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Central and peripheral contributions of T-type calcium channels in pain

Molecular brain, 2022-05, Vol.15 (1), p.39-39, Article 39

2022

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Targeting voltage-gated calcium channels in neurological and psychiatric diseases

Nature reviews. Drug discovery, 2016-01, Vol.15 (1), p.19-34

Zamponi, Gerald W.

2016

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Electrophysiological characterization of a CaV3.1 calcium channel mutation linked to trigeminal neuralgia

Pflügers Archiv, 2023-06, Vol.475 (6), p.711-718

2023

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A molecular complex of Cav1.2/CaMKK2/CaMK1a in caveolae is responsible for vascular remodeling via excitation–transcription coupling

Proceedings of the National Academy of Sciences - PNAS, 2022-04, Vol.119 (16), p.e2117435119-e2117435119

2022

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The terpenes camphene and alpha-bisabolol inhibit inflammatory and neuropathic pain via Cav3.2 T-type calcium channels

Molecular brain, 2021-11, Vol.14 (1), p.1-166, Article 166

2021

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Trafficking and stability of voltage-gated calcium channels

Cellular and molecular life sciences : CMLS, 2012-03, Vol.69 (6), p.843-856

2012

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A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain

Molecular pain, 2016, Vol.12

2016

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Low voltage activation of KCa1.1 current by Cav3-KCa1.1 complexes

PloS one, 2013-04, Vol.8 (4), p.e61844-e61844

2013

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Ethosuximide inhibits acute histamine- and chloroquine-induced scratching behavior in mice

Molecular brain, 2021-03, Vol.14 (1), p.46-5, Article 46

2021

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Analgesia by intrathecal delta-9-tetrahydrocannabinol is dependent on Cav3.2 calcium channels

Molecular brain, 2023-05, Vol.16 (1), p.47-47, Article 47

2023

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Genetic T-type calcium channelopathies

Journal of medical genetics, 2020-01, Vol.57 (1), p.1-10

2020

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Electrophysiological characterization of a Cav3.2 calcium channel missense variant associated with epilepsy and hearing loss

Molecular brain, 2023-09, Vol.16 (1), p.1-68, Article 68

2023

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Splice-variant specific effects of a CACNA1H mutation associated with writer's cramp

Molecular brain, 2021-09, Vol.14 (1), p.145-145, Article 145

2021

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A CACNA1A variant associated with trigeminal neuralgia alters the gating of Cav2.1 channels

Molecular brain, 2021-01, Vol.14 (1), p.4-4, Article 4

2021

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Small Organic Molecule Disruptors of Cav3.2 - USP5 Interactions Reverse Inflammatory and Neuropathic Pain

Molecular pain, 2015-03, Vol.11, p.12-12

2015

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Disrupting USP5/Cav3.2 interactions protects female mice from mechanical hypersensitivity during peripheral inflammation

Molecular brain, 2018-10, Vol.11 (1), p.60-60, Article 60

2018

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Regulation of N-type calcium channels by nociceptin receptors and its possible role in neurological disorders

Molecular brain, 2022-11, Vol.15 (1), p.95-13, Article 95

2022

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The de novo CACNA1A pathogenic variant Y1384C associated with hemiplegic migraine, early onset cerebellar atrophy and developmental delay leads to a loss of Cav2.1 channel function

Molecular brain, 2021-02, Vol.14 (1), p.27-27, Article 27

2021

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Block of voltage-gated calcium channels by peptide toxins

Neuropharmacology, 2017-12, Vol.127, p.109-115

2017

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SUMOylation regulates USP5-Cav3.2 calcium channel interactions

Molecular brain, 2019-08, Vol.12 (1), p.73-73, Article 73

2019

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Modeling temperature- and Cav3 subtype-dependent alterations in T-type calcium channel mediated burst firing

Molecular brain, 2021-07, Vol.14 (1), p.115-115, Article 115

2021

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Recent advances in the development of T‐type calcium channel blockers for pain intervention

British journal of pharmacology, 2018-06, Vol.175 (12), p.2375-2383

2018

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CaVβ-subunit dependence of forward and reverse trafficking of CaV1.2 calcium channels

Molecular brain, 2022-05, Vol.15 (1), p.1-43, Article 43

2022

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Identification of interleukin-1 beta as a key mediator in the upregulation of Cav3.2–USP5 interactions in the pain pathway

Molecular pain, 2017-01, Vol.13, p.1744806917724698-1744806917724698

2017

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The T-type calcium channelosome

Pflügers Archiv, 2024-02, Vol.476 (2), p.163-177

2024

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Central and peripheral contributions of T-type calcium channels in pain

Targeting voltage-gated calcium channels in neurological and psychiatric diseases

Electrophysiological characterization of a CaV3.1 calcium channel mutation linked to trigeminal neuralgia

A molecular complex of Cav1.2/CaMKK2/CaMK1a in caveolae is responsible for vascular remodeling via excitation–transcription coupling

The terpenes camphene and alpha-bisabolol inhibit inflammatory and neuropathic pain via Cav3.2 T-type calcium channels

Trafficking and stability of voltage-gated calcium channels

A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain

Low voltage activation of KCa1.1 current by Cav3-KCa1.1 complexes

Ethosuximide inhibits acute histamine- and chloroquine-induced scratching behavior in mice

Analgesia by intrathecal delta-9-tetrahydrocannabinol is dependent on Cav3.2 calcium channels

Genetic T-type calcium channelopathies

Electrophysiological characterization of a Cav3.2 calcium channel missense variant associated with epilepsy and hearing loss

Splice-variant specific effects of a CACNA1H mutation associated with writer's cramp

A CACNA1A variant associated with trigeminal neuralgia alters the gating of Cav2.1 channels

Small Organic Molecule Disruptors of Cav3.2 - USP5 Interactions Reverse Inflammatory and Neuropathic Pain

Disrupting USP5/Cav3.2 interactions protects female mice from mechanical hypersensitivity during peripheral inflammation

Regulation of N-type calcium channels by nociceptin receptors and its possible role in neurological disorders

The de novo CACNA1A pathogenic variant Y1384C associated with hemiplegic migraine, early onset cerebellar atrophy and developmental delay leads to a loss of Cav2.1 channel function

Block of voltage-gated calcium channels by peptide toxins

SUMOylation regulates USP5-Cav3.2 calcium channel interactions

Modeling temperature- and Cav3 subtype-dependent alterations in T-type calcium channel mediated burst firing

Recent advances in the development of T‐type calcium channel blockers for pain intervention

CaVβ-subunit dependence of forward and reverse trafficking of CaV1.2 calcium channels

Identification of interleukin-1 beta as a key mediator in the upregulation of Cav3.2–USP5 interactions in the pain pathway

The T-type calcium channelosome

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