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N- and L-Type Voltage-Dependent Ca2+ Channels Contribute to the Generation of After-Discharges in the Spinal Ventral Root After Cessation of Noxious Mechanical Stimulation

Journal of Pharmacological Sciences, 2012, Vol.119(1), pp.82-90

2012

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Involvement of Supraspinal Imidazoline Receptors and Descending Monoaminergic Pathways in Tizanidine-Induced Inhibition of Rat Spinal Reflexes

Journal of Pharmacological Sciences, 2005, Vol.99(1), pp.52-60

2005

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L-Type Ca2+ Channels Mediate the Slow Ca2+-Dependent Afterhyperpolarization Current in Rat CA3 Pyramidal Cells In Vitro

Journal of neurophysiology, 1998-11, Vol.80 (5), p.2268-2273

1998

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GABAB Receptors Do Not Mediate the Inhibitory Actions of Gabapentin on the Spinal Reflex in Rats

Journal of Pharmacological Sciences, 2004, Vol.96(4), pp.444-449

2004

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Presynaptic I1-Imidazoline Receptors Reduce GABAergic Synaptic Transmission in Striatal Medium Spiny Neurons

The Journal of neuroscience, 2006-02, Vol.26 (6), p.1795-1802

2006

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Inhibition of hyperpolarization-activated cation currents by phencyclidine and some sigma ligands in rat hippocampal CA1 pyramidal neurons in vitro

Neuropharmacology, 2007-09, Vol.53 (3), p.406-414

Tanabe, Mitsuo

2007

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Spinal ventral root after-discharges as a pain index: Involvement of NK-1 and NMDA receptors

Brain research, 2006-04, Vol.1082 (1), p.115-123

2006

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Endogenous GABA Does Not Mediate the Inhibitory Effects of Gabapentin on Spinal Reflexes in Rats

Journal of Pharmacological Sciences, 2004, Vol.94(2), pp.137-143

2004

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Effects of milnacipran, a 5‐HT and noradrenaline reuptake inhibitor, on C‐fibre‐evoked field potentials in spinal long‐term potentiation and neuropathic pain

British journal of pharmacology, 2012-10, Vol.167 (3), p.537-547

2012

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Gabapentin depresses C-fiber-evoked field potentials in rat spinal dorsal horn only after induction of long-term potentiation

Experimental neurology, 2006-12, Vol.202 (2), p.280-286

2006

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Phenytoin and carbamazepine delay the initial depression of the population spike upon exposure to in vitro ischemia and promote its post-ischemic functional recovery in rat hippocampal slices

European journal of pharmacology, 2006-12, Vol.553 (1), p.104-108

2006

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Spinal cord injury-specific depression of monosynaptic spinal reflex transmission by l-5-hydroxytryptophan results from loss of the 5-HT uptake system and not 5-HT receptor supersensitivity

Experimental neurology, 2006-11, Vol.202 (1), p.258-261

2006

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Endogenously released 5-hydroxytryptamine depresses the spinal monosynaptic reflex via 5-HT1D receptors

European journal of pharmacology, 2004-10, Vol.503 (1-3), p.55-61

2004

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Serotonergic depression of spinal monosynaptic transmission is mediated by 5-HT1B receptors

European journal of pharmacology, 2003-12, Vol.482 (1-3), p.155-161

2003

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Functional alteration of inhibitory influences on spinal motor output in painful diabetic neuropathy in rats

Neuroscience letters, 2005-12, Vol.389 (3), p.152-156

2005

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NMDA Receptors in the Spinal Cord Exert Excitatory Influences on Spinal Motor Output in Rats

The Japanese Journal of Pharmacology, 2001, Vol.87(4), pp.343-346

2001

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Apamin-Sensitive Conductance Mediates the K+ Current Response During Chemical Ischemia in CA3 Pyramidal Cells

Journal of neurophysiology, 1999-12, Vol.82 (6), p.2876-2882

1999

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Paired pulse facilitation of GABAergic IPSCs in ventral horn neurons in neonatal rat spinal cord

Brain research, 1996-04, Vol.716 (1), p.101-106

1996

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Preferential enhancement of inhibitory synaptic transmission by CS-722 in the ventral horn neurons of neonatal rat spinal cord

European journal of pharmacology, 1996-10, Vol.313 (3), p.187-189

1996

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Sites of Action of CS-722, a Newly Synthesized Centrally Acting Muscle Relaxant

The Japanese Journal of Pharmacology, 1993, Vol.63(3), pp.385-389

1993

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Local Cerebral Glucose Utilization in Rats with Decerebrate Rigidity and Effects of Centrally Acting Muscle Relaxants, Diazepam and Tizanidine

Biological and Pharmaceutical Bulletin, 1993/01/15, Vol.16(1), pp.33-35

1993

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Picolinic acid and indole-2-carboxylic acid: two types of glycinergic compounds modulate motor function differentially

General pharmacology, 1997-04, Vol.28 (4), p.555-560

1997

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Exercise training improves cardiac function-related gene levels through thyroid hormone receptor signaling in aged rats

American journal of physiology. Heart and circulatory physiology, 2004-05, Vol.286 (5), p.H1696-H1705

2004

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Aging-induced decrease in the PPAR-alpha level in hearts is improved by exercise training

American journal of physiology. Heart and circulatory physiology, 2002-11, Vol.283 (5), p.H1750-H1760

2002

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Effect of an omega-3 lipid emulsion in reducing oxidative stress in a rat model of intestinal ischemia−reperfusion injury

Pediatric surgery international, 2012-09, Vol.28 (9), p.913-918

2012

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N- and L-Type Voltage-Dependent Ca2+ Channels Contribute to the Generation of After-Discharges in the Spinal Ventral Root After Cessation of Noxious Mechanical Stimulation

Involvement of Supraspinal Imidazoline Receptors and Descending Monoaminergic Pathways in Tizanidine-Induced Inhibition of Rat Spinal Reflexes

L-Type Ca2+ Channels Mediate the Slow Ca2+-Dependent Afterhyperpolarization Current in Rat CA3 Pyramidal Cells In Vitro

GABAB Receptors Do Not Mediate the Inhibitory Actions of Gabapentin on the Spinal Reflex in Rats

Presynaptic I1-Imidazoline Receptors Reduce GABAergic Synaptic Transmission in Striatal Medium Spiny Neurons

Inhibition of hyperpolarization-activated cation currents by phencyclidine and some sigma ligands in rat hippocampal CA1 pyramidal neurons in vitro

Spinal ventral root after-discharges as a pain index: Involvement of NK-1 and NMDA receptors

Endogenous GABA Does Not Mediate the Inhibitory Effects of Gabapentin on Spinal Reflexes in Rats

Effects of milnacipran, a 5‐HT and noradrenaline reuptake inhibitor, on C‐fibre‐evoked field potentials in spinal long‐term potentiation and neuropathic pain

Gabapentin depresses C-fiber-evoked field potentials in rat spinal dorsal horn only after induction of long-term potentiation

Phenytoin and carbamazepine delay the initial depression of the population spike upon exposure to in vitro ischemia and promote its post-ischemic functional recovery in rat hippocampal slices

Spinal cord injury-specific depression of monosynaptic spinal reflex transmission by l-5-hydroxytryptophan results from loss of the 5-HT uptake system and not 5-HT receptor supersensitivity

Endogenously released 5-hydroxytryptamine depresses the spinal monosynaptic reflex via 5-HT1D receptors

Serotonergic depression of spinal monosynaptic transmission is mediated by 5-HT1B receptors

Functional alteration of inhibitory influences on spinal motor output in painful diabetic neuropathy in rats

NMDA Receptors in the Spinal Cord Exert Excitatory Influences on Spinal Motor Output in Rats

Apamin-Sensitive Conductance Mediates the K+ Current Response During Chemical Ischemia in CA3 Pyramidal Cells

Paired pulse facilitation of GABAergic IPSCs in ventral horn neurons in neonatal rat spinal cord

Preferential enhancement of inhibitory synaptic transmission by CS-722 in the ventral horn neurons of neonatal rat spinal cord

Sites of Action of CS-722, a Newly Synthesized Centrally Acting Muscle Relaxant

Local Cerebral Glucose Utilization in Rats with Decerebrate Rigidity and Effects of Centrally Acting Muscle Relaxants, Diazepam and Tizanidine

Picolinic acid and indole-2-carboxylic acid: two types of glycinergic compounds modulate motor function differentially

Exercise training improves cardiac function-related gene levels through thyroid hormone receptor signaling in aged rats

Aging-induced decrease in the PPAR-alpha level in hearts is improved by exercise training

Effect of an omega-3 lipid emulsion in reducing oxidative stress in a rat model of intestinal ischemia−reperfusion injury

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