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543 Support for Wound Healing As the Mechanism for Columnar Metaplasia of the Esophagus in a Rodent Model of Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-109-S-109

2015

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NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Cellular and molecular gastroenterology and hepatology, 2016-07, Vol.2 (4), p.439-453

2016

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Autocrine VEGF Signaling Promotes Proliferation of Neoplastic Barrett's Epithelial Cells Through a PLC-Dependent Pathway

Gastroenterology (New York, N.Y. 1943), 2014-02, Vol.146 (2), p.461-472.e6

2014

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Sa1885 ΔNp63 Upregulates Expression of Squamous Cytokeratins and Increases Proliferation in Squamous Cells, but Decreases Proliferation in Barrett's Cells: A Potential Role for Downregulation of ΔNp63 in Barrett's Pathogenesis

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-346-S-346

2015

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Sa1884 BMP4 Induces SOX9 and Columnar Cytokeratin Expression in Esophageal Squamous Cells and Increases Proliferation in Barrett's Cells Through the p38MAPK Pathway: Potential Roles for BMP4-p38MAPK Signaling in Barrett's Pathogenesis and Carcinogenesis

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-346-S-346

2015

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Tu1181 The Induction of Acute Reflux Esophagitis in Humans Causes Hypocontractile Esophageal Motility Alterations

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-813-S-814

2015

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Sa1842 In Esophageal Epithelial Cells From Barrett's Patients, Protein Kinase a Phosphorylates SOX9, Enhancing Its Binding to Target Gene Promoters: A Potential Role for SOX9 in Barrett's Pathogenesis

Gastroenterology (New York, N.Y. 1943), 2014-05, Vol.146 (5), p.S-309-S-310

2014

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Columnar-Lined Esophagus Develops via Wound Repair in a Surgical Model of Reflux Esophagitis

Cellular and molecular gastroenterology and hepatology, 2018-01, Vol.6 (4), p.389-404

2018

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202 Omeprazole and Fluticasone Inhibit IL-13-Stimulated Eotaxin-3 Expression by Esophageal Epithelial Cells Through Different Mechanisms and With Additive Effects: Rationale for Combining PPIs With Topical Steroids for EoE Patients

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-51-S-51

2015

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203 A STAT6 Inhibitor (AS1517499) and a Tyrosine Kinase Inhibitor (Leflunomide) Block IL-13/STAT6 Signaling and Eotaxin-3 Expression in Esophageal Fibroblasts and Epithelial Cells: Promising Agents to Improve Inflammation and Prevent Fibrostenosis in EoE

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-51-S-52

2015

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544 In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Activate VEGF Signaling to Induce Epithelial Mesenchymal Transition Through ZEB2, a Transcriptional Repressor of E-Cadherin

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-109-S-109

2015

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500 The Induction of Pathological Acid Reflux With Acute Reflux Esophagitis in Humans Is Associated With T Lymphocyte-Predominant Inflammation of the Esophageal Mucosa: A New Paradigm for the Pathogenesis of Reflux Esophagitis

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-97-S-97

2015

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860 Development and Characterization of a Surgical, Mouse Model of Reflux Esophagitis and Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-1064-S-1064

2013

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Tu2065 In Rats After Esophagojejunostomy, Reflux Esophagitis is Accompanied by the Expression of SOX-9 in Basal Cells of the Squamous Epithelium and in Barrett's Metaplasia

Gastroenterology (New York, N.Y. 1943), 2012-05, Vol.142 (5), p.S-1108-S-1109

2012

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387 Tumor Cells in Human Esophageal Adenocarcinomas Exhibit Active VEGF/VEGFR2 Signaling and, in Mice Bearing Xenografts of Transformed Barrett's Cells, Sunitinib (A VEGFR2 Inhibitor) Blocks Tumor Growth in an Angiogenesis-Independent Fashion

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-74-S-74

2013

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990 Nitric Oxide Profoundly Decreases Sox-2 Expression and AKT Signaling in Esophageal Squamous Cells: A Potential Mechanism Underlying Barrett's Metaplasia

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-182-S-182

2013

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M1619 Evolving Management Trends in the Treatment of Gallbladder Disease

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-868-S-868

2010

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975 Laparoscopic Assisted Total Gastrectomy With D2 Dissection and Roux-en-Y J Pouch

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-863-S-863

2010

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T1567 Laparoscopic Ischemic Pre-Conditioning of Gastric Conduit: Reducing Anastomotic Complications of Minimally Invasive Esophagectomy

Gastroenterology (New York, N.Y. 1943), 2009, Vol.136 (5), p.A-913-A-913

2009

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W1951 Increased Gastric Conduit Ischemia as Measured by Optical Fiber Spectroscopy During Minimally Invasive Esophagectomy is Associated With Development of Anastomotic Complications

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-906-S-906

2010

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T2079 Pneumoperitoneum in NOTES: Examining the Physiologic Effects of Sequential Endoluminal and Intraperitoneal Insufflation

Gastroenterology (New York, N.Y. 1943), 2009, Vol.136 (5), p.A-924-A-924

2009

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455 Impact of Institutional Case Volume On Inpatient Morbidity and Mortality After Paraesophageal Hernia Repair

Gastroenterology (New York, N.Y. 1943), 2009, Vol.136 (5), p.A-872-A-872

2009

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GATA4 Regulates Development of the Squamocolumnar Junction in the GI Tract: Implications for Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S87-S87

2017

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In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S29-S30

2017

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543 Support for Wound Healing As the Mechanism for Columnar Metaplasia of the Esophagus in a Rodent Model of Barrett's Esophagus

NOD-Like Receptor Protein 3 Inflammasome Priming and Activation in Barrett’s Epithelial Cells

Autocrine VEGF Signaling Promotes Proliferation of Neoplastic Barrett's Epithelial Cells Through a PLC-Dependent Pathway

Sa1885 ΔNp63 Upregulates Expression of Squamous Cytokeratins and Increases Proliferation in Squamous Cells, but Decreases Proliferation in Barrett's Cells: A Potential Role for Downregulation of ΔNp63 in Barrett's Pathogenesis

Sa1884 BMP4 Induces SOX9 and Columnar Cytokeratin Expression in Esophageal Squamous Cells and Increases Proliferation in Barrett's Cells Through the p38MAPK Pathway: Potential Roles for BMP4-p38MAPK Signaling in Barrett's Pathogenesis and Carcinogenesis

Tu1181 The Induction of Acute Reflux Esophagitis in Humans Causes Hypocontractile Esophageal Motility Alterations

Sa1842 In Esophageal Epithelial Cells From Barrett's Patients, Protein Kinase a Phosphorylates SOX9, Enhancing Its Binding to Target Gene Promoters: A Potential Role for SOX9 in Barrett's Pathogenesis

Columnar-Lined Esophagus Develops via Wound Repair in a Surgical Model of Reflux Esophagitis

202 Omeprazole and Fluticasone Inhibit IL-13-Stimulated Eotaxin-3 Expression by Esophageal Epithelial Cells Through Different Mechanisms and With Additive Effects: Rationale for Combining PPIs With Topical Steroids for EoE Patients

203 A STAT6 Inhibitor (AS1517499) and a Tyrosine Kinase Inhibitor (Leflunomide) Block IL-13/STAT6 Signaling and Eotaxin-3 Expression in Esophageal Fibroblasts and Epithelial Cells: Promising Agents to Improve Inflammation and Prevent Fibrostenosis in EoE

544 In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Activate VEGF Signaling to Induce Epithelial Mesenchymal Transition Through ZEB2, a Transcriptional Repressor of E-Cadherin

500 The Induction of Pathological Acid Reflux With Acute Reflux Esophagitis in Humans Is Associated With T Lymphocyte-Predominant Inflammation of the Esophageal Mucosa: A New Paradigm for the Pathogenesis of Reflux Esophagitis

860 Development and Characterization of a Surgical, Mouse Model of Reflux Esophagitis and Barrett's Esophagus

Tu2065 In Rats After Esophagojejunostomy, Reflux Esophagitis is Accompanied by the Expression of SOX-9 in Basal Cells of the Squamous Epithelium and in Barrett's Metaplasia

387 Tumor Cells in Human Esophageal Adenocarcinomas Exhibit Active VEGF/VEGFR2 Signaling and, in Mice Bearing Xenografts of Transformed Barrett's Cells, Sunitinib (A VEGFR2 Inhibitor) Blocks Tumor Growth in an Angiogenesis-Independent Fashion

990 Nitric Oxide Profoundly Decreases Sox-2 Expression and AKT Signaling in Esophageal Squamous Cells: A Potential Mechanism Underlying Barrett's Metaplasia

M1619 Evolving Management Trends in the Treatment of Gallbladder Disease

975 Laparoscopic Assisted Total Gastrectomy With D2 Dissection and Roux-en-Y J Pouch

T1567 Laparoscopic Ischemic Pre-Conditioning of Gastric Conduit: Reducing Anastomotic Complications of Minimally Invasive Esophagectomy

W1951 Increased Gastric Conduit Ischemia as Measured by Optical Fiber Spectroscopy During Minimally Invasive Esophagectomy is Associated With Development of Anastomotic Complications

T2079 Pneumoperitoneum in NOTES: Examining the Physiologic Effects of Sequential Endoluminal and Intraperitoneal Insufflation

455 Impact of Institutional Case Volume On Inpatient Morbidity and Mortality After Paraesophageal Hernia Repair

A Summary of the 2016 James W. Freston Conference of the American Gastroenterological Association: Intestinal Metaplasia in the Esophagus and Stomach: Origins, Differences, Similarities and Significance

GATA4 Regulates Development of the Squamocolumnar Junction in the GI Tract: Implications for Barrett's Esophagus

In Esophageal Fibroblasts, TH2 Cytokines Induce Pro-Fibrotic Periostin Expression that can be Blocked by JAK-STAT6 Inhibitors

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