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Novel therapies for coeliac disease

Journal of internal medicine, 2011-06, Vol.269 (6), p.604-613

2011

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Structural Basis for Gluten Intolerance in Celiac Sprue

Science (American Association for the Advancement of Science), 2002-09, Vol.297 (5590), p.2275-2279

2002

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Gluten induces an intestinal cytokine response strongly dominated by interferon gamma in patients with celiac disease

Gastroenterology (New York, N.Y. 1943), 1998-09, Vol.115 (3), p.551-563

1998

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HLA susceptibility genes in celiac disease: Genetic mapping and role in pathogenesis

Gastroenterology (New York, N.Y. 1943), 1993-09, Vol.105 (3), p.910-922

1993

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Gluten specific, HLA-DQ restricted T cells from coeliac mucosa produce cytokines with Th1 or Th0 profile dominated by interferon gamma

Gut, 1995-12, Vol.37 (6), p.766-776

1995

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Gliadin-specific HLA-DQ(α0501,β10201) restricted T cells isolated from the small intestinal mucosa of celiac disease patients

The Journal of experimental medicine, 1993-07, Vol.178 (1), p.187-196

1993

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Breast milk against coeliac disease

Gut, 2002-12, Vol.51 (6), p.767-768

Sollid, L M

2002

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Evidence for a primary association of celiac disease to a particular HLA-DQ α/β heterodimer

The Journal of experimental medicine, 1989, Vol.169 (1), p.345-350

1989

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Production of a panel of recombinant gliadins for the characterisation of T cell reactivity in coeliac disease

Gut, 2000-01, Vol.46 (1), p.46-51

2000

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Immunobiology and immunopathology of human gut mucosa: Humoral immunity and intraepithelial lymphocytes

Gastroenterology (New York, N.Y. 1943), 1989-12, Vol.97 (6), p.1562-1584

1989

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Role of Tissue Transglutaminase in Celiac Disease

Journal of pediatric gastroenterology and nutrition, 2000-03, Vol.30 (3), p.232-240

2000

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The effects of atorvastatin on gluten‐induced intestinal T cell responses in coeliac disease

Clinical and experimental immunology, 2005-11, Vol.142 (2), p.333-340

2005

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Getting to grips with gluten

Gut, 2000-12, Vol.47 (6), p.743-745

2000

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Histological changes in small bowel mucosa induced by gliadin sensitive T lymphocytes can be blocked by anti-interferon gamma antibody

Gut, 1995-06, Vol.36 (6), p.874-879

1995

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Heterogeneous reactivity patterns of HLA-DQ-restricted, small intestinal T-cell clones from patients with celiac disease

Gastroenterology (New York, N.Y. 1943), 1997-03, Vol.112 (3), p.752-759

1997

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Coeliac Disease Candidate Genes: No Association with Functional Polymorphisms in Matrix Metalloproteinase 1 and 3 Gene Promoters

Scandinavian journal of gastroenterology, 2002-08, Vol.37 (8), p.931-935

2002

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HLA Genotypes and the Increased Incidence of Coeliac Disease in Sweden

Scandinavian journal of gastroenterology, 1996, Vol.31 (11), p.1092-1097

1996

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FOXP3 polymorphisms in type 1 diabetes and coeliac disease

Journal of autoimmunity, 2006-09, Vol.27 (2), p.140-144

2006

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T Cells from the Peripheral Blood of Coeliac Disease Patients Recognize Gluten Antigens when Presented by HLA‐DR, ‐DQ, or ‐DP Molecules

Scandinavian journal of immunology, 1994-06, Vol.39 (6), p.567-574

1994

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T cells recognize a peptide derived from α-gliadin presented by the celiac disease-associated HLA-DQ (α1 ∗0501, β1 ∗0201) heterodimer

Human immunology, 1994-04, Vol.39 (4), p.243-252

1994

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Systematic review: early infant feeding and the prevention of coeliac disease

Alimentary pharmacology & therapeutics, 2012-10, Vol.36 (7), p.607-618

2012

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T lymphocyte recognition of a celiac disease-associated cis- or trans-encoded HLA-DQ alpha/beta-heterodimer

The Journal of immunology (1950), 1990-07, Vol.145 (1), p.136-139

1990

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Patients with multiple sclerosis carry DQB1 genes which encode shared polymorphic amino acid sequences

Human immunology, 1989-06, Vol.25 (2), p.103-110

1989

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Susceptibility to develop celiac disease is primarily associated with HLA-DQ alleles

Human immunology, 1990-11, Vol.29 (3), p.157-165

1990

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HLA-DR and -DQ genotypes of celiac disease patients serologically typed to be non-DR3 or non-DR5/7

Human immunology, 1992-11, Vol.35 (3), p.188-192

1992

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Novel therapies for coeliac disease

Structural Basis for Gluten Intolerance in Celiac Sprue

Gluten induces an intestinal cytokine response strongly dominated by interferon gamma in patients with celiac disease

HLA susceptibility genes in celiac disease: Genetic mapping and role in pathogenesis

Gluten specific, HLA-DQ restricted T cells from coeliac mucosa produce cytokines with Th1 or Th0 profile dominated by interferon gamma

Gliadin-specific HLA-DQ(α0501,β10201) restricted T cells isolated from the small intestinal mucosa of celiac disease patients

Breast milk against coeliac disease

Evidence for a primary association of celiac disease to a particular HLA-DQ α/β heterodimer

Production of a panel of recombinant gliadins for the characterisation of T cell reactivity in coeliac disease

Immunobiology and immunopathology of human gut mucosa: Humoral immunity and intraepithelial lymphocytes

Role of Tissue Transglutaminase in Celiac Disease

The effects of atorvastatin on gluten‐induced intestinal T cell responses in coeliac disease

Getting to grips with gluten

Histological changes in small bowel mucosa induced by gliadin sensitive T lymphocytes can be blocked by anti-interferon gamma antibody

Heterogeneous reactivity patterns of HLA-DQ-restricted, small intestinal T-cell clones from patients with celiac disease

Coeliac Disease Candidate Genes: No Association with Functional Polymorphisms in Matrix Metalloproteinase 1 and 3 Gene Promoters

HLA Genotypes and the Increased Incidence of Coeliac Disease in Sweden

FOXP3 polymorphisms in type 1 diabetes and coeliac disease

T Cells from the Peripheral Blood of Coeliac Disease Patients Recognize Gluten Antigens when Presented by HLA‐DR, ‐DQ, or ‐DP Molecules

T cells recognize a peptide derived from α-gliadin presented by the celiac disease-associated HLA-DQ (α1 ∗0501, β1 ∗0201) heterodimer

Systematic review: early infant feeding and the prevention of coeliac disease

T lymphocyte recognition of a celiac disease-associated cis- or trans-encoded HLA-DQ alpha/beta-heterodimer

Patients with multiple sclerosis carry DQB1 genes which encode shared polymorphic amino acid sequences

Susceptibility to develop celiac disease is primarily associated with HLA-DQ alleles

HLA-DR and -DQ genotypes of celiac disease patients serologically typed to be non-DR3 or non-DR5/7

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