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2,3,7,8-tetrachlorodibenzo-p-dioxin reduces myocardial hypoxia and vascular endothelial growth factor expression during chick embryo development

Birth defects research. A Clinical and molecular teratology, 2004-02, Vol.70 (2), p.51-58

2004

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Inhibition of neovascularization by environmental agents

Cardiovascular toxicology, 2005-01, Vol.5 (2), p.215-226

2005

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Cardiac hypertrophy in Aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure

Toxicology and applied pharmacology, 2003-12, Vol.193 (2), p.177-187

2003

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Vascular endothelial growth factor rescues 2,3,7,8-tetrachlorodibenzo-p-dioxin inhibition of coronary vasculogenesis

Birth defects research. A Clinical and molecular teratology, 2003-07, Vol.67 (7), p.496-503

2003

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Dibenzo[def,p]Chrysene (DBC) Suppresses Antibody Formation in Spleen Cells Following Oral Exposures of Mice

Journal of Toxicology and Environmental Health, Part A, 2013-01, Vol.76 (1), p.16-24

2013

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NFATc3 contributes to intermittent hypoxia-induced arterial remodeling in mice

American journal of physiology. Heart and circulatory physiology, 2010-08, Vol.299 (2), p.H356-H363

2010

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An activated renin–angiotensin system maintains normal blood pressure in aryl hydrocarbon receptor heterozygous mice but not in null mice

Biochemical pharmacology, 2010-07, Vol.80 (2), p.197-204

2010

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Effects of Aryl Hydrocarbon Receptor-Mediated Early Life Stage Toxicity on Lake Trout Populations in Lake Ontario during the 20th Century

Environmental science & technology, 2003-09, Vol.37 (17), p.3864-3877

2003

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Endothelin-1–Mediated Increase in Reactive Oxygen Species and NADPH Oxidase Activity in Hearts of Aryl Hydrocarbon Receptor (AhR) Null Mice

Toxicological sciences, 2005-11, Vol.88 (1), p.265-273

2005

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Characterizing the role of endothelin-1 in the progression of cardiac hypertrophy in aryl hydrocarbon receptor (AhR) null mice

Toxicology and applied pharmacology, 2006-04, Vol.212 (2), p.127-135

2006

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Altering HIF-1α Through 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) Exposure Affects Coronary Vessel Development

Cardiovascular toxicology, 2013-06, Vol.13 (2), p.161-167

2013

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Prospective study of risk factors for development of non-insulin dependent diabetes in middle aged British men

BMJ (Online), 1995-03, Vol.310 (6979), p.560

1995

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Perinatal 2,3,7,8-Tetrachlorodibenzo-p-dioxin Exposure Sensitizes Offspring to Angiotensin II-induced Hypertension

Cardiovascular toxicology, 2008-09, Vol.8 (3), p.145-154

2008

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Toxicity of 2,3,7,8-tetrachlorodibenzo-P-dioxin to brook trout (lsalvelinus fontinalis) during early development

Environmental toxicology and chemistry, 1994-05, Vol.13 (5), p.817-820

1994

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Unraveling Gene-Gene Interactions Regulated by Ligands of the Aryl Hydrocarbon Receptor

Environmental health perspectives, 2004-03, Vol.112 (4), p.403-412

2004

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Correlation of cardiotoxicity mediated by halogenated aromatic hydrocarbons to aryl hydrocarbon receptor activation

Toxicological sciences, 2001-05, Vol.61 (1), p.187-196

2001

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2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) inhibition of coronary vasculogenesis is mediated, in part, by reduced responsiveness to endogenous angiogenic stimuli, including vascular endothelial growth factor A (VEGF-A)

Birth defects research. A Clinical and molecular teratology, 2005-06, Vol.73 (6), p.440-446

2005

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Overview of Developmental Heart Defects by Dioxins, PCBs, and Pesticides

Journal of environmental science and health. Part C, Environmental carcinogenesis & ecotoxicology reviews, 2009-10, Vol.27 (4), p.276-285

2009

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Potencies of polychlorinated dibenzo- p-dioxin, dibenzofuran, and biphenyl congeners, relative to 2,3,7,8-tetrachlorodibenzo- p-dioxin, for producing early life stage mortality in rainbow trout ( Oncorhynchus mykiss)

Aquatic toxicology, 1991, Vol.21 (3), p.219-237

1991

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Early Developmental 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Exposure Decreases Chick Embryo Heart Chronotropic Response to Isoproterenol but Not to Agents Affecting Signals Downstream of the Beta-Adrenergic Receptor

Toxicological sciences, 2005-02, Vol.83 (2), p.363-371

2005

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Molecular cloning and expression of abscisic acid-responsive genes in embryos of dormant wheat seeds

Plant physiology (Bethesda), 1991-03, Vol.95 (3), p.814-821

1991

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Effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on development of anuran amphibians

Environmental toxicology and chemistry, 1997-02, Vol.16 (2), p.230-240

1997

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Synthesis of Abscisic Acid-Responsive, Heat-Stable Proteins in Embryonic Axes of Dormant Wheat Grain

Plant physiology (Bethesda), 1990-06, Vol.93 (2), p.662-667

1990

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Molecular characterization and developmental expression of the aryl hydrocarbon receptor from the chick embryo

Comparative biochemistry and physiology. C, Comparative pharmacology and toxicology, 2000-07, Vol.126 (3), p.305-319

2000

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Abstract 298: Cytochrome P4501A1 Contributes to Nitric Oxide-Dependent Vasodilation and Blood Pressure Lowering on an Omega-3 Polyunsaturated Fatty Acid-Enriched Diet

Hypertension (Dallas, Tex. 1979), 2012-09, Vol.60 (suppl_1)

2012

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2,3,7,8-tetrachlorodibenzo-p-dioxin reduces myocardial hypoxia and vascular endothelial growth factor expression during chick embryo development

Inhibition of neovascularization by environmental agents

Cardiac hypertrophy in Aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure

Vascular endothelial growth factor rescues 2,3,7,8-tetrachlorodibenzo-p-dioxin inhibition of coronary vasculogenesis

Dibenzo[def,p]Chrysene (DBC) Suppresses Antibody Formation in Spleen Cells Following Oral Exposures of Mice

NFATc3 contributes to intermittent hypoxia-induced arterial remodeling in mice

An activated renin–angiotensin system maintains normal blood pressure in aryl hydrocarbon receptor heterozygous mice but not in null mice

Effects of Aryl Hydrocarbon Receptor-Mediated Early Life Stage Toxicity on Lake Trout Populations in Lake Ontario during the 20th Century

Endothelin-1–Mediated Increase in Reactive Oxygen Species and NADPH Oxidase Activity in Hearts of Aryl Hydrocarbon Receptor (AhR) Null Mice

Characterizing the role of endothelin-1 in the progression of cardiac hypertrophy in aryl hydrocarbon receptor (AhR) null mice

Altering HIF-1α Through 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) Exposure Affects Coronary Vessel Development

Prospective study of risk factors for development of non-insulin dependent diabetes in middle aged British men

Perinatal 2,3,7,8-Tetrachlorodibenzo-p-dioxin Exposure Sensitizes Offspring to Angiotensin II-induced Hypertension

Toxicity of 2,3,7,8-tetrachlorodibenzo-P-dioxin to brook trout (lsalvelinus fontinalis) during early development

Unraveling Gene-Gene Interactions Regulated by Ligands of the Aryl Hydrocarbon Receptor

Correlation of cardiotoxicity mediated by halogenated aromatic hydrocarbons to aryl hydrocarbon receptor activation

2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) inhibition of coronary vasculogenesis is mediated, in part, by reduced responsiveness to endogenous angiogenic stimuli, including vascular endothelial growth factor A (VEGF-A)

Overview of Developmental Heart Defects by Dioxins, PCBs, and Pesticides

Potencies of polychlorinated dibenzo- p-dioxin, dibenzofuran, and biphenyl congeners, relative to 2,3,7,8-tetrachlorodibenzo- p-dioxin, for producing early life stage mortality in rainbow trout ( Oncorhynchus mykiss)

Early Developmental 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Exposure Decreases Chick Embryo Heart Chronotropic Response to Isoproterenol but Not to Agents Affecting Signals Downstream of the Beta-Adrenergic Receptor

Molecular cloning and expression of abscisic acid-responsive genes in embryos of dormant wheat seeds

Effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on development of anuran amphibians

Synthesis of Abscisic Acid-Responsive, Heat-Stable Proteins in Embryonic Axes of Dormant Wheat Grain

Molecular characterization and developmental expression of the aryl hydrocarbon receptor from the chick embryo

Abstract 298: Cytochrome P4501A1 Contributes to Nitric Oxide-Dependent Vasodilation and Blood Pressure Lowering on an Omega-3 Polyunsaturated Fatty Acid-Enriched Diet

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