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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

Brain (London, England : 1878), 2005-08, Vol.128 (8), p.1847-1854

2005

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International Spinal Cord Injury Pain Classification: part I. Background and description

Spinal cord, 2012-06, Vol.50 (6), p.413-417

2012

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Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

Neurology, 2012-05, Vol.78 (21), p.1635

2012

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International Spinal Cord Injury Pain (ISCIP) Classification: Part 2. Initial validation using vignettes

Spinal cord, 2012-06, Vol.50 (6), p.404-412

2012

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Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Experimental neurology, 2014-03, Vol.253, p.174-179

2014

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Intravenous administration of auto serum-expanded autologous mesenchymal stem cells in stroke

Brain (London, England : 1878), 2011-06, Vol.134 (Pt 6), p.1790-1807

2011

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A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities

Neuroscience, 2001-01, Vol.102 (2), p.307-317

2001

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Sodium Channels in Normal and Pathological Pain

Annual review of neuroscience, 2010-01, Vol.33 (1), p.325-347

2010

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Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination

Brain (London, England : 1878), 2003-07, Vol.126 (7), p.1552-1561

2003

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Activated polymorphonuclear cells promote injury and excitability of dorsal root ganglia neurons

Experimental neurology, 2008-04, Vol.210 (2), p.286-294

2008

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Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

Pain (Amsterdam), 1999-12, Vol.83 (3), p.591-600

1999

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Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5

Brain (London, England : 1878), 2010-03, Vol.133 (3), p.835-846

2010

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Molecular and Functional Remodeling of Electrogenic Membrane of Hypothalamic Neurons in Response to Changes in Their Input

Proceedings of the National Academy of Sciences - PNAS, 1999-02, Vol.96 (3), p.1088-1093

1999

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The pentapeptide QYNAD does not block voltage-gated sodium channels

Neurology, 2003-01, Vol.60 (2), p.224-229

2003

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Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar purkinje cells

Brain research, 2003-01, Vol.959 (2), p.235-242

2003

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Physiological and genetic analysis of multiple sodium channel variants in a model of genetic absence epilepsy

Neurobiology of disease, 2014-07, Vol.67, p.180-190

2014

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Preferential expression of IGF-I in small DRG neurons and down-regulation following injury

Neuroreport, 2002-09, Vol.13 (13), p.1649-1652

2002

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Tapered withdrawal of phenytoin removes protective effect in EAE without inflammatory rebound and mortality

Journal of the neurological sciences, 2014-06, Vol.341 (1), p.8-12

2014

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Acquired channelopathies in nerve injury and MS

Neurology, 2001-06, Vol.56 (12), p.1621-1627

Waxman, S G

2001

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Characterizing the mechanisms of progression in multiple sclerosis: evidence and new hypotheses for future directions

Archives of neurology (Chicago), 2005-09, Vol.62 (9), p.1345

2005

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Dysregulation of sodium channel expression in cortical neurons in a rodent model of absence epilepsy

Brain research, 2004-03, Vol.1000 (1), p.102-109

2004

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Na+-Ca2+ exchanger mediates Ca2+ influx during anoxia in mammalian central nervous system white matter

Annals of neurology, 1991-09, Vol.30 (3), p.375-380

1991

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Demyelination in spinal cord injury

Journal of the Neurological Sciences, 1989-06, Vol.91 (1), p.1-14

Waxman, Stephen G.

1989

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Sodium channel blockers and axonal protection in neuroinflammatory disease

Brain (London, England : 1878), 2005-01, Vol.128 (1), p.5-6

WAXMAN, Stephen G

2005

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Low Density of Sodium Channels Supports Action Potential Conduction in Axons of Neonatal Rat Optic Nerve

Proceedings of the National Academy of Sciences - PNAS, 1989-02, Vol.86 (4), p.1406-1410

1989

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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

International Spinal Cord Injury Pain Classification: part I. Background and description

Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

International Spinal Cord Injury Pain (ISCIP) Classification: Part 2. Initial validation using vignettes

Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Intravenous administration of auto serum-expanded autologous mesenchymal stem cells in stroke

A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities

Sodium Channels in Normal and Pathological Pain

Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination

Activated polymorphonuclear cells promote injury and excitability of dorsal root ganglia neurons

Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5

Molecular and Functional Remodeling of Electrogenic Membrane of Hypothalamic Neurons in Response to Changes in Their Input

The pentapeptide QYNAD does not block voltage-gated sodium channels

Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar purkinje cells

Physiological and genetic analysis of multiple sodium channel variants in a model of genetic absence epilepsy

Preferential expression of IGF-I in small DRG neurons and down-regulation following injury

Tapered withdrawal of phenytoin removes protective effect in EAE without inflammatory rebound and mortality

Acquired channelopathies in nerve injury and MS

Characterizing the mechanisms of progression in multiple sclerosis: evidence and new hypotheses for future directions

Dysregulation of sodium channel expression in cortical neurons in a rodent model of absence epilepsy

Na+-Ca2+ exchanger mediates Ca2+ influx during anoxia in mammalian central nervous system white matter

Demyelination in spinal cord injury

Sodium channel blockers and axonal protection in neuroinflammatory disease

Low Density of Sodium Channels Supports Action Potential Conduction in Axons of Neonatal Rat Optic Nerve

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