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Bradykinin increases blood-tumor barrier permeability by down-regulating the expression levels of ZO-1, occludin, and claudin-5 and rearranging actin cytoskeleton

Journal of neuroscience research, 2008-04, Vol.86 (5), p.1153-1168

2008

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Permeability of the blood-tumor barrier is enhanced by combining vascular endothelial growth factor with papaverine

Journal of neuroscience research, 2014-06, Vol.92 (6), p.703-713

2014

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Effects of Erythropoietin on Blood–Brain Barrier Tight Junctions in Ischemia–Reperfusion Rats

Journal of molecular neuroscience, 2013-02, Vol.49 (2), p.369-379

2013

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Inhibition of c-Jun N-terminal kinase prevents blood-brain barrier disruption and normalizes the expression of tight junction proteins clautin-5 and ZO-1 in a rat model of subarachnoid hemorrhage

Acta neurochirurgica, 2012-08, Vol.154 (8), p.1469-1476

2012

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Effects of combination treatment of dexamethasone and melatonin on brain injury in intracerebral hemorrhage model in rats

Brain research, 2009-04, Vol.1264, p.98-103

2009

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Effects of Poly (ADP-ribose) Polymerase Inhibitor 3-Aminobenzamide on Blood–Brain Barrier and Dopaminergic Neurons of Rats with Lipopolysaccharide-Induced Parkinson’s Disease

Journal of molecular neuroscience, 2014-05, Vol.53 (1), p.1-9

2014

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Low dose of bradykinin selectively increases intracellular calcium in glioma cells

Journal of the neurological sciences, 2007-07, Vol.258 (1), p.44-51

2007

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Role of ATP synthase alpha subunit in low-dose endothelial monocyte-activating polypeptide-II-induced opening of the blood–tumor barrier

Journal of the neurological sciences, 2011-01, Vol.300 (1), p.52-58

2011

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Bradykinin selectively modulates the blood-tumor barrier via calcium-induced calcium release

Journal of neuroscience research, 2009-02, Vol.87 (3), p.660-667

2009

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Bradykinin increased the permeability of BTB via NOS/NO/ZONAB-mediating down-regulation of claudin-5 and occludin

Biochemical and biophysical research communications, 2015-08, Vol.464 (1), p.118-125

2015

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Low-dose endothelial monocyte-activating polypeptide-II increases permeability of blood–tumor barrier via a PKC-ζ/PP2A-dependent signaling mechanism

Experimental cell research, 2015-02, Vol.331 (2), p.257-266

2015

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Endothelial-monocyte-activating polypeptide II induces rat C6 glioma cell apoptosis via the mitochondrial pathway

Biochemical and biophysical research communications, 2015-02, Vol.457 (4), p.595-601

2015

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Bradykinin increases the permeability of the blood-tumor barrier by the caveolae-mediated transcellular pathway

Journal of neuro-oncology, 2010-09, Vol.99 (2), p.187-194

2010

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Low-Frequency Ultrasound Irradiation Increases Blood–Tumor Barrier Permeability by Transcellular Pathway in a Rat Glioma Model

Journal of molecular neuroscience, 2012-09, Vol.48 (1), p.281-290

2012

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Low-dose Endothelial Monocyte-activating Polypeptide-II Increases Permeability of Blood–tumor Barrier by Caveolae-mediated Transcellular Pathway

Journal of molecular neuroscience, 2014-03, Vol.52 (3), p.313-322

2014

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Role of RhoA/ROCK Signaling in Endothelial-Monocyte-Activating Polypeptide II Opening of the Blood–Tumor Barrier: Role of RhoA/ROCK Signaling in EMAP II Opening of the BTB

Journal of molecular neuroscience, 2012-03, Vol.46 (3), p.666-676

2012

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Endothelial-monocyte-activating polypeptide II increases blood–tumor barrier permeability by down-regulating the expression levels of tight junction associated proteins

Brain research, 2010-03, Vol.1319, p.13-20

2010

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Roles of Serine/Threonine Phosphatases in Low-Dose Endothelial Monocyte-Activating Polypeptide-II-Induced Opening of Blood–Tumor Barrier

Journal of molecular neuroscience, 2015-09, Vol.57 (1), p.11-20

2015

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Glioma Cells Promote the Expression of Vascular Cell Adhesion Molecule-1 on Bone Marrow-Derived Mesenchymal Stem Cells: a Possible Mechanism for Their Tropism Toward Gliomas

Journal of molecular neuroscience, 2012-09, Vol.48 (1), p.127-135

2012

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Low-Dose Endothelial Monocyte-Activating Polypeptide-II Increases Blood–Tumor Barrier Permeability by Activating the RhoA/ROCK/PI3K Signaling Pathway

Journal of molecular neuroscience, 2016-06, Vol.59 (2), p.193-202

2016

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Functions for the cAMP/Epac/Rap1 Signaling Pathway in Low-Dose Endothelial Monocyte-Activating Polypeptide-II-Induced Opening of Blood–Tumor Barrier

Journal of molecular neuroscience, 2015-09, Vol.57 (1), p.1-10

2015

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Role of cAMP-Dependent Protein Kinase A Activity in Low-Dose Endothelial Monocyte-Activating Polypeptide-II-Induced Opening of Blood–Tumor Barrier

Journal of molecular neuroscience, 2015-05, Vol.56 (1), p.60-69

2015

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Mechanisms of the increase in the permeability of the blood–tumor barrier obtained by combining low-frequency ultrasound irradiation with small-dose bradykinin

Journal of neuro-oncology, 2009-08, Vol.94 (1), p.41-50

2009

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Low-Dose Endothelial Monocyte-Activating Polypeptide-II Induces Blood–Tumor Barrier Opening Via the cAMP/PKA/Rac1 Pathway

Journal of molecular neuroscience, 2016-02, Vol.58 (2), p.153-161

2016

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Platelet-derived growth factor BB mediates the glioma-induced migration of bone marrow-derived mesenchymal stem cells by promoting the expression of vascular cell adhesion molecule-1 through the PI3K, P38 MAPK and NF-κB pathways

Oncology reports, 2013-12, Vol.30 (6), p.2755-2764

2013

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Bradykinin increases blood-tumor barrier permeability by down-regulating the expression levels of ZO-1, occludin, and claudin-5 and rearranging actin cytoskeleton

Permeability of the blood-tumor barrier is enhanced by combining vascular endothelial growth factor with papaverine

Effects of Erythropoietin on Blood–Brain Barrier Tight Junctions in Ischemia–Reperfusion Rats

Inhibition of c-Jun N-terminal kinase prevents blood-brain barrier disruption and normalizes the expression of tight junction proteins clautin-5 and ZO-1 in a rat model of subarachnoid hemorrhage

Effects of combination treatment of dexamethasone and melatonin on brain injury in intracerebral hemorrhage model in rats

Effects of Poly (ADP-ribose) Polymerase Inhibitor 3-Aminobenzamide on Blood–Brain Barrier and Dopaminergic Neurons of Rats with Lipopolysaccharide-Induced Parkinson’s Disease

Low dose of bradykinin selectively increases intracellular calcium in glioma cells

Role of ATP synthase alpha subunit in low-dose endothelial monocyte-activating polypeptide-II-induced opening of the blood–tumor barrier

Bradykinin selectively modulates the blood-tumor barrier via calcium-induced calcium release

Bradykinin increased the permeability of BTB via NOS/NO/ZONAB-mediating down-regulation of claudin-5 and occludin

Low-dose endothelial monocyte-activating polypeptide-II increases permeability of blood–tumor barrier via a PKC-ζ/PP2A-dependent signaling mechanism

Endothelial-monocyte-activating polypeptide II induces rat C6 glioma cell apoptosis via the mitochondrial pathway

Bradykinin increases the permeability of the blood-tumor barrier by the caveolae-mediated transcellular pathway

Low-Frequency Ultrasound Irradiation Increases Blood–Tumor Barrier Permeability by Transcellular Pathway in a Rat Glioma Model

Low-dose Endothelial Monocyte-activating Polypeptide-II Increases Permeability of Blood–tumor Barrier by Caveolae-mediated Transcellular Pathway

Role of RhoA/ROCK Signaling in Endothelial-Monocyte-Activating Polypeptide II Opening of the Blood–Tumor Barrier: Role of RhoA/ROCK Signaling in EMAP II Opening of the BTB

Endothelial-monocyte-activating polypeptide II increases blood–tumor barrier permeability by down-regulating the expression levels of tight junction associated proteins

Roles of Serine/Threonine Phosphatases in Low-Dose Endothelial Monocyte-Activating Polypeptide-II-Induced Opening of Blood–Tumor Barrier

Glioma Cells Promote the Expression of Vascular Cell Adhesion Molecule-1 on Bone Marrow-Derived Mesenchymal Stem Cells: a Possible Mechanism for Their Tropism Toward Gliomas

Low-Dose Endothelial Monocyte-Activating Polypeptide-II Increases Blood–Tumor Barrier Permeability by Activating the RhoA/ROCK/PI3K Signaling Pathway

Functions for the cAMP/Epac/Rap1 Signaling Pathway in Low-Dose Endothelial Monocyte-Activating Polypeptide-II-Induced Opening of Blood–Tumor Barrier

Role of cAMP-Dependent Protein Kinase A Activity in Low-Dose Endothelial Monocyte-Activating Polypeptide-II-Induced Opening of Blood–Tumor Barrier

Mechanisms of the increase in the permeability of the blood–tumor barrier obtained by combining low-frequency ultrasound irradiation with small-dose bradykinin

Low-Dose Endothelial Monocyte-Activating Polypeptide-II Induces Blood–Tumor Barrier Opening Via the cAMP/PKA/Rac1 Pathway

Platelet-derived growth factor BB mediates the glioma-induced migration of bone marrow-derived mesenchymal stem cells by promoting the expression of vascular cell adhesion molecule-1 through the PI3K, P38 MAPK and NF-κB pathways

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