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Hematologically Important Mutations: The Autosomal Recessive Forms of Chronic Granulomatous Disease (First Update)

Blood cells, molecules, & diseases, 2000-10, Vol.26 (5), p.561-565

2000

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Monoclonal antibody CL5 recognizes the amino terminal domain of human phagocyte flavocytochrome b558 large subunit, gp91phox

European journal of haematology, 2005-04, Vol.74 (4), p.337-347

2005

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Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes

Blood, 2001, Vol.97 (1), p.305-311

2001

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Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease (Second Update)

Blood cells, molecules, & diseases, 2001, Vol.27 (1), p.16-26

2001

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Unusual polyclonal anti-gp91 peptide antibody interactions with X-linked chronic granulomatous disease-derived human neutrophils are not from compensatory expression of Nox proteins 1, 3, or 4

European journal of haematology, 2005-03, Vol.74 (3), p.241-249

2005

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Identification of a novel NCF-1 (p47-phox) pseudogene not containing the signature GT deletion: significance for A47 degrees chronic granulomatous disease carrier detection

Blood, 2002-09, Vol.100 (5), p.1845

2002

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Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease

Blood cells, molecules, & diseases, 1996, Vol.22 (1), p.90-95

1996

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Protein Phosphorylation in Neutrophils From Patients With p67-phox-Deficient Chronic Granulomatous Disease

Blood, 1996-05, Vol.87 (10), p.4404-4410

1996

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Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease—An Update

Blood cells, molecules, & diseases, 1997-12, Vol.23 (3), p.443-450

1997

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Hematologically Important Mutations: The Autosomal Recessive Forms of Chronic Granulomatous Disease

Blood cells, molecules, & diseases, 1996, Vol.22 (3), p.268-270

1996

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Hematologically Important Mutations: The Autosomal Recessive Forms of Chronic Granulomatous Disease (First Update)

Monoclonal antibody CL5 recognizes the amino terminal domain of human phagocyte flavocytochrome b558 large subunit, gp91phox

Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes

Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease (Second Update)

Unusual polyclonal anti-gp91 peptide antibody interactions with X-linked chronic granulomatous disease-derived human neutrophils are not from compensatory expression of Nox proteins 1, 3, or 4

Identification of a novel NCF-1 (p47-phox) pseudogene not containing the signature GT deletion: significance for A47 degrees chronic granulomatous disease carrier detection

Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease

Protein Phosphorylation in Neutrophils From Patients With p67-phox-Deficient Chronic Granulomatous Disease

Hematologically Important Mutations: X-Linked Chronic Granulomatous Disease—An Update

Hematologically Important Mutations: The Autosomal Recessive Forms of Chronic Granulomatous Disease

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