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Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation

mBio, 2019-03, Vol.10 (2)

2019

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An Interaction between Human Papillomavirus 16 E2 and TopBP1 Is Required for Optimum Viral DNA Replication and Episomal Genome Establishment

Journal of Virology, 2012-12, Vol.86 (23), p.12806-12815

2012

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Levels of the E2 interacting protein TopBP1 modulate papillomavirus maintenance stage replication

Virology (New York, N.Y.), 2015-04, Vol.478, p.129-136

2015

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The Deacetylase SIRT1 Regulates the Replication Properties of Human Papillomavirus 16 E1 and E2

Journal of virology, 2017-05, Vol.91 (10)

2017

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Adenovirus 12 E4orf6 inhibits ATR activation by promoting TOPBP1 degradation

Proceedings of the National Academy of Sciences - PNAS, 2010-07, Vol.107 (27), p.12251-12256

2010

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TopBP1 Regulates Human Papillomavirus Type 16 E2 Interaction with Chromatin

Journal of Virology, 2007-04, Vol.81 (8), p.4338-4342

2007

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DNA Damage Reduces the Quality, but Not the Quantity of Human Papillomavirus 16 E1 and E2 DNA Replication

Viruses, 2016-06, Vol.8 (6), p.175

2016

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Human Papillomavirus 16 E2 Interaction with TopBP1 Is Required for E2 and Viral Genome Stability during the Viral Life Cycle

Journal of virology, 2023-03, Vol.97 (3), p.e0006323-e0006323

2023

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Human papillomavirus 16 E2 blocks cellular senescence in response to activation of the DNA damage response

Virology (New York, N.Y.), 2022-10, Vol.575, p.54-62

2022

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Direct interaction with the BRD4 carboxyl-terminal motif (CTM) and TopBP1 is required for human papillomavirus 16 E2 association with mitotic chromatin and plasmid segregation function

Journal of virology, 2023-10, Vol.97 (10), p.e0078223

2023

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Evidence supporting a role for TopBP1 and Brd4 in the initiation but not continuation of human papillomavirus 16 E1/E2-mediated DNA replication

Journal of virology, 2015-05, Vol.89 (9), p.4980-4991

2015

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Human Papillomavirus 16 E2 Regulates Keratinocyte Gene Expression Relevant to Cancer and the Viral Life Cycle

Journal of virology, 2019-02, Vol.93 (4)

2019

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A new strategy for the in vitro selection of stapled peptide inhibitors by mRNA display

Chemical communications (Cambridge, England), 2019-08, Vol.55 (61), p.8959-8962

2019

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P53 represses human papillomavirus type 16 DNA replication via the viral E2 protein

Virology journal, 2008-01, Vol.5 (1), p.5-5, Article 5

2008

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Failure to interact with Brd4 alters the ability of HPV16 E2 to regulate host genome expression and cellular movement

Virus research, 2016-01, Vol.211, p.1-8

2016

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Calcein represses human papillomavirus 16 E1-E2 mediated DNA replication via blocking their binding to the viral origin of replication

Virology (New York, N.Y.), 2017-08, Vol.508, p.180-187

2017

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Regulation of human genome expression and RNA splicing by human papillomavirus 16 E2 protein

Virology (New York, N.Y.), 2014-11, Vol.468, p.10-18

2014

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Human papillomavirus 16 E2 stability and transcriptional activation is enhanced by E1 via a direct protein–protein interaction

Virology (New York, N.Y.), 2011-05, Vol.414 (1), p.26-33

2011

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The human papillomavirus 16 E2 protein is stabilised in S phase

Virology (New York, N.Y.), 2009-11, Vol.394 (2), p.194-199

2009

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Small interfering RNA targeting bovine papillomavirus type 1 E2 induces apoptosis in equine sarcoid transformed fibroblasts

Virus research, 2009-10, Vol.145 (1), p.162-165

2009

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An Enhanced Epithelial Response of a Papillomavirus Promoter to Transcriptional Activators

The Journal of biological chemistry, 1999-09, Vol.274 (39), p.27839-27844

1999

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A Novel Silencer Element in the Bovine Papillomavirus Type 4 Promoter Represses the Transcriptional Response to Papillomavirus E2 Protein

Journal of Virology, 2001-03, Vol.75 (6), p.2829-2838

2001

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Immunohistochemical expression of TopBP1 in feline mammary neoplasia in relation to histological grade, Ki67, ERalpha and p53

The veterinary journal (1997), 2008-02, Vol.175 (2), p.218-226

2008

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Transformation by jun : requirement for leucine zipper, basic region and transactivation domain and enhancement by fos

Oncogene, 1992-06, Vol.7 (6), p.1119-1125

1992

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Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation

CK2 Phosphorylation of Human Papillomavirus 16 E2 on Serine 23 Promotes Interaction with TopBP1 and Is Critical for E2 Interaction with Mitotic Chromatin and the Viral Life Cycle

An Interaction between Human Papillomavirus 16 E2 and TopBP1 Is Required for Optimum Viral DNA Replication and Episomal Genome Establishment

Levels of the E2 interacting protein TopBP1 modulate papillomavirus maintenance stage replication

The Deacetylase SIRT1 Regulates the Replication Properties of Human Papillomavirus 16 E1 and E2

Adenovirus 12 E4orf6 inhibits ATR activation by promoting TOPBP1 degradation

TopBP1 Regulates Human Papillomavirus Type 16 E2 Interaction with Chromatin

DNA Damage Reduces the Quality, but Not the Quantity of Human Papillomavirus 16 E1 and E2 DNA Replication

Human Papillomavirus 16 E2 Interaction with TopBP1 Is Required for E2 and Viral Genome Stability during the Viral Life Cycle

Human papillomavirus 16 E2 blocks cellular senescence in response to activation of the DNA damage response

Direct interaction with the BRD4 carboxyl-terminal motif (CTM) and TopBP1 is required for human papillomavirus 16 E2 association with mitotic chromatin and plasmid segregation function

Evidence supporting a role for TopBP1 and Brd4 in the initiation but not continuation of human papillomavirus 16 E1/E2-mediated DNA replication

Human Papillomavirus 16 E2 Regulates Keratinocyte Gene Expression Relevant to Cancer and the Viral Life Cycle

A new strategy for the in vitro selection of stapled peptide inhibitors by mRNA display

P53 represses human papillomavirus type 16 DNA replication via the viral E2 protein

Failure to interact with Brd4 alters the ability of HPV16 E2 to regulate host genome expression and cellular movement

Calcein represses human papillomavirus 16 E1-E2 mediated DNA replication via blocking their binding to the viral origin of replication

Regulation of human genome expression and RNA splicing by human papillomavirus 16 E2 protein

Human papillomavirus 16 E2 stability and transcriptional activation is enhanced by E1 via a direct protein–protein interaction

The human papillomavirus 16 E2 protein is stabilised in S phase

Small interfering RNA targeting bovine papillomavirus type 1 E2 induces apoptosis in equine sarcoid transformed fibroblasts

An Enhanced Epithelial Response of a Papillomavirus Promoter to Transcriptional Activators

A Novel Silencer Element in the Bovine Papillomavirus Type 4 Promoter Represses the Transcriptional Response to Papillomavirus E2 Protein

Immunohistochemical expression of TopBP1 in feline mammary neoplasia in relation to histological grade, Ki67, ERalpha and p53

Transformation by jun : requirement for leucine zipper, basic region and transactivation domain and enhancement by fos

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