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Heme peroxidase HPX-2 protects Caenorhabditis elegans from pathogens

PLoS genetics, 2019-01, Vol.15 (1), p.e1007944-e1007944

2019

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A heritable antiviral RNAi response limits Orsay virus infection in Caenorhabditis elegans N2

PloS one, 2014-02, Vol.9 (2), p.e89760-e89760

2014

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Caenorhabditis elegans: a simple nematode infection model for Penicillium marneffei

PloS one, 2014-09, Vol.9 (9), p.e108764

2014

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Using a health-rating system to evaluate the usefulness of Caenorhabditis elegans as a model for anthelmintic study

PloS one, 2017-06, Vol.12 (6), p.e0179376-e0179376

2017

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Cranberry extract standardized for proanthocyanidins promotes the immune response of Caenorhabditis elegans to Vibrio cholerae through the p38 MAPK pathway and HSF-1

PloS one, 2014-07, Vol.9 (7), p.e103290

2014

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The pore-forming protein Cry5B elicits the pathogenicity of Bacillus sp. against Caenorhabditis elegans

PloS one, 2011-12, Vol.6 (12), p.e29122

2011

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LEM-3 - A LEM domain containing nuclease involved in the DNA damage response in C. elegans

PloS one, 2012-02, Vol.7 (2), p.e24555

2012

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Depletion of the C. elegans NAC engages the unfolded protein response, resulting in increased chaperone expression and apoptosis

PloS one, 2012-09, Vol.7 (9), p.e44038-e44038

2012

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WWP-1 is a novel modulator of the DAF-2 insulin-like signaling network involved in pore-forming toxin cellular defenses in Caenorhabditis elegans

PloS one, 2010-03, Vol.5 (3), p.e9494-e9494

2010

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RNAi screen of DAF-16/FOXO target genes in C. elegans links pathogenesis and dauer formation

PloS one, 2010-12, Vol.5 (12), p.e15902-e15902

2010

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Programmed Cell Death Mediated by ced-3 and ced-4 Protects Caenorhabditis elegans from Salmonella typhimurium-Mediated Killing

Proceedings of the National Academy of Sciences - PNAS, 2001-02, Vol.98 (5), p.2735-2739

2001

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DAF-2/insulin-like signaling in C. elegans modifies effects of dietary restriction and nutrient stress on aging, stress and growth

PloS one, 2007-11, Vol.2 (11), p.e1240-e1240

2007

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Salmonella typhimurium proliferates and establishes a persistent infection in the intestine of Caenorhabditis elegans

Current biology, 2000-11, Vol.10 (23), p.1539-1542

2000

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Caenorhabditis elegans as a host for the study of host–pathogen interactions

Current Opinion in Microbiology, 2002-02, Vol.5 (1), p.97-101

2002

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Caenorhabditis elegans-Based Screen Identifies Salmonella Virulence Factors Required for Conserved Host-Pathogen Interactions

Current biology, 2004-06, Vol.14 (11), p.1018-1024

2004

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Cellular Responses to Infections in Caenorhabditis elegans

Encyclopedia of Cell Biology, 2015, p.845-852

2015

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Yersinia pestis kills Caenorhabditis elegans by a biofilm-independent process that involves novel virulence factors

EMBO reports, 2005-10, Vol.6 (10), p.992-997

2005

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Intestinal infection regulates behavior and learning via neuroendocrine signaling

eLife, 2019-11, Vol.8

2019

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Neural control of behavioral and molecular defenses in C. elegans

Current opinion in neurobiology, 2020-06, Vol.62, p.34-40

2020

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Role of the nervous system in the control of proteostasis during innate immune activation: insights from C. elegans

PLoS pathogens, 2013-08, Vol.9 (8), p.e1003433-e1003433

2013

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G-Protein-Coupled Receptor SRBC-48 Protects against Dendrite Degeneration and Reduced Longevity Due to Infection

Cell reports (Cambridge), 2020-05, Vol.31 (7), p.107662-107662, Article 107662

2020

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Myotubularin-related protein protects against neuronal degeneration mediated by oxidative stress or infection

The Journal of biological chemistry, 2022-03, Vol.298 (3), p.101614-101614, Article 101614

2022

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Heat-Shock Transcription Factor (HSF)-1 Pathway Required for Caenorhabditis elegans Immunity

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (35), p.13092-13097

2006

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Endoplasmic Reticulum Stress Caused by Lipoprotein Accumulation Suppresses Immunity against Bacterial Pathogens and Contributes to Immunosenescence

mBio, 2017-05, Vol.8 (3)

2017

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Dissection of a sensorimotor circuit underlying pathogen aversion in C. elegans

BMC biology, 2022-10, Vol.20 (1), p.229-229, Article 229

2022

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Heme peroxidase HPX-2 protects Caenorhabditis elegans from pathogens

A heritable antiviral RNAi response limits Orsay virus infection in Caenorhabditis elegans N2

Caenorhabditis elegans: a simple nematode infection model for Penicillium marneffei

Using a health-rating system to evaluate the usefulness of Caenorhabditis elegans as a model for anthelmintic study

Cranberry extract standardized for proanthocyanidins promotes the immune response of Caenorhabditis elegans to Vibrio cholerae through the p38 MAPK pathway and HSF-1

The pore-forming protein Cry5B elicits the pathogenicity of Bacillus sp. against Caenorhabditis elegans

LEM-3 - A LEM domain containing nuclease involved in the DNA damage response in C. elegans

Depletion of the C. elegans NAC engages the unfolded protein response, resulting in increased chaperone expression and apoptosis

WWP-1 is a novel modulator of the DAF-2 insulin-like signaling network involved in pore-forming toxin cellular defenses in Caenorhabditis elegans

RNAi screen of DAF-16/FOXO target genes in C. elegans links pathogenesis and dauer formation

Programmed Cell Death Mediated by ced-3 and ced-4 Protects Caenorhabditis elegans from Salmonella typhimurium-Mediated Killing

DAF-2/insulin-like signaling in C. elegans modifies effects of dietary restriction and nutrient stress on aging, stress and growth

Salmonella typhimurium proliferates and establishes a persistent infection in the intestine of Caenorhabditis elegans

Caenorhabditis elegans as a host for the study of host–pathogen interactions

Caenorhabditis elegans-Based Screen Identifies Salmonella Virulence Factors Required for Conserved Host-Pathogen Interactions

Cellular Responses to Infections in Caenorhabditis elegans

Yersinia pestis kills Caenorhabditis elegans by a biofilm-independent process that involves novel virulence factors

Intestinal infection regulates behavior and learning via neuroendocrine signaling

Neural control of behavioral and molecular defenses in C. elegans

Role of the nervous system in the control of proteostasis during innate immune activation: insights from C. elegans

G-Protein-Coupled Receptor SRBC-48 Protects against Dendrite Degeneration and Reduced Longevity Due to Infection

Myotubularin-related protein protects against neuronal degeneration mediated by oxidative stress or infection

Heat-Shock Transcription Factor (HSF)-1 Pathway Required for Caenorhabditis elegans Immunity

Endoplasmic Reticulum Stress Caused by Lipoprotein Accumulation Suppresses Immunity against Bacterial Pathogens and Contributes to Immunosenescence

Dissection of a sensorimotor circuit underlying pathogen aversion in C. elegans

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