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High post‐treatment platelet reactivity identified low‐responders to dual antiplatelet therapy at increased risk of recurrent cardiovascular events after stenting for acute coronary syndrome

Journal of thrombosis and haemostasis, 2006-03, Vol.4 (3), p.542-549

2006

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Assessment of epicardial fat volume and myocardial triglyceride content in severely obese subjects: relationship to metabolic profile, cardiac function and visceral fat

International Journal of Obesity, 2012-03, Vol.36 (3), p.422-430

2012

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Endothelial cell markers and the risk of coronary heart disease: The prospective epidemiological study of myocardial infarction (PRIME) study

Circulation (New York, N.Y.), 2004-03, Vol.109 (11), p.1343-1348

2004

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Genetic risk factors for venous thrombosis in women using combined oral contraceptives: update of the PILGRIM study

Clinical genetics, 2017-01, Vol.91 (1), p.131-136

2017

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PAI-1, obesity, insulin resistance and risk of cardiovascular events

Thrombosis and haemostasis, 1997-07, Vol.78 (1), p.656

1997

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Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Arteriosclerosis, thrombosis, and vascular biology, 2007-10, Vol.27 (10), p.2250

2007

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Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Arteriosclerosis, thrombosis, and vascular biology, 2002-05, Vol.22 (5), p.867

2002

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Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

Arteriosclerosis, thrombosis, and vascular biology, 2000-09, Vol.20 (9), p.2156

2000

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Metabolic determinants are much more important than genetic polymorphisms in determining the PAI-1 activity and antigen plasma concentrations: a family study with part of the Stanislas Cohort

Arteriosclerosis, thrombosis, and vascular biology, 1998-01, Vol.18 (1), p.84

1998

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Epicardial Adipose Tissue Extent: Relationship With Age, Body Fat Distribution, and Coronaropathy

Obesity (Silver Spring, Md.), 2008-11, Vol.16 (11), p.2424-2430

2008

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Increased plasma plasminogen activator inhibitor 1 levels. A possible link between insulin resistance and atherothrombosis

Diabetologia, 1991-07, Vol.34 (7), p.457-462

1991

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The use of wide-scale mental agility testing to identify people at risk of dementia: crucial or harmful?

The Journal of the Royal College of Physicians of Edinburgh, 2014, Vol.44 (1), p.30-35

2014

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Divergent effects of obesity on fragility fractures

Clinical interventions in aging, 2014-01, Vol.9, p.1629-1636

2014

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Interaction between the C-260T polymorphism of the CD14 gene and the plasma IL-6 concentration on the risk of myocardial infarction: the HIFMECH study

Atherosclerosis, 2005-04, Vol.179 (2), p.317-323

2005

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The Association Between Chronic Illness and Functional Change Among Participants in a Comprehensive Geriatric Assessment Program

Journal of the American Geriatrics Society (JAGS), 1998-06, Vol.46 (6), p.677-682

1998

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The A -844G polymorphism in the PAI-1 gene is associated with a higher risk of venous thrombosis in factor V Leiden carriers

Arteriosclerosis, thrombosis, and vascular biology, 2000-05, Vol.20 (5), p.1387

2000

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The plasminogen activator inhibitor‐1 −675 4G/5G genotype influences the risk of myocardial infarction associated with elevated plasma proinsulin and insulin concentrations in men from Europe: the HIFMECH Study

Journal of thrombosis and haemostasis, 2003-11, Vol.1 (11), p.2322-2329

2003

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Activated thrombin activatable fibrinolysis inhibitor levels are associated with the risk of cardiovascular death in patients with coronary artery disease: the AtheroGene study

Journal of thrombosis and haemostasis, 2009-01, Vol.7 (1), p.49-57

2009

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Regulation of fibrinolysis in the development of atherothrombosis: role of adipose tissue

Thrombosis and haemostasis, 1999-08, Vol.82 (2), p.832

1999

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CD11b+ leukocyte microparticles are associated with high‐risk angiographic lesions and recurrent cardiovascular events in acute coronary syndromes

Journal of thrombosis and haemostasis, 2011-09, Vol.9 (9), p.1870-1873

2011

Volltextzugriff (PDF)

Thrombogenic and Fibrinolytic Factors and Cardiovascular Risk in Non-insulin-dependent Diabetes Mellitus

Annals of medicine (Helsinki), 1996, Vol.28 (4), p.371-380

1996

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Fat Cell Function and Fibrinolysis

Hormone and metabolic research, 2000-11, Vol.32 (11/12), p.504-508

2000

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Fibrinolytic factors and the risk of myocardial infarction or sudden death in patients with angina pectoris

Circulation (New York, N.Y.), 1996-11, Vol.94 (9), p.2057-2063

1996

Volltextzugriff (PDF)

Identification of polymorphisms in the promoter and the 3′ region of the TAFI gene: evidence that plasma TAFI antigen levels are strongly genetically controlled

Blood, 2001-04, Vol.97 (7), p.2053-2058

2001

Volltextzugriff (PDF)

Individualizing Nighttime Incontinence Care in Nursing Home Residents

Nursing research (New York), 1998-07, Vol.47 (4), p.197-204

1998

Volltextzugriff (PDF)

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High post‐treatment platelet reactivity identified low‐responders to dual antiplatelet therapy at increased risk of recurrent cardiovascular events after stenting for acute coronary syndrome

Assessment of epicardial fat volume and myocardial triglyceride content in severely obese subjects: relationship to metabolic profile, cardiac function and visceral fat

Endothelial cell markers and the risk of coronary heart disease: The prospective epidemiological study of myocardial infarction (PRIME) study

Genetic risk factors for venous thrombosis in women using combined oral contraceptives: update of the PILGRIM study

PAI-1, obesity, insulin resistance and risk of cardiovascular events

Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

Metabolic determinants are much more important than genetic polymorphisms in determining the PAI-1 activity and antigen plasma concentrations: a family study with part of the Stanislas Cohort

Epicardial Adipose Tissue Extent: Relationship With Age, Body Fat Distribution, and Coronaropathy

Increased plasma plasminogen activator inhibitor 1 levels. A possible link between insulin resistance and atherothrombosis

The use of wide-scale mental agility testing to identify people at risk of dementia: crucial or harmful?

Divergent effects of obesity on fragility fractures

Interaction between the C-260T polymorphism of the CD14 gene and the plasma IL-6 concentration on the risk of myocardial infarction: the HIFMECH study

The Association Between Chronic Illness and Functional Change Among Participants in a Comprehensive Geriatric Assessment Program

The A -844G polymorphism in the PAI-1 gene is associated with a higher risk of venous thrombosis in factor V Leiden carriers

The plasminogen activator inhibitor‐1 −675 4G/5G genotype influences the risk of myocardial infarction associated with elevated plasma proinsulin and insulin concentrations in men from Europe: the HIFMECH Study

Activated thrombin activatable fibrinolysis inhibitor levels are associated with the risk of cardiovascular death in patients with coronary artery disease: the AtheroGene study

Regulation of fibrinolysis in the development of atherothrombosis: role of adipose tissue

CD11b+ leukocyte microparticles are associated with high‐risk angiographic lesions and recurrent cardiovascular events in acute coronary syndromes

Thrombogenic and Fibrinolytic Factors and Cardiovascular Risk in Non-insulin-dependent Diabetes Mellitus

Fat Cell Function and Fibrinolysis

Fibrinolytic factors and the risk of myocardial infarction or sudden death in patients with angina pectoris

Identification of polymorphisms in the promoter and the 3′ region of the TAFI gene: evidence that plasma TAFI antigen levels are strongly genetically controlled

Individualizing Nighttime Incontinence Care in Nursing Home Residents

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