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Extracellular signal-regulated mitogen-activated protein kinase inhibitors decrease amphetamine-induced behavior and neuropeptide gene expression in the striatum

Neuroscience, 2006, Vol.138 (4), p.1289-1298

2006

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Amphetamine up-regulates activator of G-protein signaling 1 mRNA and protein levels in rat frontal cortex: the role of dopamine and glucocorticoid receptors

Neuroscience, 2010-06, Vol.168 (1), p.96-107

2010

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Relapse to cocaine seeking increases activity-regulated gene expression differentially in the prefrontal cortex of abstinent rats

Psychopharmacologia, 2008-05, Vol.198 (1), p.77-91

2008

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Context-driven cocaine-seeking in abstinent rats increases activity-regulated gene expression in the basolateral amygdala and dorsal hippocampus differentially following short and long periods of abstinence

Neuroscience, 2010-10, Vol.170 (2), p.570-579

2010

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Oxytocin Reduces Cocaine Seeking and Reverses Chronic Cocaine-Induced Changes in Glutamate Receptor Function

The international journal of neuropsychopharmacology, 2015-01, Vol.18 (1), p.pyu009-pyu009

2015

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Amphetamine‐induced locomotion and gene expression are altered in BDNF heterozygous mice

Genes, brain and behavior, 2008-11, Vol.7 (8), p.906-914

2008

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Relapse to cocaine-seeking increases activity-regulated gene expression differentially in the striatum and cerebral cortex of rats following short or long periods of abstinence

Brain Structure and Function, 2008-09, Vol.213 (1-2), p.215-227

2008

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Fine‐tuning our understanding of the baroreflex: size‐dependent regulation of action potential subpopulations

The Journal of physiology, 2020-06, Vol.598 (12), p.2291-2292

2020

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Effects of brain‐derived neurotrophic factor on dopaminergic function and motor behavior during aging

Genes, brain and behavior, 2011-03, Vol.10 (2), p.186-198

2011

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Kappa opioid receptor immunoreactivity in the nucleus accumbens and caudate–putamen is primarily associated with synaptic vesicles in axons

Neuroscience, 2000, Vol.96 (1), p.91-99

2000

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The κ-opioid agonist, U-69593, decreases acute amphetamine-evoked behaviors and calcium-dependent dialysate levels of dopamine and glutamate in the ventral striatum

Journal of neurochemistry, 1999-09, Vol.73 (3), p.1066-1074

1999

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A single injection of amphetamine or methamphetamine induces dynamic alterations in c- fos, zif/268 and preprodynorphin messenger RNA expression in rat forebrain

Neuroscience, 1995-09, Vol.68 (1), p.83-95

1995

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N-Methyl- d-aspartate receptors and p38 mitogen-activated protein kinase are required for cAMP-dependent cyclase response element binding protein and Elk-1 phosphorylation in the striatum

Neuroscience, 2000-01, Vol.101 (3), p.607-617

2000

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Cyclic AMP and mitogen‐activated protein kinases are required for glutamate‐dependent cyclic AMP response element binding protein and Elk‐1 phosphorylation in the dorsal striatum in vivo

Journal of neurochemistry, 2001-01, Vol.76 (2), p.401-412

2001

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The role of kainic acid/AMPA and metabotropic glutamate receptors in the regulation of opioid mRNA expression and the onset of pain-related behavior following excitotoxic spinal cord injury

Neuroscience, 2001-01, Vol.104 (3), p.863-874

2001

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Repeated amphetamine administration induces a prolonged augmentation of phosphorylated cyclase response element-binding protein and Fos-related antigen immunoreactivity in rat striatum

Neuroscience, 1995-11, Vol.69 (2), p.441-457

1995

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Muscarinic receptors regulate striatal neuropeptide gene expression in normal and amphetamine-treated rats

Neuroscience, 1996-11, Vol.75 (1), p.43-56

1996

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Delta opioid receptors regulate calcium-dependent, amphetamine-evoked glutamate levels in the rat striatum: an in vivo microdialysis study

Brain research, 2000-04, Vol.861 (2), p.296-304

2000

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Translocation of zinc may contribute to seizure-induced death of neurons

Brain research, 1989-02, Vol.480 (1), p.317-321

1989

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Local μ and δ opioid receptors regulate amphetamine-induced behavior and neuropeptide mRNA in the striatum

Neuroscience, 2003-01, Vol.121 (2), p.387-398

2003

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Repeated cocaine administration alters the expression of genes in corticolimbic circuitry after a 3‐week withdrawal: a DNA macroarray study

Journal of neurochemistry, 2002-09, Vol.82 (5), p.1290-1299

2002

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Opioid peptide messenger RNA expression is increased at spinal and supraspinal levels following excitotoxic spinal cord injury

Neuroscience, 2000-01, Vol.99 (1), p.189-197

2000

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Presynaptic κ-opioid and muscarinic receptors inhibit the calcium-dependent component of evoked glutamate release from striatal synaptosomes

Journal of neurochemistry, 1999-09, Vol.73 (3), p.1058-1065

1999

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Spinal and supraspinal changes in opioid mRNA expression are related to the onset of pain behaviors following excitotoxic spinal cord injury

Pain (Amsterdam), 2001-02, Vol.90 (1), p.181-190

2001

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κ Receptor Activation Attenuates l‐trans‐Pyrrolidine‐2,4‐Dicarboxylic Acid‐Evoked Glutamate Levels in the Striatum

Journal of neurochemistry, 1998-02, Vol.70 (2), p.626-634

1998

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Extracellular signal-regulated mitogen-activated protein kinase inhibitors decrease amphetamine-induced behavior and neuropeptide gene expression in the striatum

Amphetamine up-regulates activator of G-protein signaling 1 mRNA and protein levels in rat frontal cortex: the role of dopamine and glucocorticoid receptors

Relapse to cocaine seeking increases activity-regulated gene expression differentially in the prefrontal cortex of abstinent rats

Context-driven cocaine-seeking in abstinent rats increases activity-regulated gene expression in the basolateral amygdala and dorsal hippocampus differentially following short and long periods of abstinence

Oxytocin Reduces Cocaine Seeking and Reverses Chronic Cocaine-Induced Changes in Glutamate Receptor Function

Amphetamine‐induced locomotion and gene expression are altered in BDNF heterozygous mice

Relapse to cocaine-seeking increases activity-regulated gene expression differentially in the striatum and cerebral cortex of rats following short or long periods of abstinence

Fine‐tuning our understanding of the baroreflex: size‐dependent regulation of action potential subpopulations

Effects of brain‐derived neurotrophic factor on dopaminergic function and motor behavior during aging

Kappa opioid receptor immunoreactivity in the nucleus accumbens and caudate–putamen is primarily associated with synaptic vesicles in axons

The κ-opioid agonist, U-69593, decreases acute amphetamine-evoked behaviors and calcium-dependent dialysate levels of dopamine and glutamate in the ventral striatum

A single injection of amphetamine or methamphetamine induces dynamic alterations in c- fos, zif/268 and preprodynorphin messenger RNA expression in rat forebrain

N-Methyl- d-aspartate receptors and p38 mitogen-activated protein kinase are required for cAMP-dependent cyclase response element binding protein and Elk-1 phosphorylation in the striatum

Cyclic AMP and mitogen‐activated protein kinases are required for glutamate‐dependent cyclic AMP response element binding protein and Elk‐1 phosphorylation in the dorsal striatum in vivo

The role of kainic acid/AMPA and metabotropic glutamate receptors in the regulation of opioid mRNA expression and the onset of pain-related behavior following excitotoxic spinal cord injury

Repeated amphetamine administration induces a prolonged augmentation of phosphorylated cyclase response element-binding protein and Fos-related antigen immunoreactivity in rat striatum

Muscarinic receptors regulate striatal neuropeptide gene expression in normal and amphetamine-treated rats

Delta opioid receptors regulate calcium-dependent, amphetamine-evoked glutamate levels in the rat striatum: an in vivo microdialysis study

Translocation of zinc may contribute to seizure-induced death of neurons

Local μ and δ opioid receptors regulate amphetamine-induced behavior and neuropeptide mRNA in the striatum

Repeated cocaine administration alters the expression of genes in corticolimbic circuitry after a 3‐week withdrawal: a DNA macroarray study

Opioid peptide messenger RNA expression is increased at spinal and supraspinal levels following excitotoxic spinal cord injury

Presynaptic κ-opioid and muscarinic receptors inhibit the calcium-dependent component of evoked glutamate release from striatal synaptosomes

Spinal and supraspinal changes in opioid mRNA expression are related to the onset of pain behaviors following excitotoxic spinal cord injury

κ Receptor Activation Attenuates l‐trans‐Pyrrolidine‐2,4‐Dicarboxylic Acid‐Evoked Glutamate Levels in the Striatum

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