopathy

British journal of pharmacology, 2014-06, Vol.171 (11), p.2913-2924

2014

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Visualizing Subcellular Localization of a Protein in the Heart using Quantum Dots-Mediated Immuno-Labeling followed by Transmission Electron Microscopy

Journal of visualized experiments, 2022-09 (187)

2022

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Sigmar1 Deficiency Results in Pulmonary Fibrosis Associated with Increased Pulmonary Inflammation and Surfactant Protein Levels in Mice Lung

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Neurogranin regulates calcium-dependent cardiac hypertrophy

Experimental and molecular pathology, 2022-08, Vol.127, p.104815, Article 104815

2022

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Sigmar1’s Molecular, Cellular, and Biological Functions in Regulating Cellular Pathophysiology

Sigmar1 regulates endoplasmic reticulum stress-induced C/EBP-homologous protein expression in cardiomyocytes

Pleiotropic effects of mdivi-1 in altering mitochondrial dynamics, respiration, and autophagy in cardiomyocytes

Diastolic dysfunction in Alzheimer’s disease model mice is associated with Aβ-amyloid aggregate formation and mitochondrial dysfunction

Cardiac Dysfunction in the Sigma 1 Receptor Knockout Mouse Associated With Impaired Mitochondrial Dynamics and Bioenergetics

Sigmar1 ablation leads to lung pathological changes associated with pulmonary fibrosis, inflammation, and altered surfactant proteins levels

Deletion of Sigmar1 leads to increased arterial stiffness and altered mitochondrial respiration resulting in vascular dysfunction

Dysfunctional Mitochondrial Dynamic and Oxidative Phosphorylation Precedes Cardiac Dysfunction in R120G-αB-Crystallin-Induced Desmin-Related Cardiomyopathy

Molecular Perspectives of Mitochondrial Adaptations and Their Role in Cardiac Proteostasis

Aberrant Mitochondrial Fission Is Maladaptive in Desmin Mutation-Induced Cardiac Proteotoxicity

Neurogranin regulates eNOS function and endothelial activation

Mitochondrial dysfunction and autophagy activation are associated with cardiomyopathy developed by extended methamphetamine self-administration in rats

Methamphetamine causes cardiovascular dysfunction via cystathionine gamma lyase and hydrogen sulfide depletion

Pathological Sequelae Associated with Skeletal Muscle Atrophy and Histopathology in G93ASOD1 Mice

Targeted deletion of T‐cell S1P receptor 1 ameliorates cardiac fibrosis in streptozotocin‐induced diabetic mice

Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration

Depletion of T lymphocytes ameliorates cardiac fibrosis in streptozotocin-induced diabetic cardiomyopathy

Inhibition of PKC-[theta] preserves cardiac function and reduces fibrosis in streptozotocin-induced diabetic cardiomyopathy

Monitoring Mitochondrial Morphology and Respiration in Doxorubicin-Induced Cardiomyopathy

Chemical Architecture of Block Copolymers Differentially Abrogate Cardiotoxicity and Maintain the Anticancer Efficacy of Doxorubicin

Molecular Characterization of Skeletal Muscle Dysfunction in Sigma 1 Receptor (Sigmar1) Knockout Mice

Inhibition of PKC‐θ preserves cardiac function and reduces fibrosis in streptozotocin‐induced diabetic cardiomyopathy

Visualizing Subcellular Localization of a Protein in the Heart using Quantum Dots-Mediated Immuno-Labeling followed by Transmission Electron Microscopy

Sigmar1 Deficiency Results in Pulmonary Fibrosis Associated with Increased Pulmonary Inflammation and Surfactant Protein Levels in Mice Lung

Neurogranin regulates calcium-dependent cardiac hypertrophy

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