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Non-melanoma Skin Cancer in Canada Chapter 3: Management of Actinic Keratoses

Journal of cutaneous medicine and surgery, 2015-05, Vol.19 (3), p.227-238

2015

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Gene expression profiling following NRF2 and KEAP1 siRNA knockdown in human lung fibroblasts identifies CCL11/Eotaxin-1 as a novel NRF2 regulated gene

Respiratory research, 2012-10, Vol.13 (1), p.92-92, Article 92

2012

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Non-melanoma Skin Cancer in Canada Chapter 5: Management of Squamous Cell Carcinoma

Journal of cutaneous medicine and surgery, 2015-05, Vol.19 (3), p.249-259

2015

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Non-melanoma Skin Cancer in Canada Chapter 4: Management of Basal Cell Carcinoma

Journal of cutaneous medicine and surgery, 2015-05, Vol.19 (3), p.239-248

2015

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Discovery of disubstituted phenanthrene imidazoles as potent, selective and orally active mPGES-1 inhibitors

Bioorganic & medicinal chemistry letters, 2009-10, Vol.19 (20), p.5837-5841

2009

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Substituted phenanthrene imidazoles as potent, selective, and orally active mPGES-1 inhibitors

Bioorganic & medicinal chemistry letters, 2007-12, Vol.17 (24), p.6816-6820

2007

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Non-melanoma Skin Cancer in Canada Chapter 2: Primary Prevention of Non-melanoma Skin Cancer

Journal of cutaneous medicine and surgery, 2015-05, Vol.19 (3), p.216-226

2015

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Non-melanoma Skin Cancer in Canada Chapter 1: Introduction to the Guidelines

Journal of cutaneous medicine and surgery, 2015-05, Vol.19 (3), p.205-215

2015

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Trisubstituted ureas as potent and selective mPGES-1 inhibitors

Bioorganic & medicinal chemistry letters, 2011-03, Vol.21 (5), p.1488-1492

2011

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Pyridazinones as selective cyclooxygenase-2 inhibitors

Bioorganic & medicinal chemistry letters, 2003-02, Vol.13 (4), p.597-600

2003

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Management of Malignant Melanoma: Best Practices

Journal of cutaneous medicine and surgery, 2009-03, Vol.13 (2), p.55-73

2009

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Design, synthesis, and biological evaluation of 8-biarylquinolines: A novel class of PDE4 inhibitors

Bioorganic & medicinal chemistry, 2008-02, Vol.18 (4), p.1407-1412

2008

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2,3-Diarylcyclopentenones as Orally Active, Highly Selective Cyclooxygenase-2 Inhibitors

Journal of medicinal chemistry, 1999-04, Vol.42 (7), p.1274-1281

1999

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Alkyl-bridged substituted 8-arylquinolines as highly potent PDE IV inhibitors

Bioorganic & medicinal chemistry letters, 2009-09, Vol.19 (17), p.5266-5269

2009

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Comparison of inhibition of ovalbumin-induced bronchoconstriction in guinea pigs and in vitro inhibition of tumor necrosis factor-α formation with phosphodiesterase 4 (PDE4) selective inhibitors

Biochemical pharmacology, 2002-04, Vol.63 (8), p.1527-1535

2002

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Non-melanoma Skin Cancer in Canada Chapter 3: Management of Actinic Keratoses

Gene expression profiling following NRF2 and KEAP1 siRNA knockdown in human lung fibroblasts identifies CCL11/Eotaxin-1 as a novel NRF2 regulated gene

Non-melanoma Skin Cancer in Canada Chapter 5: Management of Squamous Cell Carcinoma

Non-melanoma Skin Cancer in Canada Chapter 4: Management of Basal Cell Carcinoma

Discovery of disubstituted phenanthrene imidazoles as potent, selective and orally active mPGES-1 inhibitors

Substituted phenanthrene imidazoles as potent, selective, and orally active mPGES-1 inhibitors

Non-melanoma Skin Cancer in Canada Chapter 2: Primary Prevention of Non-melanoma Skin Cancer

Non-melanoma Skin Cancer in Canada Chapter 1: Introduction to the Guidelines

Trisubstituted ureas as potent and selective mPGES-1 inhibitors

Pyridazinones as selective cyclooxygenase-2 inhibitors

Management of Malignant Melanoma: Best Practices

Discovery of a substituted 8-arylquinoline series of PDE4 inhibitors: Structure–activity relationship, optimization, and identification of a highly potent, well tolerated, PDE4 inhibitor

Design, synthesis, and biological evaluation of 8-biarylquinolines: A novel class of PDE4 inhibitors

2,3-Diarylcyclopentenones as Orally Active, Highly Selective Cyclooxygenase-2 Inhibitors

Alkyl-bridged substituted 8-arylquinolines as highly potent PDE IV inhibitors

Comparison of inhibition of ovalbumin-induced bronchoconstriction in guinea pigs and in vitro inhibition of tumor necrosis factor-α formation with phosphodiesterase 4 (PDE4) selective inhibitors

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