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The Biphasic Relationship between Regional Brain Senile Plaque and Neurofibrillary Tangle Distributions: Modification by Age, Sex, and APOE Polymorphism

Annals of the New York Academy of Sciences, 2004-06, Vol.1019 (1), p.24-28

2004

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Are cases with tau pathology occurring in the absence of Aβ deposits part of the AD-related pathological process?

Acta neuropathologica, 2014-12, Vol.128 (6), p.767-772

2014

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Histological correlates of postmortem ultra-high-resolution single-section MRI in cortical cerebral microinfarcts

Acta neuropathologica communications, 2020-03, Vol.8 (1), p.33-33, Article 33

2020

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Nerve cells immunoreactive for p62 in select hypothalamic and brainstem nuclei of controls and Parkinson’s disease cases

Journal of Neural Transmission, 2011-05, Vol.118 (5), p.809-819

2011

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Longitudinal Diffusion Tensor Imaging Resembles Patterns of Pathology Progression in Behavioral Variant Frontotemporal Dementia (bvFTD)

Frontiers in aging neuroscience, 2018-03, Vol.10, p.47-47

2018

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100 years of Lewy pathology

Nature reviews. Neurology, 2013-01, Vol.9 (1), p.13-24

2013

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Vulnerability of Select Neuronal Types to Alzheimer's Disease

Annals of the New York Academy of Sciences, 2000-12, Vol.924 (1), p.53-61

2000

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Development of alpha-synuclein immunoreactive astrocytes in the forebrain parallels stages of intraneuronal pathology in sporadic Parkinson's disease

Acta neuropathologica, 2007-09, Vol.114 (3), p.231-241

2007

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Potential Pathways of Abnormal Tau and α-Synuclein Dissemination in Sporadic Alzheimer's and Parkinson's Diseases

Cold Spring Harbor perspectives in biology, 2016-11, Vol.8 (11), p.a023630

2016

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Top-Down Projections Direct the Gradual Progression of Alzheimer-Related Tau Pathology Throughout the Neocortex

Advances in experimental medicine and biology, 2019, Vol.1184, p.291-303

2019

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Staging of Alzheimer-Related Cortical Destruction

International psychogeriatrics, 1997-12, Vol.9 (S1), p.257-261

1997

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High prevalence of thorn-shaped astrocytes in the aged human medial temporal lobe

Neurobiology of aging, 2004-03, Vol.25 (3), p.397-405

2004

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Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer’s disease

Acta neuropathologica, 2009-10, Vol.118 (4), p.475-485

2009

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Neuroanatomy and pathology of sporadic Parkinson's disease

Advances in anatomy, embryology and cell biology, 2009, Vol.201, p.1

2009

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Demonstration of Amyloid Deposits and Neurofibrillary Changes in Whole Brain Sections

Brain pathology (Zurich, Switzerland), 1991-04, Vol.1 (3), p.213-216

1991

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Staging of Alzheimer-related cortical destruction

European neurology, 1993, Vol.33 (6), p.403-408

1993

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α-Synuclein immunopositive Parkinson's disease-related inclusion bodies in lower brain stem nuclei

Acta neuropathologica, 2001-03, Vol.101 (3), p.195-201

2001

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Amyloid-β may be released from non-junctional varicosities of axons generated from abnormal tau-containing brainstem nuclei in sporadic Alzheimer’s disease: a hypothesis

Acta neuropathologica, 2013-08, Vol.126 (2), p.303-306

2013

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Cortical influences drive amyotrophic lateral sclerosis

Journal of neurology, neurosurgery and psychiatry, 2017-11, Vol.88 (11), p.917-924

2017

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Neurodegeneration in normal brain aging and disease

Science of aging knowledge environment, 2004-06, Vol.2004 (23), p.pe26

2004

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Anterior Cingulate Cortex TDP-43 Pathology in Sporadic Amyotrophic Lateral Sclerosis

Journal of neuropathology and experimental neurology, 2018-01, Vol.77 (1), p.74-83

2018

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Reply: the early pathological process in sporadic Alzheimer’s disease

Acta neuropathologica, 2013-10, Vol.126 (4), p.615-618

2013

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A timeline for Parkinson's disease

Parkinsonism & related disorders, 2010-02, Vol.16 (2), p.79-84

2010

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Pathological TDP-43 changes in Betz cells differ from those in bulbar and spinal α-motoneurons in sporadic amyotrophic lateral sclerosis

Acta neuropathologica, 2017-01, Vol.133 (1), p.79-90

2017

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Amyloid β-protein (Aβ)-containing astrocytes are located preferentially near N-terminal-truncated Aβ deposits in the human entorhinal cortex

Acta neuropathologica, 2000-12, Vol.100 (6), p.608-617

2000

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The Biphasic Relationship between Regional Brain Senile Plaque and Neurofibrillary Tangle Distributions: Modification by Age, Sex, and APOE Polymorphism

Are cases with tau pathology occurring in the absence of Aβ deposits part of the AD-related pathological process?

Histological correlates of postmortem ultra-high-resolution single-section MRI in cortical cerebral microinfarcts

Nerve cells immunoreactive for p62 in select hypothalamic and brainstem nuclei of controls and Parkinson’s disease cases

Longitudinal Diffusion Tensor Imaging Resembles Patterns of Pathology Progression in Behavioral Variant Frontotemporal Dementia (bvFTD)

100 years of Lewy pathology

Vulnerability of Select Neuronal Types to Alzheimer's Disease

Development of alpha-synuclein immunoreactive astrocytes in the forebrain parallels stages of intraneuronal pathology in sporadic Parkinson's disease

Potential Pathways of Abnormal Tau and α-Synuclein Dissemination in Sporadic Alzheimer's and Parkinson's Diseases

Top-Down Projections Direct the Gradual Progression of Alzheimer-Related Tau Pathology Throughout the Neocortex

Staging of Alzheimer-Related Cortical Destruction

High prevalence of thorn-shaped astrocytes in the aged human medial temporal lobe

Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer’s disease

Neuroanatomy and pathology of sporadic Parkinson's disease

Demonstration of Amyloid Deposits and Neurofibrillary Changes in Whole Brain Sections

Staging of Alzheimer-related cortical destruction

α-Synuclein immunopositive Parkinson's disease-related inclusion bodies in lower brain stem nuclei

Amyloid-β may be released from non-junctional varicosities of axons generated from abnormal tau-containing brainstem nuclei in sporadic Alzheimer’s disease: a hypothesis

Cortical influences drive amyotrophic lateral sclerosis

Neurodegeneration in normal brain aging and disease

Anterior Cingulate Cortex TDP-43 Pathology in Sporadic Amyotrophic Lateral Sclerosis

Reply: the early pathological process in sporadic Alzheimer’s disease

A timeline for Parkinson's disease

Pathological TDP-43 changes in Betz cells differ from those in bulbar and spinal α-motoneurons in sporadic amyotrophic lateral sclerosis

Amyloid β-protein (Aβ)-containing astrocytes are located preferentially near N-terminal-truncated Aβ deposits in the human entorhinal cortex

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