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Transcript profiling of cytokinin action in Arabidopsis roots and shoots discovers largely similar but also organ-specific responses

BMC plant biology, 2012-07, Vol.12 (1), p.112-112, Article 112

2012

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High Rates of Forward Transmission Events after Acute/Early HIV-1 Infection

The Journal of infectious diseases, 2007-04, Vol.195 (7), p.951-959

2007

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Development of a G118R mutation in HIV-1 integrase following a switch to dolutegravir monotherapy leading to cross-resistance to integrase inhibitors

Journal of antimicrobial chemotherapy, 2016-07, Vol.71 (7), p.1948-1953

2016

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Compensation by the E138K Mutation in HIV-1 Reverse Transcriptase for Deficits in Viral Replication Capacity and Enzyme Processivity Associated with the M184I/V Mutations

Journal of Virology, 2011-11, Vol.85 (21), p.11300-11308

2011

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Signature Nucleotide Polymorphisms at Positions 64 and 65 in Reverse Transcriptase Favor the Selection of the K65R Resistance Mutation in HIV-1 Subtype C

The Journal of infectious diseases, 2009-10, Vol.200 (8), p.1202-1206

2009

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Transmission dynamics of the M184V drug resistance mutation in primary HIV infection

Journal of antimicrobial chemotherapy, 2011-10, Vol.66 (10), p.2346-2349

2011

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Impact of clade diversity on HIV-1 virulence, antiretroviral drug sensitivity and drug resistance

Journal of antimicrobial chemotherapy, 2003-02, Vol.51 (2), p.229-240

2003

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Template Usage Is Responsible for the Preferential Acquisition of the K65R Reverse Transcriptase Mutation in Subtype C Variants of Human Immunodeficiency Virus Type 1

Journal of Virology, 2009-02, Vol.83 (4), p.2029-2033

2009

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Characterization of the E138K Resistance Mutation in HIV-1 Reverse Transcriptase Conferring Susceptibility to Etravirine in B and Non-B HIV-1 Subtypes

Antimicrobial Agents and Chemotherapy, 2011-02, Vol.55 (2), p.600-607

2011

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Basis for Early and Preferential Selection of the E138K Mutation in HIV-1 Reverse Transcriptase

Antimicrobial Agents and Chemotherapy, 2013-10, Vol.57 (10), p.4681-4688

2013

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Multidrug-Resistant Variants of HIV Type 1 (HIV-1) Can Exist in Cells as Defective Quasispecies and Be Rescued by Superinfection with Other Defective HIV-1 Variants

The Journal of infectious diseases, 2009-11, Vol.200 (9), p.1479-1483

2009

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Doravirine responses to HIV-1 viruses bearing mutations to NRTIs and NNRTIs under in vitro selective drug pressure

Journal of antimicrobial chemotherapy, 2023-08, Vol.78 (8), p.1921-1928

2023

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Variations in Reverse Transcriptase and RNase H Domain Mutations in Human Immunodeficiency Virus Type 1 Clinical Isolates Are Associated with Divergent Phenotypic Resistance to Zidovudine

Antimicrobial Agents and Chemotherapy, 2007-11, Vol.51 (11), p.3861-3869

2007

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Natural Polymorphisms in the Human Immunodeficiency Virus Type 2 Protease Can Accelerate Time to Development of Resistance to Protease Inhibitors

Antimicrobial Agents and Chemotherapy, 2007-02, Vol.51 (2), p.604-610

2007

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Subunit-Selective Mutational Analysis and Tissue Culture Evaluations of the Interactions of the E138K and M184I Mutations in HIV-1 Reverse Transcriptase

Journal of Virology, 2012-08, Vol.86 (16), p.8422-8431

2012

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Cell culture selections reveal favourable drug resistance profiles for doravirine and islatravir

Journal of antimicrobial chemotherapy, 2021-08, Vol.76 (8), p.2137-2142

2021

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Detection of Human Immunodeficiency Virus (HIV) Type 1 M184V and K103N Minority Variants in Patients with Primary HIV Infection

Antimicrobial Agents and Chemotherapy, 2009-04, Vol.53 (4), p.1670-1672

2009

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The role of polymorphisms at position 89 in the HIV-1 protease gene in the development of drug resistance to HIV-1 protease inhibitors

Journal of antimicrobial chemotherapy, 2012-04, Vol.67 (4), p.988-994

2012

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The preferential selection of K65R in HIV-1 subtype C is attenuated by nucleotide polymorphisms at thymidine analogue mutation sites

Journal of antimicrobial chemotherapy, 2013-10, Vol.68 (10), p.2192-2196

2013

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Long Dissociation of Bictegravir from HIV-1 Integrase-DNA Complexes

Antimicrobial agents and chemotherapy, 2021-04, Vol.65 (5)

2021

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M184I/V substitutions and E138K/M184I/V double substitutions in HIV reverse transcriptase do not significantly affect the antiviral activity of EFdA

Journal of antimicrobial chemotherapy, 2017-11, Vol.72 (11), p.3008-3011

2017

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Nucleoside and Nucleotide Analogs Select in Culture for Different Patterns of Drug Resistance in Human Immunodeficiency Virus Types 1 and 2

Antimicrobial Agents and Chemotherapy, 2009-02, Vol.53 (2), p.708-715

2009

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Antiviral Activity of Bictegravir and Cabotegravir against Integrase Inhibitor-Resistant SIVmac239 and HIV-1

Antimicrobial agents and chemotherapy, 2017-12, Vol.61 (12)

2017

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Human Immunodeficiency Virus Type 1 Recombinant Reverse Transcriptase Enzymes Containing the G190A and Y181C Resistance Mutations Remain Sensitive to Etravirine

Antimicrobial Agents and Chemotherapy, 2009-11, Vol.53 (11), p.4667-4672

2009

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In vitro and structural evaluation of PL-100 as a potential second-generation HIV-1 protease inhibitor

Journal of antimicrobial chemotherapy, 2013-01, Vol.68 (1), p.105-112

2013

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Transcript profiling of cytokinin action in Arabidopsis roots and shoots discovers largely similar but also organ-specific responses

High Rates of Forward Transmission Events after Acute/Early HIV-1 Infection

Development of a G118R mutation in HIV-1 integrase following a switch to dolutegravir monotherapy leading to cross-resistance to integrase inhibitors

Compensation by the E138K Mutation in HIV-1 Reverse Transcriptase for Deficits in Viral Replication Capacity and Enzyme Processivity Associated with the M184I/V Mutations

Signature Nucleotide Polymorphisms at Positions 64 and 65 in Reverse Transcriptase Favor the Selection of the K65R Resistance Mutation in HIV-1 Subtype C

Transmission dynamics of the M184V drug resistance mutation in primary HIV infection

Impact of clade diversity on HIV-1 virulence, antiretroviral drug sensitivity and drug resistance

Template Usage Is Responsible for the Preferential Acquisition of the K65R Reverse Transcriptase Mutation in Subtype C Variants of Human Immunodeficiency Virus Type 1

Characterization of the E138K Resistance Mutation in HIV-1 Reverse Transcriptase Conferring Susceptibility to Etravirine in B and Non-B HIV-1 Subtypes

Basis for Early and Preferential Selection of the E138K Mutation in HIV-1 Reverse Transcriptase

Multidrug-Resistant Variants of HIV Type 1 (HIV-1) Can Exist in Cells as Defective Quasispecies and Be Rescued by Superinfection with Other Defective HIV-1 Variants

Doravirine responses to HIV-1 viruses bearing mutations to NRTIs and NNRTIs under in vitro selective drug pressure

Variations in Reverse Transcriptase and RNase H Domain Mutations in Human Immunodeficiency Virus Type 1 Clinical Isolates Are Associated with Divergent Phenotypic Resistance to Zidovudine

Natural Polymorphisms in the Human Immunodeficiency Virus Type 2 Protease Can Accelerate Time to Development of Resistance to Protease Inhibitors

Subunit-Selective Mutational Analysis and Tissue Culture Evaluations of the Interactions of the E138K and M184I Mutations in HIV-1 Reverse Transcriptase

Cell culture selections reveal favourable drug resistance profiles for doravirine and islatravir

Detection of Human Immunodeficiency Virus (HIV) Type 1 M184V and K103N Minority Variants in Patients with Primary HIV Infection

The role of polymorphisms at position 89 in the HIV-1 protease gene in the development of drug resistance to HIV-1 protease inhibitors

The preferential selection of K65R in HIV-1 subtype C is attenuated by nucleotide polymorphisms at thymidine analogue mutation sites

Long Dissociation of Bictegravir from HIV-1 Integrase-DNA Complexes

M184I/V substitutions and E138K/M184I/V double substitutions in HIV reverse transcriptase do not significantly affect the antiviral activity of EFdA

Nucleoside and Nucleotide Analogs Select in Culture for Different Patterns of Drug Resistance in Human Immunodeficiency Virus Types 1 and 2

Antiviral Activity of Bictegravir and Cabotegravir against Integrase Inhibitor-Resistant SIVmac239 and HIV-1

Human Immunodeficiency Virus Type 1 Recombinant Reverse Transcriptase Enzymes Containing the G190A and Y181C Resistance Mutations Remain Sensitive to Etravirine

In vitro and structural evaluation of PL-100 as a potential second-generation HIV-1 protease inhibitor

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