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Effects of resveratrol and SIRT1 on PGC-1α activity and mitochondrial biogenesis: a reevaluation

PLoS biology, 2013-07, Vol.11 (7), p.e1001603-e1001603

2013

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Does calorie restriction induce mitochondrial biogenesis? A reevaluation

The FASEB journal, 2011-02, Vol.25 (2), p.785-791

2011

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High-fat diets cause insulin resistance despite an increase in muscle mitochondria

Proceedings of the National Academy of Sciences - PNAS, 2008-06, Vol.105 (22), p.7815-7820

2008

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Normal adaptations to exercise despite protection against oxidative stress

American journal of physiology: endocrinology and metabolism, 2011-11, Vol.301 (5), p.E779-E784

2011

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Raising plasma fatty acid concentration induces increased biogenesis of mitochondria in skeletal muscle

Proceedings of the National Academy of Sciences - PNAS, 2007-06, Vol.104 (25), p.10709-10713

2007

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PGC‐1α mediates a rapid, exercise‐induced downregulation of glycogenolysis in rat skeletal muscle

The Journal of physiology, 2015-02, Vol.593 (3), p.635-643

2015

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PPARβ Is Essential for Maintaining Normal Levels of PGC-1α and Mitochondria and for the Increase in Muscle Mitochondria Induced by Exercise

Cell metabolism, 2017-05, Vol.25 (5), p.1176-1185.e5

2017

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Activation of AMP kinase enhances sensitivity of muscle glucose transport to insulin

American journal of physiology: endocrinology and metabolism, 2002-01, Vol.282 (1), p.E18-E23

2002

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Skeletal muscle respiratory uncoupling prevents diet-induced obesity and insulin resistance in mice

Nature medicine, 2000-10, Vol.6 (10), p.1115-1120

2000

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How muscle insulin sensitivity is regulated: testing of a hypothesis

American journal of physiology: endocrinology and metabolism, 2006-12, Vol.291 (6), p.E1258-E1263

2006

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The nuclear receptor PPARβ/δ programs muscle glucose metabolism in cooperation with AMPK and MEF2

Genes & development, 2011-12, Vol.25 (24), p.2619-2630

2011

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IL-6 increases muscle insulin sensitivity only at superphysiological levels

American journal of physiology: endocrinology and metabolism, 2007-06, Vol.292 (6), p.E1842-E1846

2007

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Prevention of glycogen supercompensation prolongs the increase in muscle GLUT4 after exercise

American journal of physiology: endocrinology and metabolism, 2003-10, Vol.285 (4), p.E729-E736

2003

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High-fat diet-induced muscle insulin resistance: relationship to visceral fat mass

American journal of physiology. Regulatory, integrative and comparative physiology, 2000-12, Vol.279 (6), p.2057-R2065

2000

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Ca2+ and AMPK Both Mediate Stimulation of Glucose Transport by Muscle Contractions

Diabetes (New York, N.Y.), 2004-02, Vol.53 (2), p.330-335

2004

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Calcineurin Does Not Mediate Exercise-Induced Increase in Muscle GLUT4

Diabetes (New York, N.Y.), 2005-03, Vol.54 (3), p.624-628

2005

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Insulin resistance of muscle glucose transport in rats fed a high-fat diet: a reevaluation

Diabetes (New York, N.Y.), 1997-11, Vol.46 (11), p.1761

1997

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Calpain System Regulates Muscle Mass and Glucose Transporter GLUT4 Turnover

The Journal of biological chemistry, 2004-05, Vol.279 (20), p.20915-20920

2004

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Dehydroepiandrosterone (DHEA) replacement decreases insulin resistance and lowers inflammatory cytokines in aging humans

Aging (Albany, NY.), 2011-05, Vol.3 (5), p.533-542

2011

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UCP-mediated energy depletion in skeletal muscle increases glucose transport despite lipid accumulation and mitochondrial dysfunction

American journal of physiology: endocrinology and metabolism, 2004-03, Vol.286 (3), p.E347-E353

2004

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β-Adrenergic stimulation does not activate p38 MAP kinase or induce PGC-1α in skeletal muscle

American journal of physiology: endocrinology and metabolism, 2013-04, Vol.304 (8), p.E844-E852

2013

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Mechanisms underlying impaired GLUT-4 translocation in glycogen-supercompensated muscles of exercised rats

American journal of physiology: endocrinology and metabolism, 2000-12, Vol.279 (6), p.E1311-E1318

2000

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Are tyrosine kinases involved in mediating contraction-stimulated muscle glucose transport?

American journal of physiology: endocrinology and metabolism, 2006-01, Vol.290 (1), p.E123-E128

2006

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Contraction- and hypoxia-stimulated glucose transport is mediated by a Ca2+-dependent mechanism in slow-twitch rat soleus muscle

American journal of physiology: endocrinology and metabolism, 2005-06, Vol.288 (6), p.E1062-E1066

2005

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Glucose transport rate and glycogen synthase activity both limit skeletal muscle glycogen accumulation

American journal of physiology: endocrinology and metabolism, 2002-06, Vol.282 (6), p.E1214-E1221

2002

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Effects of resveratrol and SIRT1 on PGC-1α activity and mitochondrial biogenesis: a reevaluation

Does calorie restriction induce mitochondrial biogenesis? A reevaluation

High-fat diets cause insulin resistance despite an increase in muscle mitochondria

Normal adaptations to exercise despite protection against oxidative stress

Raising plasma fatty acid concentration induces increased biogenesis of mitochondria in skeletal muscle

PGC‐1α mediates a rapid, exercise‐induced downregulation of glycogenolysis in rat skeletal muscle

PPARβ Is Essential for Maintaining Normal Levels of PGC-1α and Mitochondria and for the Increase in Muscle Mitochondria Induced by Exercise

Activation of AMP kinase enhances sensitivity of muscle glucose transport to insulin

Skeletal muscle respiratory uncoupling prevents diet-induced obesity and insulin resistance in mice

How muscle insulin sensitivity is regulated: testing of a hypothesis

The nuclear receptor PPARβ/δ programs muscle glucose metabolism in cooperation with AMPK and MEF2

IL-6 increases muscle insulin sensitivity only at superphysiological levels

Prevention of glycogen supercompensation prolongs the increase in muscle GLUT4 after exercise

High-fat diet-induced muscle insulin resistance: relationship to visceral fat mass

Ca2+ and AMPK Both Mediate Stimulation of Glucose Transport by Muscle Contractions

Calcineurin Does Not Mediate Exercise-Induced Increase in Muscle GLUT4

Insulin resistance of muscle glucose transport in rats fed a high-fat diet: a reevaluation

Calpain System Regulates Muscle Mass and Glucose Transporter GLUT4 Turnover

Dehydroepiandrosterone (DHEA) replacement decreases insulin resistance and lowers inflammatory cytokines in aging humans

UCP-mediated energy depletion in skeletal muscle increases glucose transport despite lipid accumulation and mitochondrial dysfunction

β-Adrenergic stimulation does not activate p38 MAP kinase or induce PGC-1α in skeletal muscle

Mechanisms underlying impaired GLUT-4 translocation in glycogen-supercompensated muscles of exercised rats

Are tyrosine kinases involved in mediating contraction-stimulated muscle glucose transport?

Contraction- and hypoxia-stimulated glucose transport is mediated by a Ca2+-dependent mechanism in slow-twitch rat soleus muscle

Glucose transport rate and glycogen synthase activity both limit skeletal muscle glycogen accumulation

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