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Effect of calcium channel blockers on hemodynamic responses to defibrillation

The American heart journal, 1989-03, Vol.117 (3), p.569-576

1989

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Transthoracic Defibrillation Using Sequential and Simultaneous Dual Shock Pathways: Experimental Studies

Pacing and clinical electrophysiology, 1990-02, Vol.13 (2), p.207-217

1990

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Is transthoracic impedance arrythmia specific ? Experimental studies

The American heart journal, 1994, Vol.127 (4), p.842-847

1994

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Current-based transthoracic defibrillation

The American journal of cardiology, 1996, Vol.78 (10), p.1113-1118

1996

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Effect of age on diastolic left ventricular filling at rest and during inotripic stimulation and acute systemic hypertension : experimental studies in conscious beagles

The American heart journal, 1990, Vol.120 (1), p.73-81

1990

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Effect of calcium channel blockers on hemodynamic responses to defibrillation

The American heart journal, 1989-01, Vol.177 (3), p.569-576

1989

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Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

The American journal of cardiology, 1983-12, Vol.52 (10), p.1318-1325

1983

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Mechanisms responsible for decline in transthoracic impedance after DC shocks

American journal of physiology. Heart and circulatory physiology, 1989, Vol.26 (4), p.H1180-H1183

1989

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Ultrasound Characterization of Acoustic Properties of Acute Intracardiac Thrombi: Studies in a New Experimental Model

Journal of the American Society of Echocardiography, 1988-07, Vol.1 (4), p.264-270

1988

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Transient myocardial ischemia: experimental echocardiographic demonstration and evaluation of myocardial contraction abnormalities

Canadian journal of cardiology, 1986-07, Vol.Suppl A, p.136A-141A

1986

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Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

The American journal of cardiology, 1984, Vol.52 (10), p.1318-1325

1984

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Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

Clinical cancer research, 2023-05, Vol.29 (10), p.1984-1995

2023

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CLECL1+ CLL Cells: A Key Subset That Drives CLL Survival and Proliferation and Shapes a Th2-Biased Tumor Microenvironment

Blood, 2022-11, Vol.140 (Supplement 1), p.1528-1529

2022

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Serum Proteomic Analyses Suggest That the HMGB1 and Other Inflammatory Pathways Are Operational in MBL and Are Less in Overt CLL

Blood, 2021-11, Vol.138 (Supplement 1), p.2625-2625

2021

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Identification of Three Unique Clusters of Serum Proteins with Distinctive Functionalities in IGHV-Mutated MBL

Blood, 2022-11, Vol.140 (Supplement 1), p.9859-9861

2022

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Chronic lymphocytic leukemia–like monoclonal B-cell lymphocytosis exhibits an increased inflammatory signature that is reduced in early-stage chronic lymphocytic leukemia

Experimental hematology, 2021-03, Vol.95, p.68-80

2021

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Dual Inhibition of PI3K-δ and PI3K-γ By Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Patient-Derived Xenograft Model

Blood, 2018-11, Vol.132 (Supplement 1), p.4420-4420

2018

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Abstract 960: Mechanisms underlying primary ibrutinib sensitivity in CLL

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.960-960

2017

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Gene Expression and Cytokine Analyses Identify Markers of Progression from CLL-like Monoclonal B-Cell Lymphocytosis to Chronic Lymphocytic Leukemia

Blood, 2019-11, Vol.134 (Supplement_1), p.3027-3027

2019

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Deciphering the CXCL9-CXCL10-CXCL11/CXCR3 Axis in CLL-like Monoclonal B-Cell Lymphocytosis and Chronic Lymphocytic Leukemia: A New Target for Immune Activation?

Blood, 2019-11, Vol.134 (Supplement_1), p.3029-3029

2019

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CLL-like monoclonal B cell lymphocytosis displays an increased inflammatory signature that is reduced in early stage chronic lymphocytic leukemia

Experimental hematology, 2021-01, Vol.95, p.68-80

2021

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In Vivo Evidence of in Situ Cell Death Preferentially Occurring in CLL Patients with Minimal Lymphocytosis By the Diminished BCL2 and Loss of T Cell Support after Ibrutinib Treatment

Blood, 2016-12, Vol.128 (22), p.5586-5586

2016

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Abstract 4933: Duvelisib eliminates CLL B Cells, impairs CLL-supporting cells, and overcomes ibrutinib resistance in a patient-derived xenograft model

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.4933-4933

2023

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Pediatric defibrillation: importance of paddle size in determining transthoracic impedance

Pediatrics (Evanston), 1988-12, Vol.82 (6), p.914-918

1988

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Evaluation of the Remler M2000 Blood Pressure Recorder: Comparison with Intraarterial Blood Pressure Recordings Both at Hospital and at Home

Hypertension (Dallas, Tex. 1979), 1984-03, Vol.6 (2, Part 1), p.209-215

1984

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Effect of calcium channel blockers on hemodynamic responses to defibrillation

Transthoracic Defibrillation Using Sequential and Simultaneous Dual Shock Pathways: Experimental Studies

Is transthoracic impedance arrythmia specific ? Experimental studies

Current-based transthoracic defibrillation

Effect of age on diastolic left ventricular filling at rest and during inotripic stimulation and acute systemic hypertension : experimental studies in conscious beagles

Effect of calcium channel blockers on hemodynamic responses to defibrillation

Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

Mechanisms responsible for decline in transthoracic impedance after DC shocks

Ultrasound Characterization of Acoustic Properties of Acute Intracardiac Thrombi: Studies in a New Experimental Model

Transient myocardial ischemia: experimental echocardiographic demonstration and evaluation of myocardial contraction abnormalities

Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

CLECL1+ CLL Cells: A Key Subset That Drives CLL Survival and Proliferation and Shapes a Th2-Biased Tumor Microenvironment

Serum Proteomic Analyses Suggest That the HMGB1 and Other Inflammatory Pathways Are Operational in MBL and Are Less in Overt CLL

Identification of Three Unique Clusters of Serum Proteins with Distinctive Functionalities in IGHV-Mutated MBL

Chronic lymphocytic leukemia–like monoclonal B-cell lymphocytosis exhibits an increased inflammatory signature that is reduced in early-stage chronic lymphocytic leukemia

Dual Inhibition of PI3K-δ and PI3K-γ By Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Patient-Derived Xenograft Model

Abstract 960: Mechanisms underlying primary ibrutinib sensitivity in CLL

Gene Expression and Cytokine Analyses Identify Markers of Progression from CLL-like Monoclonal B-Cell Lymphocytosis to Chronic Lymphocytic Leukemia

Deciphering the CXCL9-CXCL10-CXCL11/CXCR3 Axis in CLL-like Monoclonal B-Cell Lymphocytosis and Chronic Lymphocytic Leukemia: A New Target for Immune Activation?

CLL-like monoclonal B cell lymphocytosis displays an increased inflammatory signature that is reduced in early stage chronic lymphocytic leukemia

In Vivo Evidence of in Situ Cell Death Preferentially Occurring in CLL Patients with Minimal Lymphocytosis By the Diminished BCL2 and Loss of T Cell Support after Ibrutinib Treatment

Abstract 4933: Duvelisib eliminates CLL B Cells, impairs CLL-supporting cells, and overcomes ibrutinib resistance in a patient-derived xenograft model

Pediatric defibrillation: importance of paddle size in determining transthoracic impedance

Evaluation of the Remler M2000 Blood Pressure Recorder: Comparison with Intraarterial Blood Pressure Recordings Both at Hospital and at Home

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