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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

Brain (London, England : 1878), 2005-08, Vol.128 (8), p.1847-1854

2005

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Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons

Journal of neurophysiology, 2001-08, Vol.86 (2), p.629

2001

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Vasodilatation in the rat dorsal hindpaw induced by activation of sensory neurons is reduced by paclitaxel

Neurotoxicology (Park Forest South), 2011-01, Vol.32 (1), p.140-149

2011

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Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons

The Journal of neuroscience, 2000-12, Vol.20 (23), p.8754-8761

2000

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Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons

The Journal of neuroscience, 2001-08, Vol.21 (16), p.5952-5961

2001

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Impairment of slow inactivation as a common mechanism for periodic paralysis in DIIS4-S5

Neurology, 2002-04, Vol.58 (8), p.1266-1272

2002

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SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain model

Neuroreport, 1998-04, Vol.9 (6), p.967-972

1998

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alpha -SNS Produces the Slow TTX-Resistant Sodium Current in Large Cutaneous Afferent DRG Neurons

Journal of neurophysiology, 2000-08, Vol.84 (2), p.710-718

2000

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Molecular and Functional Remodeling of Electrogenic Membrane of Hypothalamic Neurons in Response to Changes in Their Input

Proceedings of the National Academy of Sciences - PNAS, 1999-02, Vol.96 (3), p.1088-1093

1999

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The pentapeptide QYNAD does not block voltage-gated sodium channels

Neurology, 2003-01, Vol.60 (2), p.224-229

2003

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Dopamine transporter genotype predicts behavioural and neural measures of response inhibition

Molecular psychiatry, 2012-11, Vol.17 (11), p.1086-1092

2012

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Functional properties of rat and human neocortical voltage-sensitive sodium currents

Journal of neurophysiology, 1994-03, Vol.71 (3), p.1052-1064

1994

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Rare DNA variants in the brain-derived neurotrophic factor gene increase risk for attention-deficit hyperactivity disorder: a next-generation sequencing study

Molecular psychiatry, 2017-04, Vol.22 (4), p.580-584

2017

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Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

Pain (Amsterdam), 2004-04, Vol.108 (3), p.237-247

2004

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In Vivo NGF Deprivation Reduces SNS Expression and TTX-R Sodium Currents in IB4-Negative DRG Neurons

Journal of neurophysiology, 1999-02, Vol.81 (2), p.803-810

1999

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Alpha-2A adrenergic receptor gene variants are associated with increased intra-individual variability in response time

Molecular psychiatry, 2014-09, Vol.19 (9), p.1031-1036

2014

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Sodium channel inactivation defects are associated with acetazolamide-exacerbated hypokalemic periodic paralysis

Annals of neurology, 2001-09, Vol.50 (3), p.417-420

2001

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Activation and Inactivation of the Voltage-Gated Sodium Channel: Role of Segment S5 Revealed by a Novel Hyperkalaemic Periodic Paralysis Mutation

The Journal of neuroscience, 1999-06, Vol.19 (12), p.4762-4771

1999

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GDNF and NGF Reverse Changes in Repriming of TTX-Sensitive Na+ Currents Following Axotomy of Dorsal Root Ganglion Neurons

Journal of neurophysiology, 2002-08, Vol.88 (2), p.650-658

2002

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Increased Resurgent Sodium Currents in Nav1.8 Contribute to Nociceptive Sensory Neuron Hyperexcitability Associated with Peripheral Neuropathies

The Journal of neuroscience, 2019-02, Vol.39 (8), p.1539-1550

2019

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Differential block of sensory neuronal voltage-gated sodium channels by lacosamide [(2R)-2-(acetylamino)-N-benzyl-3-methoxypropanamide], lidocaine, and carbamazepine

The Journal of pharmacology and experimental therapeutics, 2008-07, Vol.326 (1), p.89-99

2008

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CAP-1A is a novel linker that binds clathrin and the voltage-gated sodium channel Na(v)1.8

Molecular and cellular neuroscience, 2005-04, Vol.28 (4), p.636-649

2005

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Characterization of a new sodium channel mutation at arginine 1448 associated with moderate paramyotonia congenita in humans

The Journal of physiology, 1999-07, Vol.518 (2), p.337-344

1999

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A double mutation in families with periodic paralysis defines new aspects of sodium channel slow inactivation

The Journal of clinical investigation, 2000-08, Vol.106 (3), p.431-438

2000

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Sodium channel expression in hypothalamic osmosensitive neurons in experimental diabetes

Neuroreport, 2002-08, Vol.13 (11), p.1481-1484

2002

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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons

Vasodilatation in the rat dorsal hindpaw induced by activation of sensory neurons is reduced by paclitaxel

Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons

Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons

Impairment of slow inactivation as a common mechanism for periodic paralysis in DIIS4-S5

SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain model

alpha -SNS Produces the Slow TTX-Resistant Sodium Current in Large Cutaneous Afferent DRG Neurons

Molecular and Functional Remodeling of Electrogenic Membrane of Hypothalamic Neurons in Response to Changes in Their Input

The pentapeptide QYNAD does not block voltage-gated sodium channels

Dopamine transporter genotype predicts behavioural and neural measures of response inhibition

Functional properties of rat and human neocortical voltage-sensitive sodium currents

Rare DNA variants in the brain-derived neurotrophic factor gene increase risk for attention-deficit hyperactivity disorder: a next-generation sequencing study

Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

In Vivo NGF Deprivation Reduces SNS Expression and TTX-R Sodium Currents in IB4-Negative DRG Neurons

Alpha-2A adrenergic receptor gene variants are associated with increased intra-individual variability in response time

Sodium channel inactivation defects are associated with acetazolamide-exacerbated hypokalemic periodic paralysis

Activation and Inactivation of the Voltage-Gated Sodium Channel: Role of Segment S5 Revealed by a Novel Hyperkalaemic Periodic Paralysis Mutation

GDNF and NGF Reverse Changes in Repriming of TTX-Sensitive Na+ Currents Following Axotomy of Dorsal Root Ganglion Neurons

Increased Resurgent Sodium Currents in Nav1.8 Contribute to Nociceptive Sensory Neuron Hyperexcitability Associated with Peripheral Neuropathies

Differential block of sensory neuronal voltage-gated sodium channels by lacosamide [(2R)-2-(acetylamino)-N-benzyl-3-methoxypropanamide], lidocaine, and carbamazepine

CAP-1A is a novel linker that binds clathrin and the voltage-gated sodium channel Na(v)1.8

Characterization of a new sodium channel mutation at arginine 1448 associated with moderate paramyotonia congenita in humans

A double mutation in families with periodic paralysis defines new aspects of sodium channel slow inactivation

Sodium channel expression in hypothalamic osmosensitive neurons in experimental diabetes

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