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The Neuronal Ceroid–Lipofuscinoses

Journal of neuropathology and experimental neurology, 2003-01, Vol.62 (1), p.1-13

HALTIA, MATTI

2003

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MRI of neuronal ceroid lipofuscinosis. II. Postmortem MRI and histopathological study of the brain in 16 cases of neuronal ceroid lipofuscinosis of juvenile or late infantile type

Neuroradiology, 1997-05, Vol.39 (5), p.371-377

1997

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Multiple sclerosis: In situ evidence for antibody- and complement-mediated demyelination

Annals of neurology, 1998-04, Vol.43 (4), p.465-471

1998

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Accuracy of the clinical diagnosis of vascular dementia: a prospective clinical and post-mortem neuropathological study

Journal of neurology, neurosurgery and psychiatry, 1988-08, Vol.51 (8), p.1037-1044

1988

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Progressive encephalopathy with edema, hypsarrhythmia, and optic atrophy (PEHO syndrome)

Clinical genetics, 1991-04, Vol.39 (4), p.287-293

1991

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Sphingolipid activator proteins (SAPs) in neuronal ceroid lipofuscinoses (NCL)

Neuropediatrics, 1997-02, Vol.28 (1), p.49-52

1997

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Variant Alzheimer Disease With Spastic Paraparesis: Neuropathological Phenotype

Journal of neuropathology and experimental neurology, 2001-05, Vol.60 (5), p.483-492

2001

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Abundant expression of HIV Nef and Rev proteins in brain astrocytes in vivo is associated with dementia

AIDS (London), 1995-09, Vol.9 (9), p.1001-1008

1995

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Familial Creutzfeldt-Jakob Disease in Finland: Epidemiological, Clinical, Pathological and Molecular Genetic Studies

European journal of epidemiology, 1991-09, Vol.7 (5), p.494-500

1991

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Accuracy of clinical diagnosis in primary degenerative dementia: correlation with neuropathological findings

Journal of neurology, neurosurgery and psychiatry, 1983-01, Vol.46 (1), p.9-13

1983

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Radiological study of the brain at various stages of human immunodeficiency virus infection : early development of brain atrophy

Neuroradiology, 1992, Vol.34 (3), p.190-196

1992

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Microangiopathy with encephalopathy, hearing loss and retinal arteriolar occlusions: Two new cases

Journal of the neurological sciences, 1988-09, Vol.86 (2), p.239-250

1988

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Topographic heterogeneity of amyloid B-protein epitopes in brains with various forms of neuronal ceroid lipofuscinoses suggesting defective processing of amyloid precursor protein

Acta neuropathologica, 1990-01, Vol.80 (1), p.26-34

1990

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Dementia associated with human immunodeficiency virus: subcortical or cortical?

Acta psychiatrica Scandinavica, 1991-04, Vol.83 (4), p.297-301

1991

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Glial filaments are a major brain fraction in infantile neuronal ceroid-lipofuscinosis

Acta neuropathologica, 1985-01, Vol.65 (3-4), p.190-194

1985

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Brain pathology in the Meckel syndrome: a study of 59 cases

Clinical neuropathology, 1985-03, Vol.4 (2), p.56

1985

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Spontaneous occlusion of the circle of Willis (moyamoya syndrome)

Clinical neuropathology, 1982, Vol.1 (1), p.11

1982

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Sphingolipid activator proteins in the neuronal ceroid-lipofuscinoses : an immunological study

Acta neuropathologica, 1995-05, Vol.89 (5), p.391-398

1995

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Neuronal Ceroid-Lipofuscinosis - Late-Infantile or Jansky-Bielschowsky Type - Revisited

Brain pathology (Zurich, Switzerland), 1996-07, Vol.6 (3), p.225-228

1996

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Toward Understanding the Neuronal Pathogenesis of Aspartylglucosaminuria: Expression of Aspartylglucosaminidase in Brain during Development

Molecular genetics and metabolism, 1999-08, Vol.67 (4), p.294-307

1999

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Aspartylglycosaminuria: a generalized storage disease. Morphological and histochemical studies

Acta neuropathologica, 1975-01, Vol.31 (3), p.243-255

1975

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Aspartylglucosaminuria: Radiologic Course of the Disease With Histopathologic Correlation

Journal of child neurology, 1997-09, Vol.12 (6), p.369-375

1997

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MRI Evaluation of the Brain in Infantile Neuronal Ceroid-Lipofuscinosis. Part 1: Postmortem MRI With Histopathologic Correlation

Journal of child neurology, 1995-11, Vol.10 (6), p.438-443

1995

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Infantile form of so-called neuronal ceroid lipofuscinosis: lipid biochemical studies, fatty acid analysis of cerebroside sulfatides and sphingomyelin, myelin density profile and lipid composition

European neurology, 1979-01, Vol.18 (5), p.312-321

1979

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Sphingolipid activator proteins (SAPS) are stored together with glycosphingolipids in the infantile neuronal ceroid-lipofuscinosis (INCL)

American journal of medical genetics, 1995-06, Vol.57 (2), p.294-297

1995

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The Neuronal Ceroid–Lipofuscinoses

MRI of neuronal ceroid lipofuscinosis. II. Postmortem MRI and histopathological study of the brain in 16 cases of neuronal ceroid lipofuscinosis of juvenile or late infantile type

Multiple sclerosis: In situ evidence for antibody- and complement-mediated demyelination

Accuracy of the clinical diagnosis of vascular dementia: a prospective clinical and post-mortem neuropathological study

Progressive encephalopathy with edema, hypsarrhythmia, and optic atrophy (PEHO syndrome)

Sphingolipid activator proteins (SAPs) in neuronal ceroid lipofuscinoses (NCL)

Variant Alzheimer Disease With Spastic Paraparesis: Neuropathological Phenotype

Abundant expression of HIV Nef and Rev proteins in brain astrocytes in vivo is associated with dementia

Familial Creutzfeldt-Jakob Disease in Finland: Epidemiological, Clinical, Pathological and Molecular Genetic Studies

Accuracy of clinical diagnosis in primary degenerative dementia: correlation with neuropathological findings

Radiological study of the brain at various stages of human immunodeficiency virus infection : early development of brain atrophy

Microangiopathy with encephalopathy, hearing loss and retinal arteriolar occlusions: Two new cases

Topographic heterogeneity of amyloid B-protein epitopes in brains with various forms of neuronal ceroid lipofuscinoses suggesting defective processing of amyloid precursor protein

Dementia associated with human immunodeficiency virus: subcortical or cortical?

Glial filaments are a major brain fraction in infantile neuronal ceroid-lipofuscinosis

Brain pathology in the Meckel syndrome: a study of 59 cases

Spontaneous occlusion of the circle of Willis (moyamoya syndrome)

Sphingolipid activator proteins in the neuronal ceroid-lipofuscinoses : an immunological study

Neuronal Ceroid-Lipofuscinosis - Late-Infantile or Jansky-Bielschowsky Type - Revisited

Toward Understanding the Neuronal Pathogenesis of Aspartylglucosaminuria: Expression of Aspartylglucosaminidase in Brain during Development

Aspartylglycosaminuria: a generalized storage disease. Morphological and histochemical studies

Aspartylglucosaminuria: Radiologic Course of the Disease With Histopathologic Correlation

MRI Evaluation of the Brain in Infantile Neuronal Ceroid-Lipofuscinosis. Part 1: Postmortem MRI With Histopathologic Correlation

Infantile form of so-called neuronal ceroid lipofuscinosis: lipid biochemical studies, fatty acid analysis of cerebroside sulfatides and sphingomyelin, myelin density profile and lipid composition

Sphingolipid activator proteins (SAPS) are stored together with glycosphingolipids in the infantile neuronal ceroid-lipofuscinosis (INCL)

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