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Hemolytic disease of the newborn due to anti-Dib: a case study and review of the literature

Transfusion (Philadelphia, Pa.), 2006-03, Vol.46 (3), p.454-460

2006

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Sodium Channels in Normal and Pathological Pain

Annual review of neuroscience, 2010-01, Vol.33 (1), p.325-347

2010

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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

Brain (London, England : 1878), 2005-08, Vol.128 (8), p.1847-1854

2005

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Gain-of-function mutations in sodium channel Nav1.9 in painful neuropathy

Brain (London, England : 1878), 2014-06, Vol.137 (6), p.1627-1642

2014

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Gain of function NaV1.7 mutations in idiopathic small fiber neuropathy

Annals of neurology, 2012-01, Vol.71 (1), p.26-39

2012

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A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons

Experimental neurology, 2010-08, Vol.224 (2), p.362-368

2010

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Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

Neurology, 2012-05, Vol.78 (21), p.1635

2012

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A sodium channel gene SCN9A polymorphism that increases nociceptor excitability

Annals of neurology, 2009-12, Vol.66 (6), p.862-866

2009

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Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

Pain (Amsterdam), 1999-12, Vol.83 (3), p.591-600

1999

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Erythromelalgia: A hereditary pain syndrome enters the molecular era

Annals of neurology, 2005-06, Vol.57 (6), p.785-788

2005

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Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Experimental neurology, 2014-03, Vol.253, p.174-179

2014

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Deletion mutation of sodium channel NaV1.7 in inherited erythromelalgia: enhanced slow inactivation modulates dorsal root ganglion neuron hyperexcitability

Brain (London, England : 1878), 2011-07, Vol.134 (7), p.1972-1986

2011

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A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis

Annals of neurology, 2012-02, Vol.71 (2), p.186-194

2012

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Inherited erythermalgia: limb pain from an S4 charge-neutral Na channelopathy

Neurology, 2006-11, Vol.67 (9), p.1563

2006

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Characterization of a de novo SCN8A mutation in a patient with epileptic encephalopathy

Epilepsy research, 2014-11, Vol.108 (9), p.1511-1518

2014

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Voltage-gated sodium channels in pain states: Role in pathophysiology and targets for treatment

Brain Research Reviews, 2009-04, Vol.60 (1), p.65-83

2009

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Functional profiles of SCN9A variants in dorsal root ganglion neurons and superior cervical ganglion neurons correlate with autonomic symptoms in small fibre neuropathy

Brain (London, England : 1878), 2012-09, Vol.135 (Pt 9), p.2613-2628

2012

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Burden and health-related quality of life of Spanish caregivers of persons with multiple sclerosis

Multiple sclerosis, 2009-11, Vol.15 (11), p.1347-1355

2009

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Anti-JC virus antibody prevalence in a multinational multiple sclerosis cohort

Multiple sclerosis, 2013-10, Vol.19 (11), p.1533-1538

2013

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Wound-healing growth factor, basic FGF, induces Erk1/2-dependent mechanical hyperalgesia

Pain (Amsterdam), 2013-10, Vol.154 (10), p.2216-2226

2013

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Riluzole in Huntington's disease: a 3-year, randomized controlled study

Annals of neurology, 2007-09, Vol.62 (3), p.262-272

2007

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Alternative splicing may contribute to time-dependent manifestation of inherited erythromelalgia

Brain (London, England : 1878), 2010-06, Vol.133 (6), p.1823-1835

2010

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Amyotrophic lateral sclerosis: Progress and prospects for treatment

Drugs (New York, N.Y.), 2003, Vol.63 (3), p.289-310

DIB, Michel

2003

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Neuropathic pain in two-generation twins carrying the sodium channel Nav1.7 functional variant R1150W

Pain (Amsterdam), 2014-10, Vol.155 (10), p.2199-2203

2014

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Two randomized vitamin D trials in ambulatory patients on anticonvulsants: impact on bone

Neurology, 2006-12, Vol.67 (11), p.2005

2006

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Hemolytic disease of the newborn due to anti-Dib: a case study and review of the literature

Sodium Channels in Normal and Pathological Pain

Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

Gain-of-function mutations in sodium channel Nav1.9 in painful neuropathy

Gain of function NaV1.7 mutations in idiopathic small fiber neuropathy

A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons

Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

A sodium channel gene SCN9A polymorphism that increases nociceptor excitability

Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

Erythromelalgia: A hereditary pain syndrome enters the molecular era

Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Deletion mutation of sodium channel NaV1.7 in inherited erythromelalgia: enhanced slow inactivation modulates dorsal root ganglion neuron hyperexcitability

A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis

Inherited erythermalgia: limb pain from an S4 charge-neutral Na channelopathy

Characterization of a de novo SCN8A mutation in a patient with epileptic encephalopathy

Voltage-gated sodium channels in pain states: Role in pathophysiology and targets for treatment

Functional profiles of SCN9A variants in dorsal root ganglion neurons and superior cervical ganglion neurons correlate with autonomic symptoms in small fibre neuropathy

Burden and health-related quality of life of Spanish caregivers of persons with multiple sclerosis

Anti-JC virus antibody prevalence in a multinational multiple sclerosis cohort

Wound-healing growth factor, basic FGF, induces Erk1/2-dependent mechanical hyperalgesia

Riluzole in Huntington's disease: a 3-year, randomized controlled study

Alternative splicing may contribute to time-dependent manifestation of inherited erythromelalgia

Amyotrophic lateral sclerosis: Progress and prospects for treatment

Neuropathic pain in two-generation twins carrying the sodium channel Nav1.7 functional variant R1150W

Two randomized vitamin D trials in ambulatory patients on anticonvulsants: impact on bone

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