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Group 3 innate lymphocytes (ILC3s) upregulate IL-22 in response to elevated intracellular cAMP levels

Cytokine (Philadelphia, Pa.), 2022-05, Vol.153, p.155862-155862, Article 155862

2022

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E3 Ubiquitin Ligase Von Hippel-Lindau Protein Promotes Th17 Differentiation

The Journal of immunology (1950), 2020-08, Vol.205 (4), p.1009-1023

2020

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The Polyamine Putrescine Is a Positive Regulator of Group 3 Innate Lymphocyte Activation

ImmunoHorizons, 2023-01, Vol.7 (1), p.41-48

2023

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Transcription Factor HIF-1α Controls Expression of the Cytokine IL-22 in CD4 T Cells

The Journal of immunology (1950), 2016-10, Vol.197 (7), p.2646-2652

2016

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Unraveling the Genetics of Autoimmunity

Cell, 2010-03, Vol.140 (6), p.791-797

2010

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Memory/effector (CD45RB(lo)) CD4 T cells are controlled directly by IL-10 and cause IL-22-dependent intestinal pathology

The Journal of experimental medicine, 2011-05, Vol.208 (5), p.1027-1040

2011

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IL-22 deficiency increases CD4 T cell responses to mucosal immunization

Vaccine, 2018-06, Vol.36 (25), p.3694-3700

2018

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Changes in Availability of Oxygen Accentuate Differences in Capsular Polysaccharide Expression by Phenotypic Variants and Clinical Isolates of Streptococcus pneumoniae

Infection and Immunity, 2001-09, Vol.69 (9), p.5430-5439

2001

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Listeria monocytogenes Phosphatidylinositol-Specific Phospholipase C Has Evolved for Virulence by Greatly Reduced Activity on GPI Anchors

Proceedings of the National Academy of Sciences - PNAS, 2005-09, Vol.102 (36), p.12927-12931

2005

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Phosphatidylinositol-Specific Phospholipase C of Bacillus anthracis Down-Modulates the Immune Response

The Journal of immunology (1950), 2005-06, Vol.174 (12), p.8011-8016

2005

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Characterization of Listeria monocytogenes Expressing Anthrolysin O and Phosphatidylinositol-Specific Phospholipase C from Bacillus anthracis

Infection and Immunity, 2005-10, Vol.73 (10), p.6639-6646

2005

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Hypoxic modulation of hepatocyte responses to the cytokine interleukin‐22

Immunology and cell biology, 2017-04, Vol.95 (4), p.380-387

2017

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Cutting Edge: CD4 and CD8 T Cells Are Intrinsically Different in Their Proliferative Responses

The Journal of immunology (1950), 2002-02, Vol.168 (4), p.1528-1532

2002

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The dual nature of TH17 cells: shifting the focus to function

Nature immunology, 2010-05, Vol.11 (6), p.471-476

2010

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Chronic Immunodeficiency in Mice Lacking RasGRP1 Results in CD4 T Cell Immune Activation and Exhaustion

Journal of Immunology, 2007-08, Vol.179 (4), p.2143-2152

2007

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RasGRP1 Regulates Antigen-Induced Developmental Programming by Naive CD8 T Cells

The Journal of immunology (1950), 2010-01, Vol.184 (2), p.666-676

2010

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Glucocorticoids inhibit group 3 innate lymphocyte IL-22 production1

The Journal of immunology (1950), 2018-07, Vol.201 (4), p.1267-1274

2018

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Glucocorticoids inhibit group 3 innate lymphocyte IL-22 production

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.43-43.1

2018

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Listeria monocytogenes virulence proteins induce surface expression of Fas ligand on T lymphocytes

Molecular microbiology, 2004-03, Vol.51 (5), p.1483-1492

2004

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Nonsecreted Bacterial Proteins Induce Recall CD8 T Cell Responses But Do Not Serve as Protective Antigens

The Journal of immunology (1950), 2002-11, Vol.169 (10), p.5805-5812

2002

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The transcription factor HIF1α controls expression of the cytokine IL-22 in CD4 T cells

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.120-120.3

2016

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Sugar-Coated Regulation of T Cells

Cell, 2005-07, Vol.122 (1), p.2-4

2005

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Activation of Antigen-Specific CD8 T Cells Results in Minimal Killing of Bystander Bacteria

The Journal of immunology (1950), 2003-12, Vol.171 (11), p.6032-6038

2003

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NLRP10 is a NOD―like receptor essential to initiate adaptive immunity by dendritic cells

Nature (London), 2012-04, Vol.484 (7395), p.510-513

2012

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Interleukin-22 deficiency alters colonic microbiota to be transmissible and colitogenic

The Journal of immunology (1950), 2013-04, Vol.190 (10), p.5306-5312

2013

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Group 3 innate lymphocytes (ILC3s) upregulate IL-22 in response to elevated intracellular cAMP levels

E3 Ubiquitin Ligase Von Hippel-Lindau Protein Promotes Th17 Differentiation

The Polyamine Putrescine Is a Positive Regulator of Group 3 Innate Lymphocyte Activation

Transcription Factor HIF-1α Controls Expression of the Cytokine IL-22 in CD4 T Cells

Unraveling the Genetics of Autoimmunity

Memory/effector (CD45RB(lo)) CD4 T cells are controlled directly by IL-10 and cause IL-22-dependent intestinal pathology

IL-22 deficiency increases CD4 T cell responses to mucosal immunization

Changes in Availability of Oxygen Accentuate Differences in Capsular Polysaccharide Expression by Phenotypic Variants and Clinical Isolates of Streptococcus pneumoniae

Listeria monocytogenes Phosphatidylinositol-Specific Phospholipase C Has Evolved for Virulence by Greatly Reduced Activity on GPI Anchors

Phosphatidylinositol-Specific Phospholipase C of Bacillus anthracis Down-Modulates the Immune Response

Characterization of Listeria monocytogenes Expressing Anthrolysin O and Phosphatidylinositol-Specific Phospholipase C from Bacillus anthracis

Hypoxic modulation of hepatocyte responses to the cytokine interleukin‐22

Cutting Edge: CD4 and CD8 T Cells Are Intrinsically Different in Their Proliferative Responses

The dual nature of TH17 cells: shifting the focus to function

Chronic Immunodeficiency in Mice Lacking RasGRP1 Results in CD4 T Cell Immune Activation and Exhaustion

RasGRP1 Regulates Antigen-Induced Developmental Programming by Naive CD8 T Cells

Glucocorticoids inhibit group 3 innate lymphocyte IL-22 production1

Glucocorticoids inhibit group 3 innate lymphocyte IL-22 production

Listeria monocytogenes virulence proteins induce surface expression of Fas ligand on T lymphocytes

Nonsecreted Bacterial Proteins Induce Recall CD8 T Cell Responses But Do Not Serve as Protective Antigens

The transcription factor HIF1α controls expression of the cytokine IL-22 in CD4 T cells

Sugar-Coated Regulation of T Cells

Activation of Antigen-Specific CD8 T Cells Results in Minimal Killing of Bystander Bacteria

NLRP10 is a NOD―like receptor essential to initiate adaptive immunity by dendritic cells

Interleukin-22 deficiency alters colonic microbiota to be transmissible and colitogenic

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