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Succinylation Links Metabolism to Protein Functions

Neurochemical research, 2019-10, Vol.44 (10), p.2346-2359

2019

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The α-Ketoglutarate Dehydrogenase Complex as a Hub of Plasticity in Neurodegeneration and Regeneration

International journal of molecular sciences, 2022-10, Vol.23 (20), p.12403

2022

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Up‐regulation of the mitochondrial malate dehydrogenase by oxidative stress is mediated by miR‐743a

Journal of neurochemistry, 2011-08, Vol.118 (3), p.440-448

2011

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Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Biochimica et biophysica acta, 2010-01, Vol.1802 (1), p.122-134

2010

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Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Human molecular genetics, 2018-08, Vol.27 (16), p.2874-2892

2018

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Serum Metabolomic and Lipidomic Profiling Reveals Novel Biomarkers of Efficacy for Benfotiamine in Alzheimer's Disease

International journal of molecular sciences, 2021-12, Vol.22 (24), p.13188

2021

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Interactions of Mitochondria/Metabolism and Calcium Regulation in Alzheimer’s Disease: A Calcinist Point of View

Neurochemical research, 2017-06, Vol.42 (6), p.1636-1648

2017

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Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Neurochemical research, 2010-12, Vol.35 (12), p.2107-2116

2010

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Mitochondria/metabolic reprogramming in the formation of neurons from peripheral cells: Cause or consequence and the implications to their utility

Neurochemistry international, 2018-07, Vol.117, p.65-76

2018

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Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism

Journal of neurochemistry, 2001-02, Vol.76 (4), p.1010-1021

2001

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Alpha‐ketoglutarate dehydrogenase complex‐dependent succinylation of proteins in neurons and neuronal cell lines

Journal of neurochemistry, 2015-07, Vol.134 (1), p.86-96

2015

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Abnormal Glucose Metabolism in Alzheimer’s Disease: Relation to Autophagy/Mitophagy and Therapeutic Approaches

Neurochemical research, 2015-12, Vol.40 (12), p.2557-2569

2015

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The human brain acetylome reveals that decreased acetylation of mitochondrial proteins associates with Alzheimer's disease

Journal of neurochemistry, 2021-07, Vol.158 (2), p.282-296

2021

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Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease

Neurochemistry international, 2009-02, Vol.54 (2), p.111-118

2009

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Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Journal of neurochemistry, 2016-12, Vol.139 (5), p.823

2016

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Selective linkage of mitochondrial enzymes to intracellular calcium stores differs between human‐induced pluripotent stem cells, neural stem cells, and neurons

Journal of neurochemistry, 2021-03, Vol.156 (6), p.867-879

2021

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Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

The Journal of biological chemistry, 2011-05, Vol.286 (20), p.17640-17648

2011

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Thiamine-dependent processes and treatment strategies in neurodegeneration

Antioxidants & redox signaling, 2007-10, Vol.9 (10), p.1605

2007

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The negative impact of α‐ketoglutarate dehydrogenase complex deficiency on matrix substrate‐level phosphorylation

The FASEB journal, 2013-06, Vol.27 (6), p.2392-2406

2013

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Mild mitochondrial metabolic deficits by α-ketoglutarate dehydrogenase inhibition cause prominent changes in intracellular autophagic signaling: Potential role in the pathobiology of Alzheimer's disease

Neurochemistry international, 2016-06, Vol.96, p.32-45

2016

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Interactions of oxidative stress with thiamine homeostasis promote neurodegeneration

Neurochemistry international, 2002-05, Vol.40 (6), p.493-504

2002

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Novel functions of the α-ketoglutarate dehydrogenase complex may mediate diverse oxidant-induced changes in mitochondrial enzymes associated with Alzheimer's disease

Biochimica et biophysica acta, 2008-04, Vol.1782 (4), p.229-238

2008

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Interactions of oxidative stress with cellular calcium dynamics and glucose metabolism in Alzheimer's disease

Free radical biology & medicine, 2002-06, Vol.32 (11), p.1061-1070

Gibson, Gary E

2002

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Mild reduction in the activity of the α‐ketoglutarate dehydrogenase complex elevates GABA shunt and glycolysis

Journal of neurochemistry, 2009-05, Vol.109 (s1), p.214-221

2009

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Selective response of various brain cell types during neurodegeneration induced by mild impairment of oxidative metabolism

Neurochemistry international, 2004-07, Vol.45 (2), p.361-369

2004

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Succinylation Links Metabolism to Protein Functions

The α-Ketoglutarate Dehydrogenase Complex as a Hub of Plasticity in Neurodegeneration and Regeneration

Up‐regulation of the mitochondrial malate dehydrogenase by oxidative stress is mediated by miR‐743a

Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Serum Metabolomic and Lipidomic Profiling Reveals Novel Biomarkers of Efficacy for Benfotiamine in Alzheimer's Disease

Interactions of Mitochondria/Metabolism and Calcium Regulation in Alzheimer’s Disease: A Calcinist Point of View

Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Mitochondria/metabolic reprogramming in the formation of neurons from peripheral cells: Cause or consequence and the implications to their utility

Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism

Alpha‐ketoglutarate dehydrogenase complex‐dependent succinylation of proteins in neurons and neuronal cell lines

Abnormal Glucose Metabolism in Alzheimer’s Disease: Relation to Autophagy/Mitophagy and Therapeutic Approaches

The human brain acetylome reveals that decreased acetylation of mitochondrial proteins associates with Alzheimer's disease

Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease

Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Selective linkage of mitochondrial enzymes to intracellular calcium stores differs between human‐induced pluripotent stem cells, neural stem cells, and neurons

Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

Thiamine-dependent processes and treatment strategies in neurodegeneration

The negative impact of α‐ketoglutarate dehydrogenase complex deficiency on matrix substrate‐level phosphorylation

Mild mitochondrial metabolic deficits by α-ketoglutarate dehydrogenase inhibition cause prominent changes in intracellular autophagic signaling: Potential role in the pathobiology of Alzheimer's disease

Interactions of oxidative stress with thiamine homeostasis promote neurodegeneration

Novel functions of the α-ketoglutarate dehydrogenase complex may mediate diverse oxidant-induced changes in mitochondrial enzymes associated with Alzheimer's disease

Interactions of oxidative stress with cellular calcium dynamics and glucose metabolism in Alzheimer's disease

Mild reduction in the activity of the α‐ketoglutarate dehydrogenase complex elevates GABA shunt and glycolysis

Selective response of various brain cell types during neurodegeneration induced by mild impairment of oxidative metabolism

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