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Nitric Oxide Increases Arterial Endotheial Permeability through Mediating VE-Cadherin Expression during Arteriogenesis

PloS one, 2015-07, Vol.10 (7), p.e0127931-e0127931

2015

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Fibrosis in endstage human heart failure: Severe changes in collagen metabolism and MMP/TIMP profiles

International journal of cardiology, 2011-08, Vol.151 (1), p.18-33

2011

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Myocytes Die by Multiple Mechanisms in Failing Human Hearts

Circulation research, 2003-04, Vol.92 (7), p.715-724

2003

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Increased Expression of Cytoskeletal, Linkage, and Extracellular Proteins in Failing Human Myocardium

Circulation research, 2000-04, Vol.86 (8), p.846-853

2000

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Human hibernating myocardium is jeopardized by apoptotic and autophagic cell death

Journal of the American College of Cardiology, 2004-06, Vol.43 (12), p.2191-2199

2004

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Unresolved Issues Regarding the Role of Apoptosis in the Pathogenesis of Ischemic Injury and Heart Failure

Journal of Molecular and Cellular Cardiology, 2000-05, Vol.32 (5), p.711-724

2000

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Apoptosis is Initiated by Myocardial Ischemia and Executed During Reperfusion

Journal of molecular and cellular cardiology, 2000-02, Vol.32 (2), p.197-208

2000

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Activation of the cardiac renin-angiotensin system and increased myocardial collagen expression in human aortic valve disease

Journal of the American College of Cardiology, 2001-04, Vol.37 (5), p.1443-1449

2001

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The role of Fas/APO 1 and apoptosis in the development of human atherosclerotic lesions

Atherosclerosis, 1997-06, Vol.131 (2), p.177-186

1997

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The cytoskeleton and related proteins in the human failing heart

Heart failure reviews, 2000-10, Vol.5 (3), p.271-280

2000

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The role of apoptosis in myocardial ischemia: a critical appraisal

Basic research in cardiology, 2001-05, Vol.96 (3), p.219-226

2001

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Translocation and Cleavage of Myocardial Dystrophin as a Common Pathway to Advanced Heart Failure: A Scheme for the Progression of Cardiac Dysfunction

Proceedings of the National Academy of Sciences - PNAS, 2004-05, Vol.101 (19), p.7381-7385

2004

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The extracellular matrix in normal and diseased myocardium

Journal of nuclear cardiology, 2001-03, Vol.8 (2), p.188-196

2001

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Ischemia induces early changes to cytoskeletal and contractile proteins in diseased human myocardium

The Journal of thoracic and cardiovascular surgery, 1995-07, Vol.110 (1), p.89-98

1995

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Matrix metalloproteinases and their tissue inhibitors in pressure-overloaded human myocardium during heart failure progression

Journal of the American College of Cardiology, 2004-10, Vol.44 (8), p.1609-1618

2004

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Reduced sarcoplasmic reticulum Ca2+ -ATPase activity and dephosphorylated phospholamban contribute to contractile dysfunction in human hibernating myocardium

Molecular and cellular biochemistry, 2006-01, Vol.282 (1-2), p.53-63

2006

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Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: Structural deterioration and compensatory mechanisms

Circulation (New York, N.Y.), 2003-02, Vol.107 (7), p.984-991

2003

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Structural correlate of atrial fibrillation in human patients

Cardiovascular research, 2002-05, Vol.54 (2), p.361-379

2002

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Connexin 43 expression and distribution in compensated and decompensated cardiac hypertrophy in patients with aortic stenosis

Cardiovascular research, 2004-05, Vol.62 (2), p.426-436

2004

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Apoptosis: measurement and technical issues

Journal of molecular and cellular cardiology, 2005, Vol.38 (1), p.15-20

2005

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The role of the cytoskeleton in heart failure

Cardiovascular research, 2000-01, Vol.45 (2), p.273-278

2000

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Altered Expression of Titin and Contractile Proteins in Failing Human Myocardium

Journal of molecular and cellular cardiology, 1994-10, Vol.26 (10), p.1291-1306

1994

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The extracellular matrix in the failing human heart

Basic research in cardiology, 1992, Vol.87 Suppl 1, p.303-309

1992

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The carboxyl-terminal region of ahnak provides a link between cardiac L-type Ca2+ channels and the actin-based cytoskeleton

The FASEB journal, 2002-08, Vol.16 (10), p.1205-1216

2002

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Nitric Oxide Increases Arterial Endotheial Permeability through Mediating VE-Cadherin Expression during Arteriogenesis

Fibrosis in endstage human heart failure: Severe changes in collagen metabolism and MMP/TIMP profiles

Myocytes Die by Multiple Mechanisms in Failing Human Hearts

Increased Expression of Cytoskeletal, Linkage, and Extracellular Proteins in Failing Human Myocardium

Human hibernating myocardium is jeopardized by apoptotic and autophagic cell death

Unresolved Issues Regarding the Role of Apoptosis in the Pathogenesis of Ischemic Injury and Heart Failure

Apoptosis is Initiated by Myocardial Ischemia and Executed During Reperfusion

Activation of the cardiac renin-angiotensin system and increased myocardial collagen expression in human aortic valve disease

The role of Fas/APO 1 and apoptosis in the development of human atherosclerotic lesions

The cytoskeleton and related proteins in the human failing heart

The role of apoptosis in myocardial ischemia: a critical appraisal

Translocation and Cleavage of Myocardial Dystrophin as a Common Pathway to Advanced Heart Failure: A Scheme for the Progression of Cardiac Dysfunction

The extracellular matrix in normal and diseased myocardium

Ischemia induces early changes to cytoskeletal and contractile proteins in diseased human myocardium

Matrix metalloproteinases and their tissue inhibitors in pressure-overloaded human myocardium during heart failure progression

Reduced sarcoplasmic reticulum Ca2+ -ATPase activity and dephosphorylated phospholamban contribute to contractile dysfunction in human hibernating myocardium

Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: Structural deterioration and compensatory mechanisms

The Cardiac Mechanical Stretch Sensor Machinery Involves a Z Disc Complex that Is Defective in a Subset of Human Dilated Cardiomyopathy

Structural correlate of atrial fibrillation in human patients

Connexin 43 expression and distribution in compensated and decompensated cardiac hypertrophy in patients with aortic stenosis

Apoptosis: measurement and technical issues

The role of the cytoskeleton in heart failure

Altered Expression of Titin and Contractile Proteins in Failing Human Myocardium

The extracellular matrix in the failing human heart

The carboxyl-terminal region of ahnak provides a link between cardiac L-type Ca2+ channels and the actin-based cytoskeleton

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