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Cbl: many adaptations to regulate protein tyrosine kinases

Nature reviews. Molecular cell biology, 2001-04, Vol.2 (4), p.294-307

2001

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Mutation of the c-Cbl TKB Domain Binding Site on the Met Receptor Tyrosine Kinase Converts It into a Transforming Protein

Molecular cell, 2001-11, Vol.8 (5), p.995-1004

2001

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Tissue Hyperplasia and Enhanced T-Cell Signalling via ZAP-70 in c-Cbl-Deficient Mice

Molecular and Cellular Biology, 1998-08, Vol.18 (8), p.4872-4882

1998

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v-cbl, an Oncogene from a Dual-Recombinant Murine Retrovirus that Induces Early B-Lineage Lymphomas

Proceedings of the National Academy of Sciences - PNAS, 1989-02, Vol.86 (4), p.1168-1172

1989

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Phosphotyrosine binding domain-dependent upregulation of the platelet-derived growth factor receptor alpha signaling cascade by transforming mutants of Cbl: implications for Cbl's function and oncogenicity

Molecular and Cellular Biology, 1997-08, Vol.17 (8), p.4597-4610

1997

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EGF receptor binding and transformation by v-cbl is ablated by the introduction of a loss-of-function mutation from the Caenorhabditis elegans sli-1 gene

Oncogene, 1997-05, Vol.14 (18), p.2239-2249

1997

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The truncation that generated the v‐cbl oncogene reveals an ability for nuclear transport, DNA binding and acute transformation

The EMBO journal, 1993-05, Vol.12 (5), p.2017-2026

1993

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Tyrosine kinase activity of the EGF receptor is enhanced by the expression of oncogenic 70Z-Cbl

Oncogene, 1997-12, Vol.15 (24), p.2909-2919

1997

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Tumour induction by activated abl involves tyrosine phosphorylation of the product of the cbl oncogene

The EMBO journal, 1994-10, Vol.13 (19), p.4515-4523

1994

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c-Cbl localizes to actin lamellae and regulates lamellipodia formation and cell morphology

Journal of cell science, 2000-01, Vol.113 Pt 2 (2), p.215-226

2000

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Serine mutations that abrogate ligand-induced ubiquitination and internalization of the EGF receptor do not affect c-Cbl association with the receptor

Oncogene, 2003-11, Vol.22 (52), p.8509-8518

2003

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The Cbl Proto-Oncogene Product Negatively Regulates the Src-Family Tyrosine Kinase Fyn by Enhancing Its Degradation

Molecular and Cellular Biology, 2000-02, Vol.20 (3), p.851-867

2000

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A mouse with a loss-of-function mutation in the c-Cbl TKB domain shows perturbed thymocyte signaling without enhancing the activity of the ZAP-70 tyrosine kinase

The Journal of experimental medicine, 2003-02, Vol.197 (4), p.503-513

2003

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Perturbed Regulation of ZAP-70 and Sustained Tyrosine Phosphorylation of LAT and SLP-76 in c-Cbl-Deficient Thymocytes

The Journal of immunology (1950), 1999-06, Vol.162 (12), p.7133-7139

1999

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The two major sites of cbl tyrosine phosphorylation in abl-transformed cells select the crkL SH2 domain

Oncogene, 1996-05, Vol.12 (9), p.1981-1989

1996

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c-Cbl Is Transiently Tyrosine-phosphorylated, Ubiquitinated, and Membrane-targeted following CSF-1 Stimulation of Macrophages (∗)

The Journal of biological chemistry, 1996-01, Vol.271 (1), p.17-20

1996

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The E mu-myc transgenic mouse. A model for high-incidence spontaneous lymphoma and leukemia of early B cells

The Journal of experimental medicine, 1988-02, Vol.167 (2), p.353-371

1988

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Oncogenic forms of Cbl abrogate the anchorage requirement but not the growth factor requirement for proliferation

Oncogene, 1998-06, Vol.16 (24), p.3159-3167

1998

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The protein product of the c-cbl oncogene rapidly complexes with the EGF receptor and is tyrosine phosphorylated following EGF stimulation

Oncogene, 1995-10, Vol.11 (8), p.1561-1567

1995

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The c- myc oncogene perturbs B lymphocyte development in Eμ- myc transgenic mice

Cell, 1986-10, Vol.47 (1), p.11-18

1986

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RING Finger Mutations that Abolish c-Cbl-Directed Polyubiquitination and Downregulation of the EGF Receptor Are Insufficient for Cell Transformation

Molecular cell, 2001-02, Vol.7 (2), p.355-365

2001

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The c-cbl proto-oncogene is preferentially expressed in thymus and testis tissue and encodes a nuclear protein

Journal of Virology, 1989-12, Vol.63 (12), p.5420-5424

1989

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c-Cbl: A regulator of T cell receptor-mediated signalling

Immunology and cell biology, 1998-10, Vol.76 (5), p.473-482

1998

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Ras-dependent, Ca2+-stimulated Activation of Nuclear Factor of Activated T Cells by a Constitutively Active Cbl Mutant in T Cells

The Journal of biological chemistry, 1997-01, Vol.272 (1), p.168-173

1997

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Essential role of E3 ubiquitin ligase activity in Cbl-b-regulated T cell functions

The Journal of immunology (1950), 2011-02, Vol.186 (4), p.2138-2147

2011

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Cbl: many adaptations to regulate protein tyrosine kinases

Mutation of the c-Cbl TKB Domain Binding Site on the Met Receptor Tyrosine Kinase Converts It into a Transforming Protein

Tissue Hyperplasia and Enhanced T-Cell Signalling via ZAP-70 in c-Cbl-Deficient Mice

v-cbl, an Oncogene from a Dual-Recombinant Murine Retrovirus that Induces Early B-Lineage Lymphomas

Phosphotyrosine binding domain-dependent upregulation of the platelet-derived growth factor receptor alpha signaling cascade by transforming mutants of Cbl: implications for Cbl's function and oncogenicity

EGF receptor binding and transformation by v-cbl is ablated by the introduction of a loss-of-function mutation from the Caenorhabditis elegans sli-1 gene

The truncation that generated the v‐cbl oncogene reveals an ability for nuclear transport, DNA binding and acute transformation

Tyrosine kinase activity of the EGF receptor is enhanced by the expression of oncogenic 70Z-Cbl

Tumour induction by activated abl involves tyrosine phosphorylation of the product of the cbl oncogene

c-Cbl localizes to actin lamellae and regulates lamellipodia formation and cell morphology

Serine mutations that abrogate ligand-induced ubiquitination and internalization of the EGF receptor do not affect c-Cbl association with the receptor

The Cbl Proto-Oncogene Product Negatively Regulates the Src-Family Tyrosine Kinase Fyn by Enhancing Its Degradation

A mouse with a loss-of-function mutation in the c-Cbl TKB domain shows perturbed thymocyte signaling without enhancing the activity of the ZAP-70 tyrosine kinase

Perturbed Regulation of ZAP-70 and Sustained Tyrosine Phosphorylation of LAT and SLP-76 in c-Cbl-Deficient Thymocytes

The two major sites of cbl tyrosine phosphorylation in abl-transformed cells select the crkL SH2 domain

c-Cbl Is Transiently Tyrosine-phosphorylated, Ubiquitinated, and Membrane-targeted following CSF-1 Stimulation of Macrophages (∗)

The E mu-myc transgenic mouse. A model for high-incidence spontaneous lymphoma and leukemia of early B cells

Oncogenic forms of Cbl abrogate the anchorage requirement but not the growth factor requirement for proliferation

The protein product of the c-cbl oncogene rapidly complexes with the EGF receptor and is tyrosine phosphorylated following EGF stimulation

The c- myc oncogene perturbs B lymphocyte development in Eμ- myc transgenic mice

RING Finger Mutations that Abolish c-Cbl-Directed Polyubiquitination and Downregulation of the EGF Receptor Are Insufficient for Cell Transformation

The c-cbl proto-oncogene is preferentially expressed in thymus and testis tissue and encodes a nuclear protein

c-Cbl: A regulator of T cell receptor-mediated signalling

Ras-dependent, Ca2+-stimulated Activation of Nuclear Factor of Activated T Cells by a Constitutively Active Cbl Mutant in T Cells

Essential role of E3 ubiquitin ligase activity in Cbl-b-regulated T cell functions

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