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Nuclear Positioning

Cell, 2013-03, Vol.152 (6), p.1376-1389

2013

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Accessorizing and anchoring the LINC complex for multifunctionality

The Journal of cell biology, 2015-01, Vol.208 (1), p.11-22

2015

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The Nuclear Envelope as a Signaling Node in Development and Disease

Developmental cell, 2009-11, Vol.17 (5), p.626-638

2009

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Prelamin A and ZMPSTE24 in premature and physiological aging

Nucleus (Austin, Tex.), 2023-12, Vol.14 (1), p.2270345-2270345

2023

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Temsirolimus activates autophagy and ameliorates cardiomyopathy caused by lamin A/C gene mutation

Science translational medicine, 2012-07, Vol.4 (144), p.144ra102

2012

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Emerin organizes actin flow for nuclear movement and centrosome orientation in migrating fibroblasts

Molecular biology of the cell, 2013-12, Vol.24 (24), p.3869-3880

2013

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Hepatocytes Deficient in Nuclear Envelope Protein Lamina-associated Polypeptide 1 are an Ideal Mammalian System to Study Intranuclear Lipid Droplets

Journal of lipid research, 2022-10, Vol.63 (10), p.100277-100277, Article 100277

2022

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TorsinA controls TAN line assembly and the retrograde flow of dorsal perinuclear actin cables during rearward nuclear movement

The Journal of cell biology, 2017-03, Vol.216 (3), p.657-674

2017

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Apoptosis-induced translocation of nesprin-2 from the nuclear envelope to mitochondria is associated with mitochondrial dysfunction

Nucleus (Austin, Tex.), 2024-12, Vol.15 (1), p.2413501

2024

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Lamina-Associated Polypeptide-1 Interacts with the Muscular Dystrophy Protein Emerin and Is Essential for Skeletal Muscle Maintenance

Developmental cell, 2013-09, Vol.26 (6), p.591-603

2013

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Reactivation of autophagy ameliorates LMNA cardiomyopathy

Autophagy, 2013-01, Vol.9 (1), p.110-111

2013

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Dynamics and molecular interactions of linker of nucleoskeleton and cytoskeleton (LINC) complex proteins

Journal of cell science, 2009-11, Vol.122 (22), p.4099-4108

2009

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Long live lamins

The Journal of cell biology, 2024-01, Vol.223 (1), p.1

2024

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Lamin A/C Depletion Enhances DNA Damage-Induced Stalled Replication Fork Arrest

Molecular and Cellular Biology, 2013-03, Vol.33 (6), p.1210-1222

2013

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LINC complex protein nesprin-2 has pro-apoptotic activity via Bcl-2 family proteins

Cell death discovery, 2024-01, Vol.10 (1), p.29-29, Article 29

2024

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The non-muscle ADF/cofilin-1 controls sarcomeric actin filament integrity and force production in striated muscle laminopathies

Cell reports (Cambridge), 2021-08, Vol.36 (8), p.109601-109601, Article 109601

2021

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LMNA cardiomyopathy: cell biology and genetics meet clinical medicine

Disease models & mechanisms, 2011-09, Vol.4 (5), p.562-568

2011

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Pharmacological inhibition of c-Jun N-terminal kinase signaling prevents cardiomyopathy caused by mutation in LMNA gene

Biochimica et biophysica acta, 2010-07, Vol.1802 (7), p.632-638

2010

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Nuclear membrane diversity: underlying tissue-specific pathologies in disease?

Current opinion in cell biology, 2015-06, Vol.34, p.101-112

2015

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Apoptotic stress induces Bax-dependent, caspase-independent redistribution of LINC complex nesprins

Cell death discovery, 2020-09, Vol.6 (1), p.90-90, Article 90

2020

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Diseases of the nuclear envelope

Cold Spring Harbor perspectives in biology, 2010-02, Vol.2 (2), p.a000760-a000760

2010

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Properties of lamin A mutants found in Emery-Dreifuss muscular dystrophy, cardiomyopathy and Dunnigan-type partial lipodystrophy

Journal of cell science, 2001-12, Vol.114 (Pt 24), p.4435-4445

2001

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The Ig-like Structure of the C-Terminal Domain of Lamin A/C, Mutated in Muscular Dystrophies, Cardiomyopathy, and Partial Lipodystrophy

Structure (London), 2002-06, Vol.10 (6), p.811-823

2002

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A lamin A/C variant causing striated muscle disease provides insights into filament organization

Journal of cell science, 2021-03, Vol.134 (6)

2021

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Inhibition of extracellular signal-regulated kinase 1/2 signaling has beneficial effects on skeletal muscle in a mouse model of Emery-Dreifuss muscular dystrophy caused by lamin A/C gene mutation

Skeletal muscle, 2013-07, Vol.3 (1), p.17-17

2013

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Nuclear Positioning

Accessorizing and anchoring the LINC complex for multifunctionality

The Nuclear Envelope as a Signaling Node in Development and Disease

Prelamin A and ZMPSTE24 in premature and physiological aging

Temsirolimus activates autophagy and ameliorates cardiomyopathy caused by lamin A/C gene mutation

Emerin organizes actin flow for nuclear movement and centrosome orientation in migrating fibroblasts

Hepatocytes Deficient in Nuclear Envelope Protein Lamina-associated Polypeptide 1 are an Ideal Mammalian System to Study Intranuclear Lipid Droplets

TorsinA controls TAN line assembly and the retrograde flow of dorsal perinuclear actin cables during rearward nuclear movement

Apoptosis-induced translocation of nesprin-2 from the nuclear envelope to mitochondria is associated with mitochondrial dysfunction

Lamina-Associated Polypeptide-1 Interacts with the Muscular Dystrophy Protein Emerin and Is Essential for Skeletal Muscle Maintenance

Reactivation of autophagy ameliorates LMNA cardiomyopathy

Dynamics and molecular interactions of linker of nucleoskeleton and cytoskeleton (LINC) complex proteins

Long live lamins

Lamin A/C Depletion Enhances DNA Damage-Induced Stalled Replication Fork Arrest

LINC complex protein nesprin-2 has pro-apoptotic activity via Bcl-2 family proteins

The non-muscle ADF/cofilin-1 controls sarcomeric actin filament integrity and force production in striated muscle laminopathies

LMNA cardiomyopathy: cell biology and genetics meet clinical medicine

Pharmacological inhibition of c-Jun N-terminal kinase signaling prevents cardiomyopathy caused by mutation in LMNA gene

Nuclear membrane diversity: underlying tissue-specific pathologies in disease?

Apoptotic stress induces Bax-dependent, caspase-independent redistribution of LINC complex nesprins

Diseases of the nuclear envelope

Properties of lamin A mutants found in Emery-Dreifuss muscular dystrophy, cardiomyopathy and Dunnigan-type partial lipodystrophy

The Ig-like Structure of the C-Terminal Domain of Lamin A/C, Mutated in Muscular Dystrophies, Cardiomyopathy, and Partial Lipodystrophy

A lamin A/C variant causing striated muscle disease provides insights into filament organization

Inhibition of extracellular signal-regulated kinase 1/2 signaling has beneficial effects on skeletal muscle in a mouse model of Emery-Dreifuss muscular dystrophy caused by lamin A/C gene mutation

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