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High post‐treatment platelet reactivity identified low‐responders to dual antiplatelet therapy at increased risk of recurrent cardiovascular events after stenting for acute coronary syndrome

Journal of thrombosis and haemostasis, 2006-03, Vol.4 (3), p.542-549

2006

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The CYP2C1917 allele is associated with better platelet response to clopidogrel in patients admitted for non‐ST acute coronary syndrome

Journal of thrombosis and haemostasis, 2009-08, Vol.7 (8), p.1409-1411

2009

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Endothelial cell markers and the risk of coronary heart disease: The prospective epidemiological study of myocardial infarction (PRIME) study

Circulation (New York, N.Y.), 2004-03, Vol.109 (11), p.1343-1348

2004

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Production of plasminogen activator inhibitor 1 by human adipose tissue: possible link between visceral fat accumulation and vascular disease

Diabetes (New York, N.Y.), 1997-05, Vol.46 (5), p.860-867

1997

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Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Arteriosclerosis, thrombosis, and vascular biology, 2007-10, Vol.27 (10), p.2250-2257

2007

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Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Arteriosclerosis, thrombosis, and vascular biology, 2002-05, Vol.22 (5), p.867-873

2002

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Effect of sleep apnea syndrome on the circadian profile of cortisol in obese men

American journal of physiology: endocrinology and metabolism, 2007-08, Vol.293 (2), p.E466-E474

2007

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Clearance of tissue plasminogen activator (TPA) and TPA/plasminogen activator inhibitor type 1 (PAI-1) complex relationship to elevated TPA antigen in patients with high PAI-1 activity levels

Circulation (New York, N.Y.), 1997-08, Vol.96 (3), p.761-768

1997

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Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

Arteriosclerosis, thrombosis, and vascular biology, 2000-09, Vol.20 (9), p.2156-2161

2000

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Plasma TAFI antigen variations in healthy subjects

Thrombosis and haemostasis, 2000-06, Vol.83 (6), p.902-905

2000

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Presence of Autoantibodies to Interleukin-8 or Neutrophil-Activating Peptide-2 in Patients With Heparin-Associated Thrombocytopenia

Blood, 1996-07, Vol.88 (2), p.410-416

1996

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PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level

Arteriosclerosis, thrombosis, and vascular biology, 1999-05, Vol.19 (5), p.1361-1365

1999

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Metabolic determinants are much more important than genetic polymorphisms in determining the PAI-1 activity and antigen plasma concentrations: a family study with part of the Stanislas Cohort

Arteriosclerosis, thrombosis, and vascular biology, 1998-01, Vol.18 (1), p.84-91

1998

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Visceral fat as a main determinant of plasminogen activator inhibitor 1 level in women

International Journal of Obesity, 1998-04, Vol.22 (4), p.312-317

1998

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Expression of adrenomedullin in adipose tissue of lean and obese women

European journal of endocrinology, 2006-07, Vol.155 (1), p.177-185

2006

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Glucocorticoids and insulin promote plasminogen activator inhibitor 1 production by human adipose tissue

Diabetes (New York, N.Y.), 1999-04, Vol.48 (4), p.890-895

1999

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Safety and effectiveness of the association ezetimibe-statin (E-S) versus high dose rosuvastatin after acute coronary syndrome: The SAFE-ES study

Annales de cardiologie et d'angeiologie, 2014-09, Vol.63 (4), p.222-227

2014

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Interaction between the C-260T polymorphism of the CD14 gene and the plasma IL-6 concentration on the risk of myocardial infarction: the HIFMECH study

Atherosclerosis, 2005-04, Vol.179 (2), p.317-323

2005

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The A -844G polymorphism in the PAI-1 gene is associated with a higher risk of venous thrombosis in factor V Leiden carriers

Arteriosclerosis, thrombosis, and vascular biology, 2000-05, Vol.20 (5), p.1387-1391

2000

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Weak and non‐independent association between plasma TAFI antigen levels and the insulin resistance syndrome

Journal of thrombosis and haemostasis, 2003-04, Vol.1 (4), p.791-797

2003

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The plasminogen activator inhibitor‐1 −675 4G/5G genotype influences the risk of myocardial infarction associated with elevated plasma proinsulin and insulin concentrations in men from Europe: the HIFMECH Study

Journal of thrombosis and haemostasis, 2003-11, Vol.1 (11), p.2322-2329

2003

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Plasma PAI-1 levels are more strongly related to liver steatosis than to adipose tissue accumulation

Arteriosclerosis, thrombosis, and vascular biology, 2003-07, Vol.23 (7), p.1262-1268

2003

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Antibodies to phosphatidylethanolamine as the only antiphospholipid antibodies found in patients with unexplained thromboses

Thrombosis and haemostasis, 2001-05, Vol.85 (5), p.800-805

2001

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Activated thrombin activatable fibrinolysis inhibitor levels are associated with the risk of cardiovascular death in patients with coronary artery disease: the AtheroGene study

Journal of thrombosis and haemostasis, 2009-01, Vol.7 (1), p.49-57

2009

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Fibrinolytic and inflammatory processes in pleural effusions

The European respiratory journal, 1995-08, Vol.8 (8), p.1352-1356

1995

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High post‐treatment platelet reactivity identified low‐responders to dual antiplatelet therapy at increased risk of recurrent cardiovascular events after stenting for acute coronary syndrome

The CYP2C1917 allele is associated with better platelet response to clopidogrel in patients admitted for non‐ST acute coronary syndrome

Endothelial cell markers and the risk of coronary heart disease: The prospective epidemiological study of myocardial infarction (PRIME) study

Production of plasminogen activator inhibitor 1 by human adipose tissue: possible link between visceral fat accumulation and vascular disease

Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Effect of sleep apnea syndrome on the circadian profile of cortisol in obese men

Clearance of tissue plasminogen activator (TPA) and TPA/plasminogen activator inhibitor type 1 (PAI-1) complex relationship to elevated TPA antigen in patients with high PAI-1 activity levels

Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

Plasma TAFI antigen variations in healthy subjects

Presence of Autoantibodies to Interleukin-8 or Neutrophil-Activating Peptide-2 in Patients With Heparin-Associated Thrombocytopenia

PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level

Metabolic determinants are much more important than genetic polymorphisms in determining the PAI-1 activity and antigen plasma concentrations: a family study with part of the Stanislas Cohort

Visceral fat as a main determinant of plasminogen activator inhibitor 1 level in women

Expression of adrenomedullin in adipose tissue of lean and obese women

Glucocorticoids and insulin promote plasminogen activator inhibitor 1 production by human adipose tissue

Safety and effectiveness of the association ezetimibe-statin (E-S) versus high dose rosuvastatin after acute coronary syndrome: The SAFE-ES study

Interaction between the C-260T polymorphism of the CD14 gene and the plasma IL-6 concentration on the risk of myocardial infarction: the HIFMECH study

The A -844G polymorphism in the PAI-1 gene is associated with a higher risk of venous thrombosis in factor V Leiden carriers

Weak and non‐independent association between plasma TAFI antigen levels and the insulin resistance syndrome

The plasminogen activator inhibitor‐1 −675 4G/5G genotype influences the risk of myocardial infarction associated with elevated plasma proinsulin and insulin concentrations in men from Europe: the HIFMECH Study

Plasma PAI-1 levels are more strongly related to liver steatosis than to adipose tissue accumulation

Antibodies to phosphatidylethanolamine as the only antiphospholipid antibodies found in patients with unexplained thromboses

Activated thrombin activatable fibrinolysis inhibitor levels are associated with the risk of cardiovascular death in patients with coronary artery disease: the AtheroGene study

Fibrinolytic and inflammatory processes in pleural effusions

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