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Combinatorial Atoh1 and Gfi1 induction enhances hair cell regeneration in the adult cochlea

Scientific reports, 2020-12, Vol.10 (1), p.21397-15, Article 21397

2020

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Inappropriate p53 activation during development induces features of CHARGE syndrome

Nature (London), 2014-10, Vol.514 (7521), p.228-232

2014

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Scar Formation and Debris Elimination during Hair Cell Degeneration in the Adult DTR Mouse

Neuroscience, 2021-01, Vol.453, p.57-68

2021

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CHD7 and retinoic acid signaling cooperate to regulate neural stem cell and inner ear development in mouse models of CHARGE syndrome

Human molecular genetics, 2014-01, Vol.23 (2), p.434-448

2014

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Development of a chronically-implanted mouse model for studies of cochlear health and implant function

Hearing research, 2021-05, Vol.404, p.108216-108216, Article 108216

2021

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The fate of outer hair cells after acoustic or ototoxic insults

Hearing research, 2006-08, Vol.218 (1), p.20-29

2006

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The complex ontogenetic trajectory of mandibular shape in a laboratory mouse

Journal of anatomy, 2013-12, Vol.223 (6), p.568-580

2013

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Connexin 26 null mice exhibit spiral ganglion degeneration that can be blocked by BDNF gene therapy

Hearing research, 2014-03, Vol.309, p.124-135

2014

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Severe streptomycin ototoxicity in the mouse utricle leads to a flat epithelium but the peripheral neural degeneration is delayed

Hearing research, 2017-11, Vol.355, p.33-41

2017

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Mice with conditional deletion of Cx26 exhibit no vestibular phenotype despite secondary loss of Cx30 in the vestibular end organs

Hearing research, 2015-10, Vol.328, p.102-112

2015

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Defects in vestibular sensory epithelia and innervation in mice with loss of Chd7 function: Implications for human CHARGE syndrome

Journal of comparative neurology (1911), 2007-10, Vol.504 (5), p.519-532

2007

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Mature middle and inner ears express Chd7 and exhibit distinctive pathologies in a mouse model of CHARGE syndrome

Hearing research, 2011-12, Vol.282 (1), p.184-195

2011

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p27(Kip1) deficiency causes organ of Corti pathology and hearing loss

Hearing research, 2006-04, Vol.214 (1-2), p.28-36

2006

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Complexity and integration in the control of inner-ear development

Genome biology, 2007-01, Vol.8 (9), p.315-315

2007

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Whirler mutant hair cells have less severe pathology than shaker 2 or double mutants

Journal of the Association for Research in Otolaryngology, 2007-09, Vol.8 (3), p.329-337

2007

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Combinatorial Atoh1 and Gfi1 induction enhances hair cell regeneration in the adult cochlea

Inappropriate p53 activation during development induces features of CHARGE syndrome

Scar Formation and Debris Elimination during Hair Cell Degeneration in the Adult DTR Mouse

CHD7 and retinoic acid signaling cooperate to regulate neural stem cell and inner ear development in mouse models of CHARGE syndrome

Development of a chronically-implanted mouse model for studies of cochlear health and implant function

The fate of outer hair cells after acoustic or ototoxic insults

The complex ontogenetic trajectory of mandibular shape in a laboratory mouse

Connexin 26 null mice exhibit spiral ganglion degeneration that can be blocked by BDNF gene therapy

Severe streptomycin ototoxicity in the mouse utricle leads to a flat epithelium but the peripheral neural degeneration is delayed

Mice with conditional deletion of Cx26 exhibit no vestibular phenotype despite secondary loss of Cx30 in the vestibular end organs

Defects in vestibular sensory epithelia and innervation in mice with loss of Chd7 function: Implications for human CHARGE syndrome

Mature middle and inner ears express Chd7 and exhibit distinctive pathologies in a mouse model of CHARGE syndrome

p27(Kip1) deficiency causes organ of Corti pathology and hearing loss

Complexity and integration in the control of inner-ear development

Whirler mutant hair cells have less severe pathology than shaker 2 or double mutants

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