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Signal transduction and functional selectivity of F15599, a preferential post‐synaptic 5‐HT1A receptor agonist

British journal of pharmacology, 2009-01, Vol.156 (2), p.338-353

2009

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Aspirin and acetaminophen reduced both Fos expression in rat lumbar spinal cord and inflammatory signs produced by carrageenin inflammation

Pain (Amsterdam), 1995-12, Vol.63 (3), p.365-375

1995

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Physiological contributions of neurokinin 1 receptor activation, and interactions with NMDA receptors, to inflammatory-evoked spinal c-Fos expression

Journal of neurophysiology, 1996-09, Vol.76 (3), p.1817-1827

1996

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The contribution of NMDA receptor activation to spinal c‐Fos expression in a model of inflammatory pain

British journal of pharmacology, 1995-09, Vol.116 (1), p.1628-1634

1995

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The contribution of GABAB receptor-mediated events to inflammatory pain processing : Carrageenan oedema and associated spinal c-Fos expression in the rat

Neuroscience, 1996-07, Vol.73 (2), p.487-496

1996

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Dose-related anti-inflammatory/analgesic effects of lornoxicam: a spinal c-Fos protein study in the rat

Inflammation research, 1998-01, Vol.47 (1), p.18-25

1998

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Antinociceptive effects of RB101(S), a complete inhibitor of enkephalin-catabolizing enzymes, are enhanced by (+)-HA966, a functional NMDA receptor antagonist: a c-Fos study in the rat spinal cord

European journal of pain, 2003-01, Vol.7 (3), p.241-249

2003

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Intraplantar morphine depresses spinal c‐Fos expression induced by carrageenin inflammation but not by noxious heat

British journal of pharmacology, 1996-06, Vol.118 (3), p.671-680

1996

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Antinociceptive effects of RB101, a complete inhibitor of enkephalin-catabolizing enzymes, are enhanced by a cholecystokinin type B receptor antagonist, as revealed by noxiously evoked spinal c-Fos expression in rats

The Journal of pharmacology and experimental therapeutics, 1997-04, Vol.281 (1), p.208

1997

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To what extent do spinal interactions between an alpha-2 adrenoceptor agonist and a mu opioid agonist influence noxiously evoked c-Fos expression in the rat? A pharmacological study

The Journal of pharmacology and experimental therapeutics, 1996-07, Vol.278 (1), p.393

1996

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The contribution of peripheral bradykinin B2 receptors to carrageenan-evoked oedema and spinal c-Fos expression in rats

European journal of pharmacology, 1997-02, Vol.320 (1), p.73-80

1997

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Interactions between NMDA- and prostaglandin receptor-mediated events in a model of inflammatory nociception

European journal of pharmacology, 1996-05, Vol.303 (1), p.91-100

1996

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Effects of the high-efficacy 5-HT1A receptor agonist, F 13640 in the formalin pain model : A c-Fos study

European journal of pharmacology, 2005-05, Vol.514 (2-3), p.121-130

2005

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Concurrent reduction of inflammation and spinal Fos-LI neurons by systemic diclofenac in the rat

Neuroscience letters, 1995-03, Vol.188 (3), p.175-178

1995

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Cholecystokinin B receptor antagonism enhances the ability of a low dose of morphine to reduce c-Fos expression in the spinal cord of the rat

Neuroscience, 1995-08, Vol.67 (3), p.731-739

1995

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Effects of nefopam on the spinal nociceptive processes: a c-Fos protein study in the rat

European journal of pharmacology, 2002-04, Vol.441 (1), p.67-74

2002

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Effects of local anaesthetics on carrageenan-evoked inflammatory nociceptive processing in the rat

British journal of anaesthesia : BJA, 1996-11, Vol.77 (5), p.645-652

1996

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The novel analgesic and high-efficacy 5-HT1A receptor agonist, F 13640 induces c-Fos protein expression in spinal cord dorsal horn neurons

Brain research, 2003-06, Vol.974 (1-2), p.212-221

2003

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Involvement of the spinoparabrachial pathway in inflammatory nociceptive processes: A c-Fos protein study in the awake rat

Journal of comparative neurology (1911), 1998-07, Vol.397 (1), p.10-28

1998

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Carrageenin-evoked c-Fos expression in rat lumbar spinal cord: the effects of indomethacin

European journal of pharmacology, 1995-01, Vol.272 (2), p.249-259

1995

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Effects of flurbiprofen and its enantiomers on the spinal c-Fos protein expression induced by noxious heat stimuli in the anaesthetized rat

European journal of pharmacology, 2000-10, Vol.406 (1), p.59-67

2000

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Influence of carbamazepine and phenytoin on spontaneous activity of cerebellar neurons

Physiological research, 1994, Vol.43 (2), p.113-116

1994

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RB101(S), a dual inhibitor of enkephalinases does not induce antinociceptive tolerance, or cross-tolerance with morphine: a c-Fos study at the spinal level

European journal of pharmacology, 2002-04, Vol.441 (3), p.141-150

2002

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Urethane anaesthesia could partly mask antinociceptive effects of non-steroidal anti-inflammatory drugs: a spinal c-Fos protein study

Brain research, 2001-02, Vol.891 (1), p.281-284

2001

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Further evidence for the interaction of μ- and δ-opioid receptors in the antinociceptive effects of the dual inhibitor of enkephalin catabolism, RB101(S): A spinal c-Fos protein study in the rat under carrageenin inflammation

Brain research, 2003-03, Vol.967 (1), p.106-112

2003

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Signal transduction and functional selectivity of F15599, a preferential post‐synaptic 5‐HT1A receptor agonist

Aspirin and acetaminophen reduced both Fos expression in rat lumbar spinal cord and inflammatory signs produced by carrageenin inflammation

Physiological contributions of neurokinin 1 receptor activation, and interactions with NMDA receptors, to inflammatory-evoked spinal c-Fos expression

The contribution of NMDA receptor activation to spinal c‐Fos expression in a model of inflammatory pain

The contribution of GABAB receptor-mediated events to inflammatory pain processing : Carrageenan oedema and associated spinal c-Fos expression in the rat

Dose-related anti-inflammatory/analgesic effects of lornoxicam: a spinal c-Fos protein study in the rat

Antinociceptive effects of RB101(S), a complete inhibitor of enkephalin-catabolizing enzymes, are enhanced by (+)-HA966, a functional NMDA receptor antagonist: a c-Fos study in the rat spinal cord

Intraplantar morphine depresses spinal c‐Fos expression induced by carrageenin inflammation but not by noxious heat

Antinociceptive effects of RB101, a complete inhibitor of enkephalin-catabolizing enzymes, are enhanced by a cholecystokinin type B receptor antagonist, as revealed by noxiously evoked spinal c-Fos expression in rats

To what extent do spinal interactions between an alpha-2 adrenoceptor agonist and a mu opioid agonist influence noxiously evoked c-Fos expression in the rat? A pharmacological study

The contribution of peripheral bradykinin B2 receptors to carrageenan-evoked oedema and spinal c-Fos expression in rats

Interactions between NMDA- and prostaglandin receptor-mediated events in a model of inflammatory nociception

Effects of the high-efficacy 5-HT1A receptor agonist, F 13640 in the formalin pain model : A c-Fos study

Concurrent reduction of inflammation and spinal Fos-LI neurons by systemic diclofenac in the rat

Cholecystokinin B receptor antagonism enhances the ability of a low dose of morphine to reduce c-Fos expression in the spinal cord of the rat

Effects of nefopam on the spinal nociceptive processes: a c-Fos protein study in the rat

Effects of local anaesthetics on carrageenan-evoked inflammatory nociceptive processing in the rat

The novel analgesic and high-efficacy 5-HT1A receptor agonist, F 13640 induces c-Fos protein expression in spinal cord dorsal horn neurons

Involvement of the spinoparabrachial pathway in inflammatory nociceptive processes: A c-Fos protein study in the awake rat

Carrageenin-evoked c-Fos expression in rat lumbar spinal cord: the effects of indomethacin

Effects of flurbiprofen and its enantiomers on the spinal c-Fos protein expression induced by noxious heat stimuli in the anaesthetized rat

Influence of carbamazepine and phenytoin on spontaneous activity of cerebellar neurons

RB101(S), a dual inhibitor of enkephalinases does not induce antinociceptive tolerance, or cross-tolerance with morphine: a c-Fos study at the spinal level

Urethane anaesthesia could partly mask antinociceptive effects of non-steroidal anti-inflammatory drugs: a spinal c-Fos protein study

Further evidence for the interaction of μ- and δ-opioid receptors in the antinociceptive effects of the dual inhibitor of enkephalin catabolism, RB101(S): A spinal c-Fos protein study in the rat under carrageenin inflammation

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