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Mitochondrial respiration without ubiquinone biosynthesis

Human molecular genetics, 2013-12, Vol.22 (23), p.4768-4783

2013

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Evolutionary conservation of the clk-1-dependent mechanism of longevity: loss of mclk1 increases cellular fitness and lifespan in mice

Genes & development, 2005-10, Vol.19 (20), p.2424-2434

2005

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Delayed aging in Ames dwarf mice. Relationships to endocrine function and body size

Results and problems in cell differentiation, 2000, Vol.29, p.181

Bartke, A

2000

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Ubiquinone Is Necessary for Mouse Embryonic Development but Is Not Essential for Mitochondrial Respiration

The Journal of biological chemistry, 2001-12, Vol.276 (49), p.46160-46164

2001

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Structural and Functional Conservation of the Caenorhabditis elegans Timing Gene clk-1

Science (American Association for the Advancement of Science), 1997-02, Vol.275 (5302), p.980-983

1997

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Mouse CLK-1 Is Imported into Mitochondria by an Unusual Process That Requires a Leader Sequence but No Membrane Potential

The Journal of biological chemistry, 2001-08, Vol.276 (31), p.29218-29225

2001

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Stem cells and genetics in the study of development, aging, and longevity

Results and problems in cell differentiation, 2000, Vol.29, p.203

Van Zant, G

2000

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Mutation accumulation in vivo and the importance of genome stability in aging and cancer

Results and problems in cell differentiation, 2000, Vol.29, p.165

2000

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Mitochondrial dysfunction and longevity in animals: Untangling the knot

Science (American Association for the Advancement of Science), 2015-12, Vol.350 (6265), p.1204-1207

2015

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Mitochondrial function and lifespan of mice with controlled ubiquinone biosynthesis

Nature communications, 2015-03, Vol.6 (1), p.6393-6393, Article 6393

2015

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When a theory of aging ages badly

Cellular and molecular life sciences : CMLS, 2010, Vol.67 (1), p.1-8

2010

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Early Mitochondrial Dysfunction in Long-lived Mclk1+/- Mice

The Journal of biological chemistry, 2008-09, Vol.283 (38), p.26217-26227

2008

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Coenzyme Q10 restores oocyte mitochondrial function and fertility during reproductive aging

Aging cell, 2015-10, Vol.14 (5), p.887-895

2015

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Different Mechanisms of Longevity in Long-Lived Mouse and Caenorhabditis elegans Mutants Revealed by Statistical Analysis of Mortality Rates

Genetics (Austin), 2016-11, Vol.204 (3), p.905-920

2016

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The submitochondrial distribution of ubiquinone affects respiration in long-lived Mclk1+/- mice

The Journal of cell biology, 2012-10, Vol.199 (2), p.215-224

2012

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A mild impairment of mitochondrial electron transport has sex-specific effects on lifespan and aging in mice

PloS one, 2011-10, Vol.6 (10), p.e26116

2011

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Reversal of the Mitochondrial Phenotype and Slow Development of Oxidative Biomarkers of Aging in Long-lived Mclk1+/− Mice

The Journal of biological chemistry, 2009-07, Vol.284 (30), p.20364-20374

2009

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Epithelial cell death is an important contributor to oxidant-mediated acute lung injury

American journal of respiratory and critical care medicine, 2011-04, Vol.183 (8), p.1043-1054

2011

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An enhanced immune response of Mclk1⁺/⁻ mutant mice is associated with partial protection from fibrosis, cancer and the development of biomarkers of aging

PloS one, 2012-11, Vol.7 (11), p.e49606

2012

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The Anti-neurodegeneration Drug Clioquinol Inhibits the Aging-associated Protein CLK-1

The Journal of biological chemistry, 2009-01, Vol.284 (1), p.314-323

2009

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Lifelong protection from global cerebral ischemia and reperfusion in long-lived Mclk1 +/ − mutants

Experimental neurology, 2010-06, Vol.223 (2), p.557-565

2010

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Targeting protein synthesis in a Myc/mTOR-driven model of anorexia-cachexia syndrome delays its onset and prolongs survival

Cancer research (Chicago, Ill.), 2012-02, Vol.72 (3), p.747-756

2012

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Genetic and molecular characterization of CLK-1/mCLK1, a conserved determinant of the rate of aging

Experimental gerontology, 2006-10, Vol.41 (10), p.940-951

2006

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Mclk1+/- mice are not resistant to the development of atherosclerosis

Lipids in health and disease, 2009-05, Vol.8 (1), p.16-16

2009

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Elevated mitochondrial reactive oxygen species generation affects the immune response via hypoxia-inducible factor-1alpha in long-lived Mclk1+/- mouse mutants

The Journal of immunology (1950), 2010-01, Vol.184 (2), p.582-590

2010

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Mitochondrial respiration without ubiquinone biosynthesis

Evolutionary conservation of the clk-1-dependent mechanism of longevity: loss of mclk1 increases cellular fitness and lifespan in mice

Delayed aging in Ames dwarf mice. Relationships to endocrine function and body size

Ubiquinone Is Necessary for Mouse Embryonic Development but Is Not Essential for Mitochondrial Respiration

Structural and Functional Conservation of the Caenorhabditis elegans Timing Gene clk-1

Mouse CLK-1 Is Imported into Mitochondria by an Unusual Process That Requires a Leader Sequence but No Membrane Potential

Stem cells and genetics in the study of development, aging, and longevity

Mutation accumulation in vivo and the importance of genome stability in aging and cancer

Mitochondrial dysfunction and longevity in animals: Untangling the knot

Mitochondrial function and lifespan of mice with controlled ubiquinone biosynthesis

When a theory of aging ages badly

Early Mitochondrial Dysfunction in Long-lived Mclk1+/- Mice

Coenzyme Q10 restores oocyte mitochondrial function and fertility during reproductive aging

Different Mechanisms of Longevity in Long-Lived Mouse and Caenorhabditis elegans Mutants Revealed by Statistical Analysis of Mortality Rates

The submitochondrial distribution of ubiquinone affects respiration in long-lived Mclk1+/- mice

A mild impairment of mitochondrial electron transport has sex-specific effects on lifespan and aging in mice

Reversal of the Mitochondrial Phenotype and Slow Development of Oxidative Biomarkers of Aging in Long-lived Mclk1+/− Mice

Epithelial cell death is an important contributor to oxidant-mediated acute lung injury

An enhanced immune response of Mclk1⁺/⁻ mutant mice is associated with partial protection from fibrosis, cancer and the development of biomarkers of aging

The Anti-neurodegeneration Drug Clioquinol Inhibits the Aging-associated Protein CLK-1

Lifelong protection from global cerebral ischemia and reperfusion in long-lived Mclk1 +/ − mutants

Targeting protein synthesis in a Myc/mTOR-driven model of anorexia-cachexia syndrome delays its onset and prolongs survival

Genetic and molecular characterization of CLK-1/mCLK1, a conserved determinant of the rate of aging

Mclk1+/- mice are not resistant to the development of atherosclerosis

Elevated mitochondrial reactive oxygen species generation affects the immune response via hypoxia-inducible factor-1alpha in long-lived Mclk1+/- mouse mutants

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