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High Dose Intravenous IgG Therapy Modulates Multiple NK Cell and T Cell Functions in Patients With Immune Dysregulation

Frontiers in immunology, 2021-05, Vol.12, p.660506-660506

2021

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Selective predisposition to bacterial infections in IRAK-4-deficient children: IRAK-4-dependent TLRs are otherwise redundant in protective immunity

The Journal of experimental medicine, 2007-10, Vol.204 (10), p.2407-2422

2007

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Endothelial growth factors VEGF and bFGF differentially enhance monocyte and neutrophil recruitment to inflammation

Journal of leukocyte biology, 2006-08, Vol.80 (2), p.247-257

2006

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Intravenous IgG (IVIG) and subcutaneous IgG (SCIG) preparations have comparable inhibitory effect on T cell activation, which is not dependent on IgG sialylation, monocytes or B cells

Clinical immunology (Orlando, Fla.), 2015-10, Vol.160 (2), p.123-132

2015

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Impaired T-cell receptor activation in IL-1 receptor–associated kinase-4–deficient patients

Journal of allergy and clinical immunology, 2010-08, Vol.126 (2), p.332-337.e2

2010

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Role of ICAM-1 and ICAM-2 and alternate CD11/CD18 ligands in neutrophil transendothelial migration

Journal of leukocyte biology, 1999-01, Vol.65 (1), p.117-126

1999

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Growth factor regulation of neutrophil-endothelial cell interactions

Journal of leukocyte biology, 2001-08, Vol.70 (2), p.225-232

2001

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Menopause and Ovariectomy Cause a Low Grade of Systemic Inflammation that May Be Prevented by Chronic Treatment with Low Doses of Estrogen or Losartan

The Journal of immunology (1950), 2009-07, Vol.183 (2), p.1393-1402

2009

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Trans- but not cis-resveratrol impairs angiotensin-II-mediated vascular inflammation through inhibition of NF-κB activation and peroxisome proliferator-activated receptor-gamma upregulation

The Journal of immunology (1950), 2010-09, Vol.185 (6), p.3718-3727

2010

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Neutrophil differentiated HL‐60 cells model Mac‐1 (CD11b/CD18)‐independent neutrophil transepithelial migration

Immunology, 2005-05, Vol.115 (1), p.108-117

2005

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Potentiation of cytokine-induced proliferation of human Natural Killer cells by intravenous immunoglobulin G

Clinical immunology (Orlando, Fla.), 2015-12, Vol.161 (2), p.373-383

2015

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Fatal Pneumococcal Meningitis in a 7-Year-Old Girl with Interleukin-1 Receptor Activated Kinase Deficiency (IRAK-4) Despite Prophylactic Antibiotic and IgG Responses to Streptococcus Pneumoniae Vaccines

Journal of clinical immunology, 2014-04, Vol.34 (3), p.267-271

2014

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Modulation of adjuvant arthritis in the rat by 2-methoxyestradiol: An effect independent of an anti-angiogenic action

International immunopharmacology, 2008-05, Vol.8 (5), p.708-716

2008

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Migration of polymorphonuclear leucocytes is influenced by dendritic cells

Immunology, 2005-03, Vol.114 (3), p.375-385

2005

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Differential role of E‐selectin and P‐selectin in T lymphocyte migration to cutaneous inflammatory reactions induced by cytokines

International immunology, 2002-07, Vol.14 (7), p.751-760

2002

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Cross-Talk between CD31 and the Signaling Lymphocytic Activation Molecule—Associated Protein during Interferon-γ Production against Mycobacterium tuberculosis

The Journal of infectious diseases, 2007-11, Vol.196 (9), p.1369-1378

2007

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Intravenous immunoglobulin G-mediated inhibition of T-cell proliferation reflects an endogenous mechanism by which IgG modulates T-cell activation

Clinical immunology (Orlando, Fla.), 2009-08, Vol.132 (2), p.222-233

2009

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Intravenous immunoglobulin G (IVIG) inhibits IL-1- and TNF-α-dependent, but not chemotactic-factor-stimulated, neutrophil transendothelial migration

Clinical immunology (Orlando, Fla.), 2011-11, Vol.141 (2), p.187-196

2011

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The Role of E-Selectin, P-Selectin, and Very Late Activation Antigen-4 in T Lymphocyte Migration to Dermal Inflammation

The Journal of immunology (1950), 2002-02, Vol.168 (4), p.1934-1939

2002

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Neutrophil migration stimulates rat intestinal epithelial cell cytokine expression during helminth infection

Journal of leukocyte biology, 2000-12, Vol.68 (6), p.821-827

2000

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Angiotensin II-Induced Mononuclear Leukocyte Interactions with Arteriolar and Venular Endothelium Are Mediated by the Release of Different CC Chemokines

Journal of Immunology, 2006-05, Vol.176 (9), p.5577-5586

2006

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Intravenous immunoglobulin G selectively inhibits IL-1α-induced neutrophil-endothelial cell adhesion

Autoimmunity (Chur, Switzerland), 2010-12, Vol.43 (8), p.619-627

2010

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CD11/CD18-independent transendothelial migration of human polymorphonuclear leukocytes and monocytes: involvement of distinct and unique mechanisms

Journal of leukocyte biology, 1995-04, Vol.57 (4), p.553-561

1995

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Pseudomonas aeruginosa activates human mast cells to induce neutrophil transendothelial migration via mast cell-derived IL-1 alpha and beta

The Journal of immunology (1950), 2002-10, Vol.169 (8), p.4522-4530

2002

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The CD11 CD18 ( ) integrins modulate neutrophil caspase activation and survival following TNF-α or endotoxin induced transendothelial migration

Immunology and cell biology, 2004-08, Vol.82 (4), p.435-446

2004

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High Dose Intravenous IgG Therapy Modulates Multiple NK Cell and T Cell Functions in Patients With Immune Dysregulation

Selective predisposition to bacterial infections in IRAK-4-deficient children: IRAK-4-dependent TLRs are otherwise redundant in protective immunity

Endothelial growth factors VEGF and bFGF differentially enhance monocyte and neutrophil recruitment to inflammation

Intravenous IgG (IVIG) and subcutaneous IgG (SCIG) preparations have comparable inhibitory effect on T cell activation, which is not dependent on IgG sialylation, monocytes or B cells

Impaired T-cell receptor activation in IL-1 receptor–associated kinase-4–deficient patients

Role of ICAM-1 and ICAM-2 and alternate CD11/CD18 ligands in neutrophil transendothelial migration

Growth factor regulation of neutrophil-endothelial cell interactions

Menopause and Ovariectomy Cause a Low Grade of Systemic Inflammation that May Be Prevented by Chronic Treatment with Low Doses of Estrogen or Losartan

Trans- but not cis-resveratrol impairs angiotensin-II-mediated vascular inflammation through inhibition of NF-κB activation and peroxisome proliferator-activated receptor-gamma upregulation

Neutrophil differentiated HL‐60 cells model Mac‐1 (CD11b/CD18)‐independent neutrophil transepithelial migration

Potentiation of cytokine-induced proliferation of human Natural Killer cells by intravenous immunoglobulin G

Fatal Pneumococcal Meningitis in a 7-Year-Old Girl with Interleukin-1 Receptor Activated Kinase Deficiency (IRAK-4) Despite Prophylactic Antibiotic and IgG Responses to Streptococcus Pneumoniae Vaccines

Modulation of adjuvant arthritis in the rat by 2-methoxyestradiol: An effect independent of an anti-angiogenic action

Migration of polymorphonuclear leucocytes is influenced by dendritic cells

Differential role of E‐selectin and P‐selectin in T lymphocyte migration to cutaneous inflammatory reactions induced by cytokines

Cross-Talk between CD31 and the Signaling Lymphocytic Activation Molecule—Associated Protein during Interferon-γ Production against Mycobacterium tuberculosis

Intravenous immunoglobulin G-mediated inhibition of T-cell proliferation reflects an endogenous mechanism by which IgG modulates T-cell activation

Intravenous immunoglobulin G (IVIG) inhibits IL-1- and TNF-α-dependent, but not chemotactic-factor-stimulated, neutrophil transendothelial migration

The Role of E-Selectin, P-Selectin, and Very Late Activation Antigen-4 in T Lymphocyte Migration to Dermal Inflammation

Neutrophil migration stimulates rat intestinal epithelial cell cytokine expression during helminth infection

Angiotensin II-Induced Mononuclear Leukocyte Interactions with Arteriolar and Venular Endothelium Are Mediated by the Release of Different CC Chemokines

Intravenous immunoglobulin G selectively inhibits IL-1α-induced neutrophil-endothelial cell adhesion

CD11/CD18-independent transendothelial migration of human polymorphonuclear leukocytes and monocytes: involvement of distinct and unique mechanisms

Pseudomonas aeruginosa activates human mast cells to induce neutrophil transendothelial migration via mast cell-derived IL-1 alpha and beta

The CD11 CD18 ( ) integrins modulate neutrophil caspase activation and survival following TNF-α or endotoxin induced transendothelial migration

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