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The α-Ketoglutarate Dehydrogenase Complex as a Hub of Plasticity in Neurodegeneration and Regeneration

International journal of molecular sciences, 2022-10, Vol.23 (20), p.12403

2022

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Up‐regulation of the mitochondrial malate dehydrogenase by oxidative stress is mediated by miR‐743a

Journal of neurochemistry, 2011-08, Vol.118 (3), p.440-448

2011

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Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Biochimica et biophysica acta, 2010-01, Vol.1802 (1), p.122-134

2010

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Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Human molecular genetics, 2018-08, Vol.27 (16), p.2874-2892

2018

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Benfotiamine and Cognitive Decline in Alzheimer's Disease: Results of a Randomized Placebo-Controlled Phase IIa Clinical Trial

Journal of Alzheimer's disease, 2020-01, Vol.78 (3), p.989

2020

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Oxidative Stress and Transcriptional Regulation in Alzheimer Disease

Alzheimer disease and associated disorders, 2007-10, Vol.21 (4), p.276-291

2007

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Interactions of Mitochondria/Metabolism and Calcium Regulation in Alzheimer’s Disease: A Calcinist Point of View

Neurochemical research, 2017-06, Vol.42 (6), p.1636-1648

2017

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Changes in inflammatory processes associated with selective vulnerability following mild impairment of oxidative metabolism

Neurobiology of disease, 2007-05, Vol.26 (2), p.353-362

2007

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Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Neurochemical research, 2010-12, Vol.35 (12), p.2107-2116

2010

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A mitocentric view of Alzheimer's disease suggests multi-faceted treatments

Journal of Alzheimer's disease, 2010-01, Vol.20 Suppl 2, p.S591

2010

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Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism

Journal of neurochemistry, 2001-02, Vol.76 (4), p.1010-1021

2001

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Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

Basic research in cardiology, 2020-09, Vol.115 (5), p.53, Article 53

2020

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Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease

Neurochemistry international, 2009-02, Vol.54 (2), p.111-118

2009

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Reductions in the mitochondrial enzyme α‐ketoglutarate dehydrogenase complex in neurodegenerative disease – beneficial or detrimental?

Journal of neurochemistry, 2016-12, Vol.139 (5), p.823-838

2016

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Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Journal of neurochemistry, 2016-12, Vol.139 (5), p.823

2016

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Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

The Journal of biological chemistry, 2011-05, Vol.286 (20), p.17640-17648

2011

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Thiamine-dependent processes and treatment strategies in neurodegeneration

Antioxidants & redox signaling, 2007-10, Vol.9 (10), p.1605

2007

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Oxidant-induced Changes in Mitochondria and Calcium Dynamics in the Pathophysiology of Alzheimer's Disease

Annals of the New York Academy of Sciences, 2008-12, Vol.1147 (1), p.221-232

2008

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Interactions of oxidative stress with thiamine homeostasis promote neurodegeneration

Neurochemistry international, 2002-05, Vol.40 (6), p.493-504

2002

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The alpha-ketoglutarate-dehydrogenase complex: a mediator between mitochondria and oxidative stress in neurodegeneration

Molecular neurobiology, 2005, Vol.31 (1-3), p.43-64

2005

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Novel functions of the α-ketoglutarate dehydrogenase complex may mediate diverse oxidant-induced changes in mitochondrial enzymes associated with Alzheimer's disease

Biochimica et biophysica acta, 2008-04, Vol.1782 (4), p.229-238

2008

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Interactions of oxidative stress with cellular calcium dynamics and glucose metabolism in Alzheimer's disease

Free radical biology & medicine, 2002-06, Vol.32 (11), p.1061-1070

Gibson, Gary E

2002

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Induction of Nitric Oxide Synthase and Microglial Responses Precede Selective Cell Death Induced by Chronic Impairment of Oxidative Metabolism

The American journal of pathology, 1998-08, Vol.153 (2), p.599-610

1998

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Impaired mitochondrial energy metabolism as a novel risk factor for selective onset and progression of dementia in oldest-old subjects

Neuropsychiatric disease and treatment, 2015-01, Vol.11 (default), p.565-574

2015

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Vascular Factors Are Critical in Selective Neuronal Loss in an Animal Model of Impaired Oxidative Metabolism

Journal of neuropathology and experimental neurology, 2000-03, Vol.59 (3), p.207-217

2000

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The α-Ketoglutarate Dehydrogenase Complex as a Hub of Plasticity in Neurodegeneration and Regeneration

Up‐regulation of the mitochondrial malate dehydrogenase by oxidative stress is mediated by miR‐743a

Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Benfotiamine and Cognitive Decline in Alzheimer's Disease: Results of a Randomized Placebo-Controlled Phase IIa Clinical Trial

Oxidative Stress and Transcriptional Regulation in Alzheimer Disease

Interactions of Mitochondria/Metabolism and Calcium Regulation in Alzheimer’s Disease: A Calcinist Point of View

Changes in inflammatory processes associated with selective vulnerability following mild impairment of oxidative metabolism

Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

A mitocentric view of Alzheimer's disease suggests multi-faceted treatments

Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism

Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease

Reductions in the mitochondrial enzyme α‐ketoglutarate dehydrogenase complex in neurodegenerative disease – beneficial or detrimental?

Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

Thiamine-dependent processes and treatment strategies in neurodegeneration

Oxidant-induced Changes in Mitochondria and Calcium Dynamics in the Pathophysiology of Alzheimer's Disease

Interactions of oxidative stress with thiamine homeostasis promote neurodegeneration

The alpha-ketoglutarate-dehydrogenase complex: a mediator between mitochondria and oxidative stress in neurodegeneration

Novel functions of the α-ketoglutarate dehydrogenase complex may mediate diverse oxidant-induced changes in mitochondrial enzymes associated with Alzheimer's disease

Interactions of oxidative stress with cellular calcium dynamics and glucose metabolism in Alzheimer's disease

Induction of Nitric Oxide Synthase and Microglial Responses Precede Selective Cell Death Induced by Chronic Impairment of Oxidative Metabolism

Impaired mitochondrial energy metabolism as a novel risk factor for selective onset and progression of dementia in oldest-old subjects

Vascular Factors Are Critical in Selective Neuronal Loss in an Animal Model of Impaired Oxidative Metabolism

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