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Peroxiredoxin1 Prevents Excessive Endothelial Activation and Early Atherosclerosis

Circulation research, 2008-09, Vol.103 (6), p.598-605

2008

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Distinct graft-versus-leukemic stem cell effects of early or delayed donor leukocyte infusions in a mouse chronic myeloid leukemia model

Blood, 2012-01, Vol.119 (1), p.273-284

2012

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Essential role for the peroxiredoxin Prdx1 in erythrocyte antioxidant defence and tumour suppression

Nature (London), 2003-07, Vol.424 (6948), p.561-565

2003

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Requirement of Src kinases Lyn, Hck and Fgr for BCR-ABL1 -induced B-lymphoblastic leukemia but not chronic myeloid leukemia

Nature genetics, 2004-05, Vol.36 (5), p.453-461

2004

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Targeting CXCR4 with cell-penetrating pepducins in lymphoma and lymphocytic leukemia

Blood, 2012-02, Vol.119 (7), p.1717-1725

2012

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The Ph-positive and Ph-negative myeloproliferative neoplasms: some topical pre-clinical and clinical issues

Haematologica (Roma), 2011-04, Vol.96 (4), p.590-601

2011

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Essential role for Stat5a/b in myeloproliferative neoplasms induced by BCR-ABL1 and JAK2V617F in mice

Blood, 2012-04, Vol.119 (15), p.3550-3560

2012

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CDK6 kinase activity is required for thymocyte development

Blood, 2011-06, Vol.117 (23), p.6120-6131

2011

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Reversibility of acute B-cell leukaemia induced by BCR - ABL1

Nature genetics, 2000-01, Vol.24 (1), p.57-60

2000

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Autologous stem cell transplant recipients tolerate haploidentical related-donor natural killer cell-enriched infusions

Transfusion (Philadelphia, Pa.), 2013-02, Vol.53 (2), p.412-418

2013

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Navigating the road toward optimal initial therapy for chronic myeloid leukemia

Current opinion in hematology, 2011-03, Vol.18 (2), p.89-97

2011

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Tyrosine kinases as targets for cancer therapy

The New England journal of medicine, 2005-07, Vol.353 (2), p.172-187

2005

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The Tel-Abl (ETV6-Abl) tyrosine kinase, product of complex (9;12) translocations in human leukemia, induces distinct myeloproliferative disease in mice

Blood, 2002-06, Vol.99 (12), p.4568-4577

2002

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Phase 2 study of subcutaneous omacetaxine mepesuccinate after TKI failure in patients with chronic-phase CML with T315I mutation

Blood, 2012-09, Vol.120 (13), p.2573-2580

2012

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The Src homology 2 domain of Bcr/Abl is required for efficient induction of chronic myeloid leukemia–like disease in mice but not for lymphoid leukemogenesis or activation of phosphatidylinositol 3-kinase

Blood, 2001-01, Vol.97 (1), p.4-13

2001

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Interleukin 3 and granulocyte-macrophage colony-stimulating factor are not required for induction of chronic myeloid leukemia-like myeloproliferative disease in mice by BCR/ABL

Blood, 2001-03, Vol.97 (5), p.1442-1450

2001

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Adoptive immunotherapy of BCR-ABL–induced chronic myeloid leukemia–like myeloproliferative disease in a murine model

Blood, 2004-12, Vol.104 (13), p.4236-4244

2004

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Pathogenesis and treatment of Ph+ leukemia: recent insights from mouse models

Current opinion in hematology, 2001-07, Vol.8 (4), p.224-230

Van Etten, R A

2001

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Modulation of hepatic acute phase gene expression by epidermal growth factor and src protein tyrosine kinases in murine and human hepatic cells

Hepatology (Baltimore, Md.), 1999-09, Vol.30 (3), p.682-697

1999

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Induction of myeloproliferative disease in mice by tyrosine kinase fusion oncogenes does not require granulocyte-macrophage colony-stimulating factor or interleukin-3

Blood, 2001-03, Vol.97 (5), p.1435-1441

2001

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The Grb2 binding site is required for the induction of chronic myeloid leukemia-like disease in mice by the Bcr/Abl tyrosine kinase

Blood, 2000-07, Vol.96 (2), p.664-670

2000

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Studying the pathogenesis of BCR-ABL+ leukemia in mice: CML: Molecular mechanisms & therapeutic strategies

Oncogene, 2002, Vol.21 (56), p.8643-8651

VAN ETTEN, Richard A

2002

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c-fos is not essential for v-abl-induced lymphomagenesis

Cancer research (Chicago, Ill.), 1995-12, Vol.55 (24), p.6196-6199

1995

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Subcellular localization of bcr, Abl, and Bcr-Abl proteins in normal and leukemic cells and correlation of expression with myeloid differentiation

The Journal of clinical investigation, 1993-10, Vol.92 (4), p.1925-1939

1993

Volltextzugriff (PDF)

v-abl Causes Hematopoietic Disease Distinct From That Caused by bcr-abl

Proceedings of the National Academy of Sciences - PNAS, 1991-08, Vol.88 (15), p.6506-6510

1991

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Peroxiredoxin1 Prevents Excessive Endothelial Activation and Early Atherosclerosis

Distinct graft-versus-leukemic stem cell effects of early or delayed donor leukocyte infusions in a mouse chronic myeloid leukemia model

Essential role for the peroxiredoxin Prdx1 in erythrocyte antioxidant defence and tumour suppression

Requirement of Src kinases Lyn, Hck and Fgr for BCR-ABL1 -induced B-lymphoblastic leukemia but not chronic myeloid leukemia

Targeting CXCR4 with cell-penetrating pepducins in lymphoma and lymphocytic leukemia

The Ph-positive and Ph-negative myeloproliferative neoplasms: some topical pre-clinical and clinical issues

Essential role for Stat5a/b in myeloproliferative neoplasms induced by BCR-ABL1 and JAK2V617F in mice

CDK6 kinase activity is required for thymocyte development

Reversibility of acute B-cell leukaemia induced by BCR - ABL1

Autologous stem cell transplant recipients tolerate haploidentical related-donor natural killer cell-enriched infusions

Navigating the road toward optimal initial therapy for chronic myeloid leukemia

Tyrosine kinases as targets for cancer therapy

The Tel-Abl (ETV6-Abl) tyrosine kinase, product of complex (9;12) translocations in human leukemia, induces distinct myeloproliferative disease in mice

Phase 2 study of subcutaneous omacetaxine mepesuccinate after TKI failure in patients with chronic-phase CML with T315I mutation

The Src homology 2 domain of Bcr/Abl is required for efficient induction of chronic myeloid leukemia–like disease in mice but not for lymphoid leukemogenesis or activation of phosphatidylinositol 3-kinase

Interleukin 3 and granulocyte-macrophage colony-stimulating factor are not required for induction of chronic myeloid leukemia-like myeloproliferative disease in mice by BCR/ABL

Adoptive immunotherapy of BCR-ABL–induced chronic myeloid leukemia–like myeloproliferative disease in a murine model

Pathogenesis and treatment of Ph+ leukemia: recent insights from mouse models

Modulation of hepatic acute phase gene expression by epidermal growth factor and src protein tyrosine kinases in murine and human hepatic cells

Induction of myeloproliferative disease in mice by tyrosine kinase fusion oncogenes does not require granulocyte-macrophage colony-stimulating factor or interleukin-3

The Grb2 binding site is required for the induction of chronic myeloid leukemia-like disease in mice by the Bcr/Abl tyrosine kinase

Studying the pathogenesis of BCR-ABL+ leukemia in mice: CML: Molecular mechanisms & therapeutic strategies

c-fos is not essential for v-abl-induced lymphomagenesis

Subcellular localization of bcr, Abl, and Bcr-Abl proteins in normal and leukemic cells and correlation of expression with myeloid differentiation

v-abl Causes Hematopoietic Disease Distinct From That Caused by bcr-abl

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