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B cell–intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice

Blood, 2012-03, Vol.119 (12), p.2819-2828

2012

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Increased Serum Levels of MRP-8/14 in Type 1 Diabetes Induce an Increased Expression of CD11b and an Enhanced Adhesion of Circulating Monocytes to Fibronectin

Diabetes (New York, N.Y.), 2004-08, Vol.53 (8), p.1979-1986

2004

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Reduced type I interferon production by dendritic cells and weakened antiviral immunity in patients with Wiskott-Aldrich syndrome protein deficiency

Journal of allergy and clinical immunology, 2013-03, Vol.131 (3), p.815-824.e2

2013

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Disease-associated missense mutations in the EVH1 domain disrupt intrinsic WASp function causing dysregulated actin dynamics and impaired dendritic cell migration

Blood, 2013-01, Vol.121 (1), p.72-84

2013

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Impaired T-cell priming in vivo resulting from dysfunction of WASp-deficient dendritic cells

Blood, 2007-12, Vol.110 (13), p.4278-4284

2007

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Recent advances in the understanding of genetic defects of neutrophil number and function

British journal of haematology, 2010-11, Vol.151 (4), p.312-326

2010

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Cytoskeletal remodeling mediated by WASp in dendritic cells is necessary for normal immune synapse formation and T-cell priming

Blood, 2011-09, Vol.118 (9), p.2492-2501

2011

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Migratory and Antigen Presentation Functions of IFN-Producing Killer Dendritic Cells

Cancer research (Chicago, Ill.), 2009-08, Vol.69 (16), p.6598-6606

2009

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An increased MRP8/14 expression and adhesion, but a decreased migration towards proinflammatory chemokines of type 1 diabetes monocytes

Clinical and experimental immunology, 2005-09, Vol.141 (3), p.509-517

2005

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Wiskott-Aldrich syndrome protein deficiency in B cells results in impaired peripheral homeostasis. Commentary

Blood, 2008, Vol.112 (10)

2008

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Brain-derived human immunodeficiency virus-1 Tat exerts differential effects on LTR transactivation and neuroimmune activation

Journal of neurovirology, 2007-04, Vol.13 (2), p.173-184

2007

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B cell–intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice

Increased Serum Levels of MRP-8/14 in Type 1 Diabetes Induce an Increased Expression of CD11b and an Enhanced Adhesion of Circulating Monocytes to Fibronectin

Reduced type I interferon production by dendritic cells and weakened antiviral immunity in patients with Wiskott-Aldrich syndrome protein deficiency

Disease-associated missense mutations in the EVH1 domain disrupt intrinsic WASp function causing dysregulated actin dynamics and impaired dendritic cell migration

Impaired T-cell priming in vivo resulting from dysfunction of WASp-deficient dendritic cells

Recent advances in the understanding of genetic defects of neutrophil number and function

Cytoskeletal remodeling mediated by WASp in dendritic cells is necessary for normal immune synapse formation and T-cell priming

Migratory and Antigen Presentation Functions of IFN-Producing Killer Dendritic Cells

An increased MRP8/14 expression and adhesion, but a decreased migration towards proinflammatory chemokines of type 1 diabetes monocytes

Wiskott-Aldrich syndrome protein deficiency in B cells results in impaired peripheral homeostasis. Commentary

Brain-derived human immunodeficiency virus-1 Tat exerts differential effects on LTR transactivation and neuroimmune activation

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