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Genetics of Inflammatory Bowel Diseases

Gastroenterology (New York, N.Y. 1943), 2015-10, Vol.149 (5), p.1163-1176.e2

2015

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Induction and Maintenance Infliximab Therapy for the Treatment of Moderate-to-Severe Crohn’s Disease in Children

Gastroenterology (New York, N.Y. 1943), 2007-03, Vol.132 (3), p.863-873

2007

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Increased Immune Reactivity Predicts Aggressive Complicating Crohn's Disease in Children

Clinical gastroenterology and hepatology, 2008-10, Vol.6 (10), p.1105-1111

2008

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Mucosal Expression of Type 2 and Type 17 Immune Response Genes Distinguishes Ulcerative Colitis From Colon-Only Crohn’s Disease in Treatment-Naive Pediatric Patients

Gastroenterology (New York, N.Y. 1943), 2017-05, Vol.152 (6), p.1345-1357.e7

2017

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Severe Pediatric Ulcerative Colitis: A Prospective Multicenter Study of Outcomes and Predictors of Response

Gastroenterology (New York, N.Y. 1943), 2010-06, Vol.138 (7), p.2282-2291

2010

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Searching for New Clues in Inflammatory Bowel Disease: Tell Tales From Pediatric IBD Natural History Studies

Gastroenterology (New York, N.Y. 1943), 2008-10, Vol.135 (4), p.1038-1041

2008

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Infliximab Is Not Associated With Increased Risk of Malignancy or Hemophagocytic Lymphohistiocytosis in Pediatric Patients With Inflammatory Bowel Disease

Gastroenterology (New York, N.Y. 1943), 2017-06, Vol.152 (8), p.1901-1914.e3

2017

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The Age of Gene Discovery in Very Early Onset Inflammatory Bowel Disease

Gastroenterology (New York, N.Y. 1943), 2012-08, Vol.143 (2), p.285-288

2012

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Pediatric Inflammatory Bowel Disease: Highlighting Pediatric Differences in IBD

Gastroenterology clinics of North America, 2009-12, Vol.38 (4), p.611-628

2009

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Induction and Maintenance Therapy With Infliximab for Children With Moderate to Severe Ulcerative Colitis

Clinical gastroenterology and hepatology, 2012-04, Vol.10 (4), p.391-399.e1

2012

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Increased Effectiveness of Early Therapy With Anti-Tumor Necrosis Factor-α vs an Immunomodulator in Children With Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2014-02, Vol.146 (2), p.383-391

2014

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Su1860 Cytomegalovirus Infection Enhances Susceptibility to Crohn's Disease in Individuals With KIR and HLA Pairs Enabling NK Cell Licensing

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S572-S573

2016

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A Pleiotropic Missense Variant in SLC39A8 Is Associated With Crohn’s Disease and Human Gut Microbiome Composition

Gastroenterology (New York, N.Y. 1943), 2016-10, Vol.151 (4), p.724-732

2016

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Reply

Gastroenterology (New York, N.Y. 1943), 2017

2017

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Characterization of Genetic Loci That Affect Susceptibility to Inflammatory Bowel Diseases in African Americans

Gastroenterology (New York, N.Y. 1943), 2015-11, Vol.149 (6), p.1575-1586

2015

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A Frameshift in CSF2RB Predominant Among Ashkenazi Jews Increases Risk for Crohn's Disease and Reduces Monocyte Signaling via GM-CSF

Gastroenterology (New York, N.Y. 1943), 2016-10, Vol.151 (4), p.710-723.e2

2016

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Oral and Fecal Microbiome Characteristics in Treatment Naïve Pediatric IBD Patients Over Time: a Prospective Longitudinal Study

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S961-S961

2017

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Su1100 Determinants of Immunogenicity in Children With Inflammatory Bowel Disease Receiving Infliximab

Gastroenterology (New York, N.Y. 1943), 2014-05, Vol.146 (5), p.S-374-S-374

2014

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Procollagen III N-Terminal Propeptide (PIIINP) as a Circulating Biomarker for Active Stricture Development in Pediatric Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S956-S956

2017

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Disruption of Epigenetic Pathways in Regulatory T Cells by FOXP3 Mutations and Pro-Inflammatory Signals During Inflammatory Bowel Disease

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S981-S982

2017

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Improvement in Biomarkers of Bone Formation During Infliximab Therapy in Pediatric Crohn's Disease: Results of the REACH Study

Clinical gastroenterology and hepatology, 2008-12, Vol.6 (12), p.1378-1384

2008

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Disruption of FOXP3–EZH2 Interaction Represents a Pathobiological Mechanism in Intestinal Inflammation

Cellular and molecular gastroenterology and hepatology, 2019-01, Vol.7 (1), p.55-71

2019

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Factors That Determine Risk for Surgery in Pediatric Patients With Crohn's Disease

Clinical gastroenterology and hepatology, 2010-09, Vol.8 (9), p.789-794.e2

2010

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Bowel Location Rather Than Disease Subtype Dominates Transcriptomic Heterogeneity in Pediatric IBD

Cellular and molecular gastroenterology and hepatology, 2018-01, Vol.6 (4), p.474-476.e3

2018

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629 Risk of Malignancy in Pediatric Inflammatory Bowel Disease: Results From the DEVELOP Registry

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S131-S131

2016

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Genetics of Inflammatory Bowel Diseases

Induction and Maintenance Infliximab Therapy for the Treatment of Moderate-to-Severe Crohn’s Disease in Children

Increased Immune Reactivity Predicts Aggressive Complicating Crohn's Disease in Children

Mucosal Expression of Type 2 and Type 17 Immune Response Genes Distinguishes Ulcerative Colitis From Colon-Only Crohn’s Disease in Treatment-Naive Pediatric Patients

Severe Pediatric Ulcerative Colitis: A Prospective Multicenter Study of Outcomes and Predictors of Response

Searching for New Clues in Inflammatory Bowel Disease: Tell Tales From Pediatric IBD Natural History Studies

Infliximab Is Not Associated With Increased Risk of Malignancy or Hemophagocytic Lymphohistiocytosis in Pediatric Patients With Inflammatory Bowel Disease

The Age of Gene Discovery in Very Early Onset Inflammatory Bowel Disease

Pediatric Inflammatory Bowel Disease: Highlighting Pediatric Differences in IBD

Induction and Maintenance Therapy With Infliximab for Children With Moderate to Severe Ulcerative Colitis

Increased Effectiveness of Early Therapy With Anti-Tumor Necrosis Factor-α vs an Immunomodulator in Children With Crohn's Disease

Su1860 Cytomegalovirus Infection Enhances Susceptibility to Crohn's Disease in Individuals With KIR and HLA Pairs Enabling NK Cell Licensing

A Pleiotropic Missense Variant in SLC39A8 Is Associated With Crohn’s Disease and Human Gut Microbiome Composition

Reply

Characterization of Genetic Loci That Affect Susceptibility to Inflammatory Bowel Diseases in African Americans

A Frameshift in CSF2RB Predominant Among Ashkenazi Jews Increases Risk for Crohn's Disease and Reduces Monocyte Signaling via GM-CSF

Oral and Fecal Microbiome Characteristics in Treatment Naïve Pediatric IBD Patients Over Time: a Prospective Longitudinal Study

Su1100 Determinants of Immunogenicity in Children With Inflammatory Bowel Disease Receiving Infliximab

Procollagen III N-Terminal Propeptide (PIIINP) as a Circulating Biomarker for Active Stricture Development in Pediatric Crohn's Disease

Disruption of Epigenetic Pathways in Regulatory T Cells by FOXP3 Mutations and Pro-Inflammatory Signals During Inflammatory Bowel Disease

Improvement in Biomarkers of Bone Formation During Infliximab Therapy in Pediatric Crohn's Disease: Results of the REACH Study

Disruption of FOXP3–EZH2 Interaction Represents a Pathobiological Mechanism in Intestinal Inflammation

Factors That Determine Risk for Surgery in Pediatric Patients With Crohn's Disease

Bowel Location Rather Than Disease Subtype Dominates Transcriptomic Heterogeneity in Pediatric IBD

629 Risk of Malignancy in Pediatric Inflammatory Bowel Disease: Results From the DEVELOP Registry

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