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Acute hydroxyurea-induced replication blockade results in replisome components disengagement from nascent DNA without causing fork collapse

Cellular and molecular life sciences : CMLS, 2020-02, Vol.77 (4), p.735-749

2020

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RAD51 is a druggable target that sustains replication fork progression upon DNA replication stress

PloS one, 2022-08, Vol.17 (8), p.e0266645-e0266645

2022

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Modeling and subtleties of K-Ras and Calmodulin interaction

PLoS computational biology, 2018-10, Vol.14 (10), p.e1006552-e1006552

2018

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Near‐tetraploid cancer cells show chromosome instability triggered by replication stress and exhibit enhanced invasiveness

The FASEB journal, 2018-07, Vol.32 (7), p.3502-3517

2018

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Pleiotropic Roles of Calmodulin in the Regulation of KRas and Rac1 GTPases: Functional Diversity in Health and Disease

International journal of molecular sciences, 2020-05, Vol.21 (10), p.3680

2020

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Phosphorylation at Ser-181 of Oncogenic KRAS Is Required for Tumor Growth

Cancer research (Chicago, Ill.), 2014-02, Vol.74 (4), p.1190-1199

2014

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KRAS phosphorylation regulates cell polarization and tumorigenic properties in colorectal cancer

Oncogene, 2021-09, Vol.40 (38), p.5730-5740

2021

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Peptidomimetics designed to bind to RAS effector domain are promising cancer therapeutic compounds

Scientific reports, 2022-09, Vol.12 (1), p.15810-15810, Article 15810

2022

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α4-α5 Helices on Surface of KRAS Can Accommodate Small Compounds That Increase KRAS Signaling While Inducing CRC Cell Death

International journal of molecular sciences, 2023-01, Vol.24 (1), p.748

2023

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clathrin-dependent pathway leads to KRas signaling on late endosomes en route to lysosomes

The Journal of cell biology, 2009-03, Vol.184 (6), p.863-879

2009

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Modulation of the Ras/Raf/MEK/ERK pathway by Ca2+, and Calmodulin

Cellular signalling, 2002-08, Vol.14 (8), p.649-654

2002

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SUMO regulates p21Cip1 intracellular distribution and with p21Cip1 facilitates multiprotein complex formation in the nucleolus upon DNA damage

PloS one, 2017-06, Vol.12 (6), p.e0178925-e0178925

2017

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p21 as a transcriptional co-repressor of S-phase and mitotic control genes

PloS one, 2012-05, Vol.7 (5), p.e37759-e37759

2012

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Proteasome inhibition reduces proliferation, collagen expression, and inflammatory cytokine production in nasal mucosa and polyp fibroblasts

The Journal of pharmacology and experimental therapeutics, 2012-10, Vol.343 (1), p.184-197

2012

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The stress-activated protein kinases p38α/β and JNK1/2 cooperate with Chk1 to inhibit mitotic entry upon DNA replication arrest

Cell cycle (Georgetown, Tex.), 2012-10, Vol.11 (19), p.3627-3637

2012

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Identification of Essential Interacting Elements in K-Ras/Calmodulin Binding and Its Role in K-Ras Localization

The Journal of biological chemistry, 2008-04, Vol.283 (16), p.10621-10631

2008

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Ribonucleoprotein HNRNPA2B1 Interacts With and Regulates Oncogenic KRAS in Pancreatic Ductal Adenocarcinoma Cells

Gastroenterology (New York, N.Y. 1943), 2014-10, Vol.147 (4), p.882-892.e8

2014

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New origin firing is inhibited by APC/CCdh1 activation in S-phase after severe replication stress

Nucleic acids research, 2016-06, Vol.44 (10), p.4745

2016

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Calmodulin Binds to K-Ras, but Not to H- or N-Ras, and Modulates Its Downstream Signaling

Molecular and Cellular Biology, 2001-11, Vol.21 (21), p.7345-7354

2001

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The SET Protein Regulates G2/M Transition by Modulating Cyclin B-Cyclin-dependent Kinase 1 Activity

The Journal of biological chemistry, 2003-01, Vol.278 (2), p.1158-1164

2003

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Oncogenic K-ras segregates at spatially distinct plasma membrane signaling platforms according to its phosphorylation status

Journal of cell science, 2013-10, Vol.126 (Pt 20), p.4553-4559

2013

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Calmodulin regulates intracellular trafficking of epidermal growth factor receptor and the MAPK signaling pathway

Molecular biology of the cell, 2002-06, Vol.13 (6), p.2057-2068

2002

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Fermented Wheat Germ Extract Inhibits Glycolysis/Pentose Cycle Enzymes and Induces Apoptosis through Poly(ADP-ribose) Polymerase Activation in Jurkat T-cell Leukemia Tumor Cells

The Journal of biological chemistry, 2002-11, Vol.277 (48), p.46408-46414

2002

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CaM interaction and Ser181 phosphorylation as new K-Ras signaling modulators

Small GTPases, 2011-03, Vol.2 (2), p.99-103

2011

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Disruption of the antiproliferative TGF-β signaling pathways in human pancreatic cancer cells

Oncogene, 1998-10, Vol.17 (15), p.1969-1978

1998

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Acute hydroxyurea-induced replication blockade results in replisome components disengagement from nascent DNA without causing fork collapse

RAD51 is a druggable target that sustains replication fork progression upon DNA replication stress

Modeling and subtleties of K-Ras and Calmodulin interaction

Near‐tetraploid cancer cells show chromosome instability triggered by replication stress and exhibit enhanced invasiveness

Pleiotropic Roles of Calmodulin in the Regulation of KRas and Rac1 GTPases: Functional Diversity in Health and Disease

Phosphorylation at Ser-181 of Oncogenic KRAS Is Required for Tumor Growth

KRAS phosphorylation regulates cell polarization and tumorigenic properties in colorectal cancer

Peptidomimetics designed to bind to RAS effector domain are promising cancer therapeutic compounds

α4-α5 Helices on Surface of KRAS Can Accommodate Small Compounds That Increase KRAS Signaling While Inducing CRC Cell Death

clathrin-dependent pathway leads to KRas signaling on late endosomes en route to lysosomes

Modulation of the Ras/Raf/MEK/ERK pathway by Ca2+, and Calmodulin

SUMO regulates p21Cip1 intracellular distribution and with p21Cip1 facilitates multiprotein complex formation in the nucleolus upon DNA damage

p21 as a transcriptional co-repressor of S-phase and mitotic control genes

Proteasome inhibition reduces proliferation, collagen expression, and inflammatory cytokine production in nasal mucosa and polyp fibroblasts

The stress-activated protein kinases p38α/β and JNK1/2 cooperate with Chk1 to inhibit mitotic entry upon DNA replication arrest

Identification of Essential Interacting Elements in K-Ras/Calmodulin Binding and Its Role in K-Ras Localization

Ribonucleoprotein HNRNPA2B1 Interacts With and Regulates Oncogenic KRAS in Pancreatic Ductal Adenocarcinoma Cells

New origin firing is inhibited by APC/CCdh1 activation in S-phase after severe replication stress

Calmodulin Binds to K-Ras, but Not to H- or N-Ras, and Modulates Its Downstream Signaling

The SET Protein Regulates G2/M Transition by Modulating Cyclin B-Cyclin-dependent Kinase 1 Activity

Oncogenic K-ras segregates at spatially distinct plasma membrane signaling platforms according to its phosphorylation status

Calmodulin regulates intracellular trafficking of epidermal growth factor receptor and the MAPK signaling pathway

Fermented Wheat Germ Extract Inhibits Glycolysis/Pentose Cycle Enzymes and Induces Apoptosis through Poly(ADP-ribose) Polymerase Activation in Jurkat T-cell Leukemia Tumor Cells

CaM interaction and Ser181 phosphorylation as new K-Ras signaling modulators

Disruption of the antiproliferative TGF-β signaling pathways in human pancreatic cancer cells

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