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Control of bone mass and remodeling by PTH receptor signaling in osteocytes

PloS one, 2008-08, Vol.3 (8), p.e2942-e2942

2008

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Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors

Scientific reports, 2020-07, Vol.10 (1), p.11933, Article 11933

2020

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Suppression of Autophagy in Osteocytes Mimics Skeletal Aging

The Journal of biological chemistry, 2013-06, Vol.288 (24), p.17432-17440

2013

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The Pathophysiological Sequence of Glucocorticoid-Induced Osteonecrosis of the Femoral Head in Male Mice

Endocrinology (Philadelphia), 2017-11, Vol.158 (11), p.3817-3831

2017

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Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency

Bone (New York, N.Y.), 2014-09, Vol.66, p.146-154

2014

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Oxidation-specific epitopes restrain bone formation

Nature communications, 2018-06, Vol.9 (1), p.2193-11, Article 2193

2018

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Sirtuin1 (Sirt1) Promotes Cortical Bone Formation by Preventing β-Catenin Sequestration by FoxO Transcription Factors in Osteoblast Progenitors

The Journal of biological chemistry, 2014-08, Vol.289 (35), p.24069-24078

2014

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Cxcl12 Deletion in Mesenchymal Cells Increases Bone Turnover and Attenuates the Loss of Cortical Bone Caused by Estrogen Deficiency in Mice

Journal of bone and mineral research, 2020-08, Vol.35 (8), p.1441-1451

2020

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Sirtuin1 Suppresses Osteoclastogenesis by Deacetylating FoxOs

Molecular endocrinology (Baltimore, Md.), 2015-10, Vol.29 (10), p.1498-1509

2015

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Skeletal Involution by Age-associated Oxidative Stress and Its Acceleration by Loss of Sex Steroids

The Journal of biological chemistry, 2007-09, Vol.282 (37), p.27285-27297

2007

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Deletion of FoxO1, 3, and 4 in Osteoblast Progenitors Attenuates the Loss of Cancellous Bone Mass in a Mouse Model of Type 1 Diabetes

Journal of bone and mineral research, 2017-01, Vol.32 (1), p.60-69

2017

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FOXOs attenuate bone formation by suppressing Wnt signaling

The Journal of clinical investigation, 2013-08, Vol.123 (8), p.3409-3419

2013

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For whom the bell tolls: Distress signals from long-lived osteocytes and the pathogenesis of metabolic bone diseases

Bone (New York, N.Y.), 2013-06, Vol.54 (2), p.272-278

2013

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RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass

Bone (New York, N.Y.), 2023-05, Vol.170, p.116702-116702, Article 116702

2023

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Reproducibility of Results in Preclinical Studies: A Perspective From the Bone Field

Journal of bone and mineral research, 2014-10, Vol.29 (10), p.2131-2140

2014

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Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency

JCI insight, 2021-05, Vol.6 (10)

2021

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Intermittent Parathyroid Hormone Administration Counteracts the Adverse Effects of Glucocorticoids on Osteoblast and Osteocyte Viability, Bone Formation, and Strength in Mice

Endocrinology (Philadelphia), 2010-06, Vol.151 (6), p.2641-2649

2010

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Estrogen receptor-α signaling in osteoblast progenitors stimulates cortical bone accrual

The Journal of clinical investigation, 2013-01, Vol.123 (1), p.394-404

2013

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Endogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice

Aging cell, 2010-04, Vol.9 (2), p.147-161

2010

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Connexin 43 Is Required for the Anti‐Apoptotic Effect of Bisphosphonates on Osteocytes and Osteoblasts In Vivo

Journal of bone and mineral research, 2008-11, Vol.23 (11), p.1712-1721

2008

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Wnt Proteins Prevent Apoptosis of Both Uncommitted Osteoblast Progenitors and Differentiated Osteoblasts by β-Catenin-dependent and -independent Signaling Cascades Involving Src/ERK and Phosphatidylinositol 3-Kinase/AKT

The Journal of biological chemistry, 2005-12, Vol.280 (50), p.41342-41351

2005

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Osteocyte Apoptosis Is Induced by Weightlessness in Mice and Precedes Osteoclast Recruitment and Bone Loss

Journal of bone and mineral research, 2006-04, Vol.21 (4), p.605-615

2006

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The Estrogen Receptor-α in Osteoclasts Mediates the Protective Effects of Estrogens on Cancellous But Not Cortical Bone

Molecular endocrinology (Baltimore, Md.), 2010-02, Vol.24 (2), p.323-334

2010

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Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency

Scientific reports, 2022-06, Vol.12 (1), p.10257-10257, Article 10257

2022

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Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice

Aging cell, 2021-08, Vol.20 (8), p.e13442-n/a

2021

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Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors

Suppression of Autophagy in Osteocytes Mimics Skeletal Aging

The Pathophysiological Sequence of Glucocorticoid-Induced Osteonecrosis of the Femoral Head in Male Mice

Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency

Oxidation-specific epitopes restrain bone formation

Sirtuin1 (Sirt1) Promotes Cortical Bone Formation by Preventing β-Catenin Sequestration by FoxO Transcription Factors in Osteoblast Progenitors

Cxcl12 Deletion in Mesenchymal Cells Increases Bone Turnover and Attenuates the Loss of Cortical Bone Caused by Estrogen Deficiency in Mice

Sirtuin1 Suppresses Osteoclastogenesis by Deacetylating FoxOs

Skeletal Involution by Age-associated Oxidative Stress and Its Acceleration by Loss of Sex Steroids

Deletion of FoxO1, 3, and 4 in Osteoblast Progenitors Attenuates the Loss of Cancellous Bone Mass in a Mouse Model of Type 1 Diabetes

FOXOs attenuate bone formation by suppressing Wnt signaling

For whom the bell tolls: Distress signals from long-lived osteocytes and the pathogenesis of metabolic bone diseases

RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass

Reproducibility of Results in Preclinical Studies: A Perspective From the Bone Field

Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency

Intermittent Parathyroid Hormone Administration Counteracts the Adverse Effects of Glucocorticoids on Osteoblast and Osteocyte Viability, Bone Formation, and Strength in Mice

Estrogen receptor-α signaling in osteoblast progenitors stimulates cortical bone accrual

Endogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice

Connexin 43 Is Required for the Anti‐Apoptotic Effect of Bisphosphonates on Osteocytes and Osteoblasts In Vivo

Wnt Proteins Prevent Apoptosis of Both Uncommitted Osteoblast Progenitors and Differentiated Osteoblasts by β-Catenin-dependent and -independent Signaling Cascades Involving Src/ERK and Phosphatidylinositol 3-Kinase/AKT

Osteocyte Apoptosis Is Induced by Weightlessness in Mice and Precedes Osteoclast Recruitment and Bone Loss

The Estrogen Receptor-α in Osteoclasts Mediates the Protective Effects of Estrogens on Cancellous But Not Cortical Bone

Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency

Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice

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