UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Inotropic Effects of Prostacyclins on the Right Ventricle Are Abolished in Isolated Rat Hearts With Right-Ventricular Hypertrophy and Failure

Journal of cardiovascular pharmacology, 2017-01, Vol.69 (1), p.1-12

2017

Volltextzugriff (PDF)

Differential Effects of Stable Prostacyclin Analogs on Smooth Muscle Proliferation and Cyclic AMP Generation in Human Pulmonary Artery

American journal of respiratory cell and molecular biology, 2002-02, Vol.26 (2), p.194-201

2002

Volltextzugriff (PDF)

Improving Interpretation of Cardiac Phenotypes and Enhancing Discovery With Expanded Knowledge in the Gene Ontology

Circulation. Genomic and precision medicine, 2018-02, Vol.11 (2), p.e001813-e001813

2018

Volltextzugriff (PDF)

Ex vivo relaxations of pulmonary arteries induced by prostacyclin mimetics are highly dependent of the precontractile agents

Prostaglandins & other lipid mediators, 2015-09, Vol.121 (Pt A), p.46-52

2015

Volltextzugriff (PDF)

The large-conductance Ca2+-activated K+ channel is essential for innate immunity

Nature (London), 2004-02, Vol.427 (6977), p.853-858

2004

Volltextzugriff (PDF)

A comparative study of PGI2 mimetics used clinically on the vasorelaxation of human pulmonary arteries and veins, role of the DP-receptor

Prostaglandins & other lipid mediators, 2013-12, Vol.107, p.48-55

2013

Volltextzugriff (PDF)

Variable effects of inhibiting iNOS and closing the vascular ATP-sensitive potassium channel (via its pore-forming and sulfonylurea receptor subunits) in endotoxic shock

Shock (Augusta, Ga.), 2009-05, Vol.31 (5), p.535-541

2009

Volltextzugriff (PDF)

Evidence that inward rectifier K+ channels mediate relaxation by the PGI2 receptor agonist cicaprost via a cyclic AMP-independent mechanism

Cardiovascular research, 2006, Vol.69 (1), p.107-115

2006

Volltextzugriff (PDF)

Differential effects of vasopressin and norepinephrine on vascular reactivity in a long-term rodent model of sepsis

Critical care medicine, 2007-10, Vol.35 (10), p.2337-2343

2007

Volltextzugriff (PDF)

Inhibition of vascular adenosine triphosphate-sensitive potassium channels by sympathetic tone during sepsis

Critical care medicine, 2012-04, Vol.40 (4), p.1261-1268

2012

Volltextzugriff (PDF)

A Mechanism for ATP-Sensitive Potassium Channel Diversity: Functional Coassembly of Two Pore-Forming Subunits

Proceedings of the National Academy of Sciences - PNAS, 2001-01, Vol.98 (2), p.729-734

2001

Volltextzugriff (PDF)

Prostacyclins have no direct inotropic effect on isolated atrial strips from the normal and pressure-overloaded human right heart

Pulmonary circulation, 2017-04, Vol.7 (2), p.339-347

2017

Volltextzugriff (PDF)

Functional Expression of Inward Rectifier Potassium Channels in Cultured Human Pulmonary Smooth Muscle Cells: Evidence for a Major Role of Kir2.4 Subunits

The Journal of membrane biology, 2006-01, Vol.213 (1), p.19-29

2006

Volltextzugriff (PDF)

THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: G protein‐coupled receptors

British journal of pharmacology, 2019-12, Vol.176 (S1), p.S21-S141

2019

Volltextzugriff (PDF)

Effects of the adenylyl cyclase inhibitor SQ22536 on iloprost‐induced vasorelaxation and cyclic AMP elevation in isolated guinea‐pig aorta

British journal of pharmacology, 1999-02, Vol.126 (4), p.845-847

1999

Volltextzugriff (PDF)

Stoking Up BKCa Channels in Hemorrhagic Shock: Which Channel Subunit Is Really Fueling the Fire?

Circulation research, 2007-08, Vol.101 (5), p.436-438

2007

Volltextzugriff (PDF)

Reversal of life-threatening, drug-related potassium-channel syndrome by glibenclamide

The Lancet (British edition), 2005-05, Vol.365 (9474), p.1873-1875

2005

Volltextzugriff (PDF)

Attenuated vascular responsiveness to K+ channel openers in diabetes mellitus: the differential role of reactive oxygen species

General physiology and biophysics, 2013-12, Vol.32 (4), p.527-534

2013

Volltextzugriff (PDF)

The pore‐forming subunit of the K ATP channel is an important molecular target for LPS‐induced vascular hyporeactivity in vitro

British journal of pharmacology, 2005-02, Vol.144 (3), p.367-375

2005

Volltextzugriff (PDF)

Assembly Limits the Pharmacological Complexity of ATP-sensitive Potassium Channels

The Journal of biological chemistry, 2002-04, Vol.277 (16), p.13717-13723

2002

Volltextzugriff (PDF)

Different molecular sites of action for the K ATP channel inhibitors, PNU‐99963 and PNU‐37883A

British journal of pharmacology, 2003-05, Vol.139 (1), p.122-128

2003

Volltextzugriff (PDF)

BK large conductance Ca²+-activated K+ channel-deficient mice are not resistant to hypotension and display reduced survival benefit following polymicrobial sepsis

Shock (Augusta, Ga.), 2011-05, Vol.35 (5), p.485-491

2011

Volltextzugriff (PDF)

Nuclear translocation of calcineurin Aβ but not calcineurin Aα by platelet-derived growth factor in rat aortic smooth muscle

American Journal of Physiology: Cell Physiology, 2007-06, Vol.292 (6), p.C2213-C2225

2007

Volltextzugriff (PDF)

The molecular site of action of KATP channel inhibitors determines their ability to inhibit iNOS-mediated relaxation in rat aorta

Cardiovascular research, 2002-10, Vol.56 (1), p.154-163

2002

Volltextzugriff (PDF)

Do Anionic Phospholipids Serve as Cofactors or Second Messengers for the Regulation of Activity of Cloned ATP-Sensitive K+ Channels?

Circulation research, 2003-10, Vol.93 (7), p.646-655

2003

Volltextzugriff (PDF)

26 – 50 von 110

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(104)
Mit Volltextzugriff
(104)

Publikationsform

Artikel
(109)
Buchkapitel
(1)

Sprache

Englisch
(110)
Japanisch
(22)

Erscheinungsjahr

n.n

Menü

Weitere Dienste

Einstellungen

Inotropic Effects of Prostacyclins on the Right Ventricle Are Abolished in Isolated Rat Hearts With Right-Ventricular Hypertrophy and Failure

Differential Effects of Stable Prostacyclin Analogs on Smooth Muscle Proliferation and Cyclic AMP Generation in Human Pulmonary Artery

Improving Interpretation of Cardiac Phenotypes and Enhancing Discovery With Expanded Knowledge in the Gene Ontology

Ex vivo relaxations of pulmonary arteries induced by prostacyclin mimetics are highly dependent of the precontractile agents

The large-conductance Ca2+-activated K+ channel is essential for innate immunity

A comparative study of PGI2 mimetics used clinically on the vasorelaxation of human pulmonary arteries and veins, role of the DP-receptor

Variable effects of inhibiting iNOS and closing the vascular ATP-sensitive potassium channel (via its pore-forming and sulfonylurea receptor subunits) in endotoxic shock

Evidence that inward rectifier K+ channels mediate relaxation by the PGI2 receptor agonist cicaprost via a cyclic AMP-independent mechanism

Differential effects of vasopressin and norepinephrine on vascular reactivity in a long-term rodent model of sepsis

Inhibition of vascular adenosine triphosphate-sensitive potassium channels by sympathetic tone during sepsis

A Mechanism for ATP-Sensitive Potassium Channel Diversity: Functional Coassembly of Two Pore-Forming Subunits

Prostacyclins have no direct inotropic effect on isolated atrial strips from the normal and pressure-overloaded human right heart

Functional Expression of Inward Rectifier Potassium Channels in Cultured Human Pulmonary Smooth Muscle Cells: Evidence for a Major Role of Kir2.4 Subunits

THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: G protein‐coupled receptors

Effects of the adenylyl cyclase inhibitor SQ22536 on iloprost‐induced vasorelaxation and cyclic AMP elevation in isolated guinea‐pig aorta

Stoking Up BKCa Channels in Hemorrhagic Shock: Which Channel Subunit Is Really Fueling the Fire?

Reversal of life-threatening, drug-related potassium-channel syndrome by glibenclamide

Attenuated vascular responsiveness to K+ channel openers in diabetes mellitus: the differential role of reactive oxygen species

The pore‐forming subunit of the K ATP channel is an important molecular target for LPS‐induced vascular hyporeactivity in vitro

Assembly Limits the Pharmacological Complexity of ATP-sensitive Potassium Channels

Different molecular sites of action for the K ATP channel inhibitors, PNU‐99963 and PNU‐37883A

BK large conductance Ca²+-activated K+ channel-deficient mice are not resistant to hypotension and display reduced survival benefit following polymicrobial sepsis

Nuclear translocation of calcineurin Aβ but not calcineurin Aα by platelet-derived growth factor in rat aortic smooth muscle

The molecular site of action of KATP channel inhibitors determines their ability to inhibit iNOS-mediated relaxation in rat aorta

Do Anionic Phospholipids Serve as Cofactors or Second Messengers for the Regulation of Activity of Cloned ATP-Sensitive K+ Channels?

Suchergebnisse filtern