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The Amino Terminus of the Vaccinia Virus E3 Protein Is Necessary To Inhibit the Interferon Response

Journal of Virology, 2012-05, Vol.86 (10), p.5895-5904

2012

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Loss of Protein Kinase PKR Expression in Human HeLa Cells Complements the Vaccinia Virus E3L Deletion Mutant Phenotype by Restoration of Viral Protein Synthesis

Journal of Virology, 2008-01, Vol.82 (2), p.840-848

2008

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A Role for Z-DNA Binding in Vaccinia Virus Pathogenesis

Proceedings of the National Academy of Sciences - PNAS, 2003-06, Vol.100 (12), p.6974-6979

2003

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Protein Kinase PKR-Dependent Activation of Mitogen-Activated Protein Kinases Occurs through Mitochondrial Adapter IPS-1 and Is Antagonized by Vaccinia Virus E3L

Journal of Virology, 2009-06, Vol.83 (11), p.5718-5725

2009

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Attenuated NYCBH vaccinia virus deleted for the E3L gene confers partial protection against lethal monkeypox virus disease in cynomolgus macaques

Vaccine, 2011-12, Vol.29 (52), p.9684-9690

2011

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Suppression of Proinflammatory Signal Transduction and Gene Expression by the Dual Nucleic Acid Binding Domains of the Vaccinia Virus E3L Proteins

Journal of Virology, 2006-10, Vol.80 (20), p.10083-10095

2006

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Evasion of the Innate Immune Type I Interferon System by Monkeypox Virus

Journal of virology, 2015-10, Vol.89 (20), p.10489-10499

2015

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The NYCBH vaccinia virus deleted for the innate immune evasion gene, E3L, protects rabbits against lethal challenge by rabbitpox virus

Vaccine, 2011-10, Vol.29 (44), p.7659-7669

2011

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The Role of the PKR-Inhibitory Genes, E3L and K3L, in Determining Vaccinia Virus Host Range

Virology (New York, N.Y.), 2002-07, Vol.299 (1), p.133-141

2002

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Inhibition of PKR by vaccinia virus: role of the N- and C-terminal domains of E3L

Virology (New York, N.Y.), 2004-07, Vol.324 (2), p.419-429

2004

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Both Carboxy- and Amino-Terminal Domains of the Vaccinia Virus Interferon Resistance Gene, E3L, Are Required for Pathogenesis in a Mouse Model

Journal of Virology, 2001-01, Vol.75 (2), p.850-856

2001

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Vaccinia viruses with mutations in the E3L gene as potential replication-competent, attenuated vaccines: Intra-nasal vaccination

Vaccine, 2008-01, Vol.26 (5), p.664-676

2008

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The attenuated NYCBH vaccinia virus deleted for the immune evasion gene, E3L, completely protects mice against heterologous challenge with ectromelia virus

Vaccine, 2011-12, Vol.29 (52), p.9691-9696

2011

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The N-terminal domain of the vaccinia virus E3L-protein is required for neurovirulence, but not induction of a protective immune response

Virology (New York, N.Y.), 2005-03, Vol.333 (2), p.263-270

2005

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The E3L Gene of Vaccinia Virus Encodes an Inhibitor of the Interferon-Induced, Double-Stranded RNA-Dependent Protein Kinase

Proceedings of the National Academy of Sciences - PNAS, 1992-06, Vol.89 (11), p.4825-4829

1992

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Regulation of mRNA Translation and Cellular Signaling by Hepatitis C Virus Nonstructural Protein NS5A

Journal of Virology, 2001-06, Vol.75 (11), p.5090-5098

2001

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The Orf virus E3L homologue is able to complement deletion of the vaccinia virus E3L gene in vitro but not in vivo

Virology (New York, N.Y.), 2003-09, Vol.314 (1), p.305-314

2003

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Complementation of Vaccinia Virus Deleted of the E3L Gene by Mutants of E3L

Virology (New York, N.Y.), 1997-12, Vol.239 (2), p.269-276

1997

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Double-stranded RNA is a trigger for apoptosis in vaccinia virus-infected cells

Journal of Virology, 1997-03, Vol.71 (3), p.1992-2003

1997

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Vaccinia Virus E3L Interferon Resistance Protein Inhibits the Interferon-Induced Adenosine Deaminase A-to-I Editing Activity

Virology (New York, N.Y.), 2001-10, Vol.289 (2), p.378-387

2001

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Reversal of the interferon-sensitive phenotype of a vaccinia virus lacking E3L by expression of the reovirus S4 gene

Journal of Virology, 1995-01, Vol.69 (1), p.499-505

1995

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Rescue of vaccinia virus lacking the E3L gene by mutants of E3L

Journal of Virology, 1995-10, Vol.69 (10), p.6605-6608

1995

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Identification of a Conserved Motif That Is Necessary for Binding of the Vaccinia Virus E3L Gene Products to Double-Stranded RNA

Virology (New York, N.Y.), 1993-06, Vol.194 (2), p.537-547

1993

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Products of the porcine group C rotavirus NSP3 gene bind specifically to double-stranded RNA and inhibit activation of the interferon-induced protein kinase PKR

Journal of Virology, 1994-06, Vol.68 (6), p.3821-3829

1994

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Complementation of deletion of the vaccinia virus E3L gene by the Escherichia coli RNase III gene

Virology (New York, N.Y.), 1997-01, Vol.227 (1), p.77-87

1997

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The Amino Terminus of the Vaccinia Virus E3 Protein Is Necessary To Inhibit the Interferon Response

Loss of Protein Kinase PKR Expression in Human HeLa Cells Complements the Vaccinia Virus E3L Deletion Mutant Phenotype by Restoration of Viral Protein Synthesis

A Role for Z-DNA Binding in Vaccinia Virus Pathogenesis

Protein Kinase PKR-Dependent Activation of Mitogen-Activated Protein Kinases Occurs through Mitochondrial Adapter IPS-1 and Is Antagonized by Vaccinia Virus E3L

Attenuated NYCBH vaccinia virus deleted for the E3L gene confers partial protection against lethal monkeypox virus disease in cynomolgus macaques

Suppression of Proinflammatory Signal Transduction and Gene Expression by the Dual Nucleic Acid Binding Domains of the Vaccinia Virus E3L Proteins

Evasion of the Innate Immune Type I Interferon System by Monkeypox Virus

The NYCBH vaccinia virus deleted for the innate immune evasion gene, E3L, protects rabbits against lethal challenge by rabbitpox virus

The Role of the PKR-Inhibitory Genes, E3L and K3L, in Determining Vaccinia Virus Host Range

Inhibition of PKR by vaccinia virus: role of the N- and C-terminal domains of E3L

Both Carboxy- and Amino-Terminal Domains of the Vaccinia Virus Interferon Resistance Gene, E3L, Are Required for Pathogenesis in a Mouse Model

Vaccinia viruses with mutations in the E3L gene as potential replication-competent, attenuated vaccines: Intra-nasal vaccination

The attenuated NYCBH vaccinia virus deleted for the immune evasion gene, E3L, completely protects mice against heterologous challenge with ectromelia virus

The N-terminal domain of the vaccinia virus E3L-protein is required for neurovirulence, but not induction of a protective immune response

The E3L Gene of Vaccinia Virus Encodes an Inhibitor of the Interferon-Induced, Double-Stranded RNA-Dependent Protein Kinase

Regulation of mRNA Translation and Cellular Signaling by Hepatitis C Virus Nonstructural Protein NS5A

The Orf virus E3L homologue is able to complement deletion of the vaccinia virus E3L gene in vitro but not in vivo

Complementation of Vaccinia Virus Deleted of the E3L Gene by Mutants of E3L

Double-stranded RNA is a trigger for apoptosis in vaccinia virus-infected cells

Vaccinia Virus E3L Interferon Resistance Protein Inhibits the Interferon-Induced Adenosine Deaminase A-to-I Editing Activity

Reversal of the interferon-sensitive phenotype of a vaccinia virus lacking E3L by expression of the reovirus S4 gene

Rescue of vaccinia virus lacking the E3L gene by mutants of E3L

Identification of a Conserved Motif That Is Necessary for Binding of the Vaccinia Virus E3L Gene Products to Double-Stranded RNA

Products of the porcine group C rotavirus NSP3 gene bind specifically to double-stranded RNA and inhibit activation of the interferon-induced protein kinase PKR

Complementation of deletion of the vaccinia virus E3L gene by the Escherichia coli RNase III gene

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