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DOT1L bridges transcription and heterochromatin formation at mammalian pericentromeres

EMBO reports, 2023-08, Vol.24 (8), p.e56492-n/a

2023

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Combinatorial targeting of a chromatin complex comprising Dot1L, menin and the tyrosine kinase BAZ1B reveals a new therapeutic vulnerability of endocrine therapy-resistant breast cancer

Breast cancer research : BCR, 2022-07, Vol.24 (1), p.1-52, Article 52

2022

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Histone Methyltransferases as a New Target for Epigenetic Action of Vorinostat

Biochemistry (Moscow), 2023-07, Vol.88 (7), p.968-978

2023

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Targeting epigenetic regulators for cancer therapy

Annals of the New York Academy of Sciences, 2014-02, Vol.1309 (1), p.30-36

2014

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Targeting the histone H3 lysine 79 methyltransferase DOT1L in MLL-rearranged leukemias

Journal of hematology and oncology, 2022-03, Vol.15 (1), p.35-35, Article 35

2022

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Abrogation of MLL–AF10 and CALM–AF10-mediated transformation through genetic inactivation or pharmacological inhibition of the H3K79 methyltransferase Dot1l

Leukemia, 2013-04, Vol.27 (4), p.813-822

2013

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Synthesis and Biological Activity of a Cytostatic Inhibitor of MLLr Leukemia Targeting the DOT1L Protein

Molecules (Basel, Switzerland), 2021-08, Vol.26 (17), p.5300

2021

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The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia

Journal of hematology and oncology, 2020-06, Vol.13 (1), p.78-78, Article 78

2020

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Methyltransferase Dot1l preferentially promotes innate IL-6 and IFN-β production by mediating H3K79me2/3 methylation in macrophages

Cellular & molecular immunology, 2020-01, Vol.17 (1), p.76-84

2020

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Combinatorial targeting of menin and the histone methyltransferase DOT1L as a novel therapeutic strategy for treatment of chemotherapy-resistant ovarian cancer

Cancer cell international, 2022-11, Vol.22 (1), p.1-336, Article 336

2022

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Inhibition of Methyltransferase DOT1L Sensitizes to Sorafenib Treatment AML Cells Irrespective of MLL-Rearrangements: A Novel Therapeutic Strategy for Pediatric AML

Cancers, 2020-07, Vol.12 (7), p.1972

2020

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The Histone Methyltransferase DOT1L Is a Functional Component of Estrogen Receptor Alpha Signaling in Ovarian Cancer Cells

Cancers, 2019-11, Vol.11 (11), p.1720

2019

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Folate deficiency reduced aberrant level of DOT1L-mediated histone H3K79 methylation causes disruptive SHH gene expression involved in neural tube defects

Epigenetics & chromatin, 2023-12, Vol.16 (1), p.50-50, Article 50

2023

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Single-cell transcriptomics defines Dot1L interacting partners and downstream target genes in the mouse molar dental pulp

The International journal of developmental biology, 2022, Vol.66 (7-8-9), p.391-400

2022

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The role of histone methylation in renal cell cancer: an update

Molecular biology reports, 2023-03, Vol.50 (3), p.2735-2742

2023

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DOT1L Methyltransferase Regulates Calcium Influx in Erythroid Progenitor Cells in Response to Erythropoietin

International journal of molecular sciences, 2022-05, Vol.23 (9), p.5137

2022

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Inhibition of the cardiac fibroblast-enriched histone methyltransferase Dot1L prevents cardiac fibrosis and cardiac dysfunction

Cell & bioscience, 2022-08, Vol.12 (1), p.1-134, Article 134

2022

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Mitotic accumulation of dimethylated lysine 79 of histone H3 is important for maintaining genome integrity during mitosis in human cells

Genetics (Austin), 2015-02, Vol.199 (2), p.423-433

2015

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Silencing or inhibition of H3K79 methyltransferase DOT1L induces cell cycle arrest by epigenetically modulating c-Myc expression in colorectal cancer

Clinical epigenetics, 2019-12, Vol.11 (1), p.199, Article 199

2019

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Functional diversity of inhibitors tackling the differentiation blockage of MLL-rearranged leukemia

Journal of hematology and oncology, 2019-06, Vol.12 (1), p.66-66, Article 66

2019

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Prognostic and therapeutic value of disruptor of telomeric silencing-1-like (DOT1L) expression in patients with ovarian cancer

Journal of hematology and oncology, 2017-01, Vol.10 (1), p.29-29, Article 29

2017

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Bromo-deaza-SAH: A potent and selective DOT1L inhibitor

Bioorganic & medicinal chemistry, 2013-04, Vol.21 (7), p.1787-1794

2013

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Bioinformatic Analyses of Broad H3K79me2 Domains in Different Leukemia Cell Line Data Sets

Cells (Basel, Switzerland), 2022-09, Vol.11 (18), p.2830

2022

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Differentiation and localization of interneurons in the developing spinal cord depends on DOT1L expression

Molecular brain, 2020-05, Vol.13 (1), p.85-85, Article 85

2020

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AF10 (MLLT10) prevents somatic cell reprogramming through regulation of DOT1L-mediated H3K79 methylation

Epigenetics & chromatin, 2021-07, Vol.14 (1), p.32-32, Article 32

2021

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DOT1L bridges transcription and heterochromatin formation at mammalian pericentromeres

Combinatorial targeting of a chromatin complex comprising Dot1L, menin and the tyrosine kinase BAZ1B reveals a new therapeutic vulnerability of endocrine therapy-resistant breast cancer

Histone Methyltransferases as a New Target for Epigenetic Action of Vorinostat

Targeting epigenetic regulators for cancer therapy

Targeting the histone H3 lysine 79 methyltransferase DOT1L in MLL-rearranged leukemias

Abrogation of MLL–AF10 and CALM–AF10-mediated transformation through genetic inactivation or pharmacological inhibition of the H3K79 methyltransferase Dot1l

Synthesis and Biological Activity of a Cytostatic Inhibitor of MLLr Leukemia Targeting the DOT1L Protein

The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia

Methyltransferase Dot1l preferentially promotes innate IL-6 and IFN-β production by mediating H3K79me2/3 methylation in macrophages

Combinatorial targeting of menin and the histone methyltransferase DOT1L as a novel therapeutic strategy for treatment of chemotherapy-resistant ovarian cancer

Inhibition of Methyltransferase DOT1L Sensitizes to Sorafenib Treatment AML Cells Irrespective of MLL-Rearrangements: A Novel Therapeutic Strategy for Pediatric AML

The Histone Methyltransferase DOT1L Is a Functional Component of Estrogen Receptor Alpha Signaling in Ovarian Cancer Cells

Folate deficiency reduced aberrant level of DOT1L-mediated histone H3K79 methylation causes disruptive SHH gene expression involved in neural tube defects

Single-cell transcriptomics defines Dot1L interacting partners and downstream target genes in the mouse molar dental pulp

The role of histone methylation in renal cell cancer: an update

DOT1L Methyltransferase Regulates Calcium Influx in Erythroid Progenitor Cells in Response to Erythropoietin

Inhibition of the cardiac fibroblast-enriched histone methyltransferase Dot1L prevents cardiac fibrosis and cardiac dysfunction

Mitotic accumulation of dimethylated lysine 79 of histone H3 is important for maintaining genome integrity during mitosis in human cells

Silencing or inhibition of H3K79 methyltransferase DOT1L induces cell cycle arrest by epigenetically modulating c-Myc expression in colorectal cancer

Functional diversity of inhibitors tackling the differentiation blockage of MLL-rearranged leukemia

Prognostic and therapeutic value of disruptor of telomeric silencing-1-like (DOT1L) expression in patients with ovarian cancer

Bromo-deaza-SAH: A potent and selective DOT1L inhibitor

Bioinformatic Analyses of Broad H3K79me2 Domains in Different Leukemia Cell Line Data Sets

Differentiation and localization of interneurons in the developing spinal cord depends on DOT1L expression

AF10 (MLLT10) prevents somatic cell reprogramming through regulation of DOT1L-mediated H3K79 methylation

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