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Nomenclature of voltage-gated sodium channels

Neuron (Cambridge, Mass.), 2000-11, Vol.28 (2), p.365-368

2000

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Small-fiber neuropathy Nav1.8 mutation shifts activation to hyperpolarized potentials and increases excitability of dorsal root ganglion neurons

The Journal of neuroscience, 2013-08, Vol.33 (35), p.14087-14097

2013

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Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons

The Journal of neuroscience, 2000-12, Vol.20 (23), p.8754-8761

2000

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The neuron as a dynamic electrogenic machine: modulation of sodium-channel expression as a basis for functional plasticity in neurons

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2000-02, Vol.355 (1394), p.199-213

Waxman, S G

2000

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Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons

The Journal of neuroscience, 2001-08, Vol.21 (16), p.5952-5961

2001

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Downregulation of Tetrodotoxin-Resistant Sodium Currents and Upregulation of a Rapidly Repriming Tetrodotoxin-Sensitive Sodium Current in Small Spinal Sensory Neurons after Nerve Injury

The Journal of neuroscience, 1997-05, Vol.17 (10), p.3503-3514

1997

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Sodium Channels in Normal and Pathological Pain

Annual review of neuroscience, 2010-01, Vol.33 (1), p.325-347

2010

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SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain model

Neuroreport, 1998-04, Vol.9 (6), p.967-972

1998

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Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination

Brain (London, England : 1878), 2003-07, Vol.126 (7), p.1552-1561

2003

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Can robots patch‐clamp as well as humans? Characterization of a novel sodium channel mutation

The Journal of physiology, 2010-06, Vol.588 (11), p.1915-1927

2010

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Rescue of alpha -SNS Sodium Channel Expression in Small Dorsal Root Ganglion Neurons After Axotomy by Nerve Growth Factor In Vivo

Journal of neurophysiology, 1998-05, Vol.79 (5), p.2668-2676

1998

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Slow Closed-State Inactivation: A Novel Mechanism Underlying Ramp Currents in Cells Expressing the hNE/PN1 Sodium Channel

The Journal of neuroscience, 1998-12, Vol.18 (23), p.9607-9619

1998

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Activated polymorphonuclear cells promote injury and excitability of dorsal root ganglia neurons

Experimental neurology, 2008-04, Vol.210 (2), p.286-294

2008

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Two tetrodotoxin-resistant sodium channels in human dorsal root ganglion neurons

FEBS letters, 1999-11, Vol.462 (1), p.117-120

1999

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Ionic mechanisms of anoxic injury in mammalian CNS white matter: role of Na+ channels and Na(+)-Ca2+ exchanger

The Journal of neuroscience, 1992-02, Vol.12 (2), p.430-439

1992

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Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

Pain (Amsterdam), 1999-12, Vol.83 (3), p.591-600

1999

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Nitric Oxide Blocks Fast, Slow, and Persistent Na+ Channels in C-Type DRG Neurons by S-Nitrosylation

Journal of neurophysiology, 2002-02, Vol.87 (2), p.761-775

2002

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Localization of the tetrodotoxin-resistant sodium channel NaN in nociceptors

Neuroreport, 2000-01, Vol.11 (1), p.199-202

2000

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Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5

Brain (London, England : 1878), 2010-03, Vol.133 (3), p.835-846

2010

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Changes in expression of voltage-gated potassium channels in dorsal root ganglion neurons following axotomy

Muscle & nerve, 1999-04, Vol.22 (4), p.502-507

1999

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Sodium channels, excitability of primary sensory neurons, and the molecular basis of pain

Muscle & nerve, 1999-09, Vol.22 (9), p.1177-1187

1999

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alpha -SNS Produces the Slow TTX-Resistant Sodium Current in Large Cutaneous Afferent DRG Neurons

Journal of neurophysiology, 2000-08, Vol.84 (2), p.710-718

2000

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Voltage-gated calcium channels in CNS white matter: role in anoxic injury

Journal of neurophysiology, 1995-07, Vol.74 (1), p.369-377

1995

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Expression of voltage-activated ion channels by astrocytes and oligodendrocytes in the hippocampal slice

Journal of neurophysiology, 1993-11, Vol.70 (5), p.1863-1873

1993

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Activated Microglia Contribute to the Maintenance of Chronic Pain after Spinal Cord Injury

The Journal of neuroscience, 2006-04, Vol.26 (16), p.4308-4317

2006

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Nomenclature of voltage-gated sodium channels

Small-fiber neuropathy Nav1.8 mutation shifts activation to hyperpolarized potentials and increases excitability of dorsal root ganglion neurons

Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons

The neuron as a dynamic electrogenic machine: modulation of sodium-channel expression as a basis for functional plasticity in neurons

Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons

Downregulation of Tetrodotoxin-Resistant Sodium Currents and Upregulation of a Rapidly Repriming Tetrodotoxin-Sensitive Sodium Current in Small Spinal Sensory Neurons after Nerve Injury

Sodium Channels in Normal and Pathological Pain

SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain model

Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination

Can robots patch‐clamp as well as humans? Characterization of a novel sodium channel mutation

Rescue of alpha -SNS Sodium Channel Expression in Small Dorsal Root Ganglion Neurons After Axotomy by Nerve Growth Factor In Vivo

Slow Closed-State Inactivation: A Novel Mechanism Underlying Ramp Currents in Cells Expressing the hNE/PN1 Sodium Channel

Activated polymorphonuclear cells promote injury and excitability of dorsal root ganglia neurons

Two tetrodotoxin-resistant sodium channels in human dorsal root ganglion neurons

Ionic mechanisms of anoxic injury in mammalian CNS white matter: role of Na+ channels and Na(+)-Ca2+ exchanger

Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

Nitric Oxide Blocks Fast, Slow, and Persistent Na+ Channels in C-Type DRG Neurons by S-Nitrosylation

Localization of the tetrodotoxin-resistant sodium channel NaN in nociceptors

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5

Changes in expression of voltage-gated potassium channels in dorsal root ganglion neurons following axotomy

Sodium channels, excitability of primary sensory neurons, and the molecular basis of pain

alpha -SNS Produces the Slow TTX-Resistant Sodium Current in Large Cutaneous Afferent DRG Neurons

Voltage-gated calcium channels in CNS white matter: role in anoxic injury

Expression of voltage-activated ion channels by astrocytes and oligodendrocytes in the hippocampal slice

Activated Microglia Contribute to the Maintenance of Chronic Pain after Spinal Cord Injury

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