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Nicotinamide mononucleotide alters mitochondrial dynamics by SIRT3‐dependent mechanism in male mice

Journal of neuroscience research, 2019-08, Vol.97 (8), p.975-990

2019

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Multi-targeted Effect of Nicotinamide Mononucleotide on Brain Bioenergetic Metabolism

Neurochemical research, 2019-10, Vol.44 (10), p.2280-2287

2019

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Nicotinamide Mononucleotide Administration Prevents Experimental Diabetes-Induced Cognitive Impairment and Loss of Hippocampal Neurons

International journal of molecular sciences, 2020-05, Vol.21 (11), p.3756

2020

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Cellular and Mitochondrial NAD Homeostasis in Health and Disease

Cells (Basel, Switzerland), 2023-05, Vol.12 (9), p.1329

2023

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Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer's disease-relevant murine model

BMC neurology, 2015-03, Vol.15 (1), p.19-19, Article 19

2015

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Acetylation in Mitochondria Dynamics and Neurodegeneration

Cells (Basel, Switzerland), 2021-11, Vol.10 (11), p.3031

2021

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Mitochondrial dynamics: cell-type and hippocampal region specific changes following global cerebral ischemia

Journal of bioenergetics and biomembranes, 2015-04, Vol.47 (1-2), p.13-31

2015

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NAD + Precursors Reverse Experimental Diabetic Neuropathy in Mice

International journal of molecular sciences, 2024-01, Vol.25 (2), p.1102

2024

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Role of NAD + -Modulated Mitochondrial Free Radical Generation in Mechanisms of Acute Brain Injury

Brain sciences, 2020-07, Vol.10 (7), p.449

2020

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Perturbed Brain Glucose Metabolism Caused by Absent SIRT3 Activity

Cells (Basel, Switzerland), 2021-09, Vol.10 (9), p.2348

2021

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NAD+ precursor modulates post-ischemic mitochondrial fragmentation and reactive oxygen species generation via SIRT3 dependent mechanisms

Experimental neurology, 2020-03, Vol.325, p.113144-113144, Article 113144

2020

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Genetic inhibition of PKCε attenuates neurodegeneration after global cerebral ischemia in male mice

Journal of neuroscience research, 2019-04, Vol.97 (4), p.444-455

2019

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Calcium‐induced precipitate formation in brain mitochondria: composition, calcium capacity, and retention

Journal of neurochemistry, 2007-08, Vol.102 (4), p.1346-1356

2007

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Calcium in ischemic cell death

Stroke (1970), 1998-03, Vol.29 (3), p.705-718

1998

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Neuron-Specific Conditional Expression of a Mitochondrially Targeted Fluorescent Protein in Mice

The Journal of neuroscience, 2006-12, Vol.26 (51), p.13123-13127

2006

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Isolation of mitochondria from the CNS

Current Protocols in Neuroscience, 2010-07, Vol.Chapter 7 (1), p.Unit 7.22

Kristian, Tibor

2010

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Interplay between NAD+ and acetyl‑CoA metabolism in ischemia-induced mitochondrial pathophysiology

Biochimica et biophysica acta. Molecular basis of disease, 2019-08, Vol.1865 (8), p.2060-2067

2019

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Significance of Mitochondrial Protein Post-translational Modifications in Pathophysiology of Brain Injury

Translational stroke research, 2018-06, Vol.9 (3), p.223-237

2018

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Overexpression of Sirtuin 1 protein in neurons prevents and reverses experimental diabetic neuropathy

Brain (London, England : 1878), 2019-12, Vol.142 (12), p.3737-3752

2019

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Cyclophilin D is expressed predominantly in mitochondria of γ-aminobutyric acidergic interneurons

Journal of neuroscience research, 2009-04, Vol.87 (5), p.1250-1259

2009

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Mitochondrial dysfunction and nicotinamide dinucleotide catabolism as mechanisms of cell death and promising targets for neuroprotection

Journal of neuroscience research, 2011-12, Vol.89 (12), p.1946-1955

2011

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Differential Neuroprotection by Cyclosporin A and FK506 Following Ischemia Corresponds with Differing Abilities to Inhibit Calcineurin and the Mitochondrial Permeability Transition

Neurobiology of disease, 2002-08, Vol.10 (3), p.219-233

2002

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Mitochondrial Dysfunction and NAD+ Metabolism Alterations in the Pathophysiology of Acute Brain Injury

Translational stroke research, 2013-12, Vol.4 (6), p.618-634

2013

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Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage

Cell calcium (Edinburgh), 2004-09, Vol.36 (3), p.221-233

Kristian, T

2004

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Methoxychlor Inhibits Brain Mitochondrial Respiration and Increases Hydrogen Peroxide Production and CREB Phosphorylation

Toxicological sciences, 2005-12, Vol.88 (2), p.495-504

2005

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Nicotinamide mononucleotide alters mitochondrial dynamics by SIRT3‐dependent mechanism in male mice

Multi-targeted Effect of Nicotinamide Mononucleotide on Brain Bioenergetic Metabolism

Nicotinamide Mononucleotide Administration Prevents Experimental Diabetes-Induced Cognitive Impairment and Loss of Hippocampal Neurons

Cellular and Mitochondrial NAD Homeostasis in Health and Disease

Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer's disease-relevant murine model

Acetylation in Mitochondria Dynamics and Neurodegeneration

Mitochondrial dynamics: cell-type and hippocampal region specific changes following global cerebral ischemia

NAD + Precursors Reverse Experimental Diabetic Neuropathy in Mice

Role of NAD + -Modulated Mitochondrial Free Radical Generation in Mechanisms of Acute Brain Injury

Perturbed Brain Glucose Metabolism Caused by Absent SIRT3 Activity

NAD+ precursor modulates post-ischemic mitochondrial fragmentation and reactive oxygen species generation via SIRT3 dependent mechanisms

Genetic inhibition of PKCε attenuates neurodegeneration after global cerebral ischemia in male mice

Calcium‐induced precipitate formation in brain mitochondria: composition, calcium capacity, and retention

Calcium in ischemic cell death

Neuron-Specific Conditional Expression of a Mitochondrially Targeted Fluorescent Protein in Mice

Isolation of mitochondria from the CNS

Interplay between NAD+ and acetyl‑CoA metabolism in ischemia-induced mitochondrial pathophysiology

Significance of Mitochondrial Protein Post-translational Modifications in Pathophysiology of Brain Injury

Overexpression of Sirtuin 1 protein in neurons prevents and reverses experimental diabetic neuropathy

Cyclophilin D is expressed predominantly in mitochondria of γ-aminobutyric acidergic interneurons

Mitochondrial dysfunction and nicotinamide dinucleotide catabolism as mechanisms of cell death and promising targets for neuroprotection

Differential Neuroprotection by Cyclosporin A and FK506 Following Ischemia Corresponds with Differing Abilities to Inhibit Calcineurin and the Mitochondrial Permeability Transition

Mitochondrial Dysfunction and NAD+ Metabolism Alterations in the Pathophysiology of Acute Brain Injury

Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage

Methoxychlor Inhibits Brain Mitochondrial Respiration and Increases Hydrogen Peroxide Production and CREB Phosphorylation

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