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Repeated social defeat causes increased anxiety-like behavior and alters splenocyte function in C57BL/6 and CD-1 mice

Brain, behavior, and immunity, 2007-05, Vol.21 (4), p.458-466

2007

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Social stress induces glucocorticoid resistance in macrophages

American journal of physiology. Regulatory, integrative and comparative physiology, 2001-06, Vol.280 (6), p.1799-R1805

2001

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Influenza Virus-Specific Immunological Memory Is Enhanced by Repeated Social Defeat

The Journal of immunology (1950), 2010-02, Vol.184 (4), p.2014-2025

2010

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Expression of glucocorticoid resistance following social stress requires a second signal

Journal of leukocyte biology, 2003-10, Vol.74 (4), p.507-513

2003

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Physical defeat reduces the sensitivity of murine splenocytes to the suppressive effects of corticosterone

Brain, behavior, and immunity, 2004-09, Vol.18 (5), p.416-424

2004

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Stress-induced modulation of NK activity during influenza viral infection: role of glucocorticoids and opioids

Brain, behavior, and immunity, 2005-03, Vol.19 (2), p.153-164

2005

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Interleukin-6 and the development of social disruption-induced glucocorticoid resistance

Journal of neuroimmunology, 2002-03, Vol.124 (1), p.9-15

2002

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Social disruption-induced glucocorticoid resistance: kinetics and site specificity

Journal of neuroimmunology, 2002-03, Vol.124 (1), p.54-61

2002

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Modulation of natural killer cell activity by restraint stress during an influenza A/PR8 infection in mice

Brain, behavior, and immunity, 2004-11, Vol.18 (6), p.526-535

2004

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Subordinate social status modulates the vulnerability to the immunological effects of social stress

Psychoneuroendocrinology, 2007-09, Vol.32 (8), p.1097-1105

2007

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Moderate exercise protects mice from death due to influenza virus

Brain, behavior, and immunity, 2005-09, Vol.19 (5), p.377-380

2005

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The inflammatory response to social defeat is increased in older mice

Physiology & behavior, 2008-02, Vol.93 (3), p.628-636

2008

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Moderate exercise early after influenza virus infection reduces the Th1 inflammatory response in lungs of mice

Exercise immunology review, 2006, Vol.12, p.97-111

2006

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Social stress increases the susceptibility to endotoxic shock

Journal of neuroimmunology, 2001-04, Vol.115 (1), p.36-45

2001

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Experimental Models of Stress and Wound Healing

World journal of surgery, 2004-03, Vol.28 (3), p.327-330

2004

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Androstenediol and dehydroepiandrosterone protect mice against lethal bacterial infections and lipopolysaccharide toxicity

Journal of medical microbiology, 1999-05, Vol.48 (5), p.425-431

1999

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Androstenediol reduces the anti-inflammatory effects of restraint stress during wound healing

Brain, behavior, and immunity, 2006-11, Vol.20 (6), p.590-596

2006

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Restraint stress alters lung gene expression in an experimental influenza A viral infection

Journal of neuroimmunology, 2005-05, Vol.162 (1), p.103-111

2005

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Epstein-Barr virus-encoded dUTPase modulates immune function and induces sickness behavior in mice

Journal of medical virology, 2004-11, Vol.74 (3), p.442-448

2004

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Endocrine regulation of the immune response to influenza virus infection with a metabolite of DHEA-androstenediol

Journal of neuroimmunology, 1997-09, Vol.78 (1), p.203-211

1997

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Androstenediol (AED) prevents neuroendocrine-mediated suppression of the immune response to an influenza viral infection

Journal of neuroimmunology, 1999-08, Vol.98 (2), p.121-129

1999

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Stress Exacerbates Age-Related Decrements in the Immune Response to an Experimental Influenza Viral Infection

The journals of gerontology. Series A, Biological sciences and medical sciences, 1998-09, Vol.53A (5), p.B347-B354

1998

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Control of the immune response by DHEA and its metabolites

Rinsho byori. The Japanese journal of clinical pathology, 1998-06, Vol.46 (6), p.505

1998

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In vitro potentiation of lymphocyte activation by dehydroepiandrosterone, androstenediol, and androstenetriol

The Journal of immunology (1950), 1994-08, Vol.153 (4), p.1544-1552

1994

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Social stress enhances IL-1beta and TNF-alpha production by Porphyromonas gingivalis lipopolysaccharide-stimulated CD11b+ cells

Physiology & behavior, 2009-09, Vol.98 (3), p.351-358

2009

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Repeated social defeat causes increased anxiety-like behavior and alters splenocyte function in C57BL/6 and CD-1 mice

Social stress induces glucocorticoid resistance in macrophages

Influenza Virus-Specific Immunological Memory Is Enhanced by Repeated Social Defeat

Expression of glucocorticoid resistance following social stress requires a second signal

Physical defeat reduces the sensitivity of murine splenocytes to the suppressive effects of corticosterone

Stress-induced modulation of NK activity during influenza viral infection: role of glucocorticoids and opioids

Interleukin-6 and the development of social disruption-induced glucocorticoid resistance

Social disruption-induced glucocorticoid resistance: kinetics and site specificity

Modulation of natural killer cell activity by restraint stress during an influenza A/PR8 infection in mice

Subordinate social status modulates the vulnerability to the immunological effects of social stress

Moderate exercise protects mice from death due to influenza virus

The inflammatory response to social defeat is increased in older mice

Moderate exercise early after influenza virus infection reduces the Th1 inflammatory response in lungs of mice

Social stress increases the susceptibility to endotoxic shock

Experimental Models of Stress and Wound Healing

Androstenediol and dehydroepiandrosterone protect mice against lethal bacterial infections and lipopolysaccharide toxicity

Androstenediol reduces the anti-inflammatory effects of restraint stress during wound healing

Restraint stress alters lung gene expression in an experimental influenza A viral infection

Epstein-Barr virus-encoded dUTPase modulates immune function and induces sickness behavior in mice

Endocrine regulation of the immune response to influenza virus infection with a metabolite of DHEA-androstenediol

Androstenediol (AED) prevents neuroendocrine-mediated suppression of the immune response to an influenza viral infection

Stress Exacerbates Age-Related Decrements in the Immune Response to an Experimental Influenza Viral Infection

Control of the immune response by DHEA and its metabolites

In vitro potentiation of lymphocyte activation by dehydroepiandrosterone, androstenediol, and androstenetriol

Social stress enhances IL-1beta and TNF-alpha production by Porphyromonas gingivalis lipopolysaccharide-stimulated CD11b+ cells

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