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3D human brain cell models: New frontiers in disease understanding and drug discovery for neurodegenerative diseases

Neurochemistry international, 2018-11, Vol.120, p.191-199

2018

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3D in vitro modelling of human patient microglia: A focus on clinical translation and drug development in neurodegenerative diseases

Journal of neuroimmunology, 2023-02, Vol.375, p.578017, Article 578017

2023

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Abnormal Function of Metalloproteins Underlies Most Neurodegenerative Diseases

Biometals in Neurodegenerative Diseases, 2017

2017

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The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

Experimental neurology, 2018-09, Vol.307, p.118-128

2018

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Acetylcholinesterase is increased in mouse neuronal and astrocyte cultures after treatment with β-amyloid peptides

Brain research, 2003-03, Vol.965 (1), p.283-286

2003

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Advanced patient-specific microglia cell models for pre-clinical studies in Alzheimer's disease

Journal of neuroinflammation, 2024-02, Vol.21 (1), p.50-50, Article 50

2024

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Advances in the Development of Disease-Modifying Treatments for Amyotrophic Lateral Sclerosis

CNS drugs, 2016-03, Vol.30 (3), p.227-243

2016

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ALS monocyte-derived microglia-like cells reveal cytoplasmic TDP-43 accumulation, DNA damage, and cell-specific impairment of phagocytosis associated with disease progression

Journal of neuroinflammation, 2022-02, Vol.19 (1), p.58-58, Article 58

2022

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Alzheimer’s disease amyloid beta and prion protein amyloidogenic peptides promote macrophage survival, DNA synthesis and enhanced proliferative response to CSF-1 (M-CSF)

Brain research, 2002-06, Vol.940 (1), p.49-54

2002

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The Alzheimer's Disease Amyloid Precursor Protein Modulates Copper-Induced Toxicity and Oxidative Stress in Primary Neuronal Cultures

The Journal of neuroscience, 1999-11, Vol.19 (21), p.9170-9179

1999

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Alzheimer's disease brain endothelial-like cells reveal differential drug transporter expression and modulation by potentially therapeutic focused ultrasound

Neurotherapeutics, 2024-01, Vol.21 (1), p.e00299-e00299, Article e00299

2024

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The Alzheimer’s therapeutic PBT2 promotes amyloid‐β degradation and GSK3 phosphorylation via a metal chaperone activity

Journal of neurochemistry, 2011-10, Vol.119 (1), p.220-230

2011

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Biometals and Alzheimer’s Disease

Biometals in Neurodegenerative Diseases, 2017

2017

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Biometals in Neurodegenerative Diseases: Mechanisms and Therapeutics

1, 2017

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Biometals in rare neurodegenerative disorders of childhood

Frontiers in aging neuroscience, 2013, Vol.5, p.14-14

2013

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Bis(thiosemicarbazonato) Cu-64 complexes for positron emission tomography imaging of Alzheimer's disease

Journal of Alzheimer's disease, 2010-01, Vol.20 (1), p.49

2010

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Blood-borne amyloid-beta dimer correlates with clinical markers of Alzheimer's disease

The Journal of neuroscience, 2010-05, Vol.30 (18), p.6315-6322

2010

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C-Jun N-terminal kinase controls TDP-43 accumulation in stress granules induced by oxidative stress

Molecular neurodegeneration, 2011-08, Vol.6 (1), p.57-57, Article 57

2011

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Categories of cortical structure

Progress in Brain Research, 1994, Vol.102, p.343-355

1994

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Changes in 5-HT7 serotonin receptor mRNA expression with aging in rat brain

Brain research. Molecular brain research., 2000-06, Vol.79 (1-2), p.163-168

2000

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Characterization of cellular senescence in aging skeletal muscle

Nature aging, 2022-07, Vol.2 (7), p.601

2022

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Chemical Elements and Oxidative Status in Neuroinflammation

Biometals in Neurodegenerative Diseases, 2017

2017

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Contrasting, Species-Dependent Modulation of Copper-Mediated Neurotoxicity by the Alzheimer's Disease Amyloid Precursor Protein

The Journal of neuroscience, 2002-01, Vol.22 (2), p.365-376

2002

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Contributors

Biometals in Neurodegenerative Diseases, 2017

2017

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The Copper bis(thiosemicarbazone) Complex Cu^sup II^(atsm) Is Protective Against Cerebral Ischemia Through Modulation of the Inflammatory Milieu

Neurotherapeutics, 2017-04, Vol.14 (2), p.519

2017

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3D human brain cell models: New frontiers in disease understanding and drug discovery for neurodegenerative diseases

3D in vitro modelling of human patient microglia: A focus on clinical translation and drug development in neurodegenerative diseases

Abnormal Function of Metalloproteins Underlies Most Neurodegenerative Diseases

The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

Acetylcholinesterase is increased in mouse neuronal and astrocyte cultures after treatment with β-amyloid peptides

Advanced patient-specific microglia cell models for pre-clinical studies in Alzheimer's disease

Advances in the Development of Disease-Modifying Treatments for Amyotrophic Lateral Sclerosis

ALS monocyte-derived microglia-like cells reveal cytoplasmic TDP-43 accumulation, DNA damage, and cell-specific impairment of phagocytosis associated with disease progression

Alzheimer’s disease amyloid beta and prion protein amyloidogenic peptides promote macrophage survival, DNA synthesis and enhanced proliferative response to CSF-1 (M-CSF)

The Alzheimer's Disease Amyloid Precursor Protein Modulates Copper-Induced Toxicity and Oxidative Stress in Primary Neuronal Cultures

Alzheimer's disease brain endothelial-like cells reveal differential drug transporter expression and modulation by potentially therapeutic focused ultrasound

The Alzheimer’s therapeutic PBT2 promotes amyloid‐β degradation and GSK3 phosphorylation via a metal chaperone activity

Biometals and Alzheimer’s Disease

Biometals in Neurodegenerative Diseases: Mechanisms and Therapeutics

Biometals in rare neurodegenerative disorders of childhood

Bis(thiosemicarbazonato) Cu-64 complexes for positron emission tomography imaging of Alzheimer's disease

Blood-borne amyloid-beta dimer correlates with clinical markers of Alzheimer's disease

C-Jun N-terminal kinase controls TDP-43 accumulation in stress granules induced by oxidative stress

Categories of cortical structure

Changes in 5-HT7 serotonin receptor mRNA expression with aging in rat brain

Characterization of cellular senescence in aging skeletal muscle

Chemical Elements and Oxidative Status in Neuroinflammation

Contrasting, Species-Dependent Modulation of Copper-Mediated Neurotoxicity by the Alzheimer's Disease Amyloid Precursor Protein

Contributors

The Copper bis(thiosemicarbazone) Complex Cu^sup II^(atsm) Is Protective Against Cerebral Ischemia Through Modulation of the Inflammatory Milieu

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